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. Author manuscript; available in PMC: 2019 Mar 1.
Published in final edited form as: Virology. 2018 Mar;516:246–257. doi: 10.1016/j.virol.2018.01.025

Fig. 6. VIM distribution is altered in HIV-infected cells.

Fig. 6

(A) Protein-protein interaction network of VIM with other candidate factors in combined SWATH dataset. (B) Immunoblots of VIM in subcellular fractions of uninfected (day 0) and HIV-1 infected Jurkat cells at dpi shown. Control blots for fractionation were performed as shown in Fig. 2.