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. 2018 Jan 1;8(1):45–60. doi: 10.7150/thno.20885

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© Ivyspring International Publisher

This is an open access article distributed under the terms of the Creative Commons Attribution (CC BY-NC) license (https://creativecommons.org/licenses/by-nc/4.0/). See http://ivyspring.com/terms for full terms and conditions.

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Comparison of PPARγ, LXRα, ABCA1, ABCG1 activity and MMP-9 expression in MMR-Lobe group vs. lobeglitazone per se, MMR vehicle, or control groups. (A) Representative images of aortic plaques co-stained with antibodies against PPARγ (green), LXRα (blue), ABCA1 (red), and ABCG1(red) in the MMR-Lobe treated group. Quantitative analysis of PPARγ (B), LXRα (C), ABCA1 (D), ABCG1 (E) activation and MMP-9 expression (F). The co-expressions of PPARγ, LXRα, ABCA1, and ABCG1 in the plaques were strongly enhanced by MMR-Lobe treatment, but the expression of MMP-9 decreased significantly in the MMR-Lobe-treated mice. Data are presented as mean ± SD. n = 6 mice per group; * P < 0.05, ** P < 0.01, *** P < 0.001 by ANOVA followed by Mann-Whitney test.