Skip to main content
EFORT Open Reviews logoLink to EFORT Open Reviews
. 2018 Jan 15;3(1):15–23. doi: 10.1302/2058-5241.3.160089

Locked posterior dislocation of the shoulder

A systematic review

Ozgur Basal 1,, Recep Dincer 2, Bulent Turk 3
PMCID: PMC5890132  PMID: 29657841

Abstract

  • Locked posterior dislocation of the shoulder is very rare. Seizures and trauma are the most common causes of this injury.

  • There is no current benchmark treatment strategy for these rare cases.

  • This study has shown that reconstruction of the shoulder joint in an anatomical way in acute and chronic cases up to 16 weeks provides good results.

  • The purpose of this study is to evaluate the results of different treatment procedures with outcomes and to compare the results of the same procedures in acute and chronic cases.

Cite this article: EFORT Open Rev 2017;3:15–23. DOI: 10.1302/2058-5241.3.160089.

Keywords: locked posterior dislocation of the shoulder, posterior fracture-dislocation of the shoulder, reverse Hill-Sachs lesion

Introduction

Locked posterior dislocation of the shoulder (LPDS) is an uncommon condition that is often misdiagnosed and becomes chronic due to an inadequate physical examination. LPDS cases include actual fracture-dislocations, impression fractures and isolated posterior dislocations without any fracture pattern.1-3 Posterior fracture-dislocation of the shoulder (PFDS) is rare in orthopaedic practice, constituting only 2% to 4% of all shoulder dislocations, and its annual incidence is 0.6 in 100 000.4,5 Impression fracture of the anteromedial humeral head (also called ‘reverse Hill-Sachs lesion’ [RHL]) is reported to occur in 40% to 90% of patients with an initial dislocation.6,7 In addition to their rarity, what makes LPDS cases important is that they are easily missed. These cases are frequently missed in the initial examination as the occurrence of the dislocation is overlooked because urgent treatment of the seizure is the priority.5,8 Cases that are diagnosed after three weeks are called ‘neglected’ PFDSs, while cases neglected for more than three weeks are called ‘chronic’ PFDSs.9 When the literature is reviewed, almost all chronic PFDS cases were reported to be LPDSs.10 However, the group of LPDS may include the cases of PFDS as well because it can only occur with a RHL. Thus, it is necessary to evaluate the surgical and conservative treatment results of two different patient groups separately. PFDS cases are classified by the number of fragments, as described by Neer.11 According to this classification, the treatment results of fractures with three and four parts are not satisfactory. The number of fragments, the time elapsed from injury to surgery, age, treatment options and the experience of the surgeon all affect the results. There is no generally accepted approach for the treatment of neglected PFDS cases. However, the treatment is decided according to the amount of impaction (%) in isolated cases accompanied by the RHL. In these isolated cases, techniques such as the disimpaction of the fracture, lesser tubercle transfer, reconstruction with allograft and the filling of the defect are described.12-15 Arthroplasty is preferred in cases in which 50% or more of the articular surface is affected.16

There is no gold standard treatment for LPDS and no specific study on the treatment algorithm has been published. This article provides a systematic review of the current literature, describes the diagnosis and discusses the different treatment options for LPDS.

Materials and methods

Posterior dislocation cases reported between 1987 and 2016 and accompanied by PFDS, as well as isolated RHLs, were included in this study. The PubMed, Web of Science and ScienceDirect databases were scanned for this purpose. Databases were searched using the term ‘posterior dislocation and shoulder’ in the title/abstract/keyword parts according to the Boolean operator scanning principles. The articles which were especially reported were those with numerical shoulder scores, such as Constant-Murley scoring (CMS). In total, 1120 articles were found in PubMed, 796 in Web of Science and 238 in ScienceDirect. Articles taken from all three databases were combined with Endnote X7 software. Duplicated publications were removed. The Endnote library was simplified according to the inclusion/exclusion criteria (Table 1). Articles in English for adult patients were included. Full-text scanning was made for 486 articles after the selection. The cases were divided into two groups according to our conformity criteria of acute or misdiagnosed/chronic. The articles were scanned with the words ‘missed’, ‘neglected’, ‘chronic’, ‘overlooked’, ‘unreduced’ and ‘undiagnosed’ in order to separate chronic or neglected cases. LPDSs that were diagnosed late were noted within the specified criteria. Finally, this study included 30 articles (111 cases) in the acutely treated LPDS group and 31 articles (91 cases) in the neglected LPDS group. In total, 104 neglected and 124 acutely treated shoulders were assessed according to the treatment and its results.

Table 1.

Inclusion/exclusion criteria

Inclusion criteria Exclusion criteria
General adult population Case reports/series with no details about patients (age, treatment, follow-up, results)
PFDS or RHL with 20% and over defect Only observation or descriptive studies without follow-up
Cases with treatment and detailed result Cases with shoulder instability or recurrence
Patients with a minimum of ten months follow-up Review articles, radiological reports, technical notes
Interval between injury and treatment reported patients Patients with glenoid bone loss or fracture
Original publications in English language At least one of these scores not reported cases (CMS, ASES, Rowe, Neer, JOA)

LPDS, locked posterior dislocation of shoulder; PFDS, posterior fracture-dislocation of shoulder; RHL, reverse Hill-Sachs lesion; CMS, Constant-Murley score; ASES, American Shoulder and Elbow Surgeons; JOA, Japanese Orthopedic Association

Statistical analysis

All data are expressed as means ± standard deviation (SD). Statistical analysis was performed using SPSS 20.0 (IBM, Chicago, IL, USA). Descriptive statistics are reported as the mean with the range for continuous measures and as the number and percentage of discrete measures. Independent samples t-test was used to compare acute and chronic groups. Non-parametric data were analysed by the Mann-Whitney U and Wilcoxon tests for two independent samples. K independent samples with the Kruskal-Wallis test was used to compare selected cases. The values of p < 0.05 were considered as significant.

Results

PFDS cases made up a major part of acutely treated LPDS cases. Of 124 acute LPDS that we included in our study, 94 (75.8%) were also PFDS. Only 20 shoulders (19.23%) in the chronic LPDS group were treated with the PFDS diagnosis. When LPDS cases are assessed aetiologically, the most frequent cause in both acute and chronic cases was a seizure. This was followed by falling and traffic accidents (Table 2). The seizure-related LPDS rate of incidence (66.7%) was higher in chronic cases than acute cases. There were 26 LPDS patients with bilateral involvement, 23 (88.46%) of which were attributed to seizures and three (11.64%) were caused by electric shock. We believe that seizure-related LPDS is frequently neglected in the first examination (p = 0.021). The other most frequent reasons for LPDS are falling (including indirect traumas), traffic accidents and electric shocks. Acute and chronic percentages are close (31.1% to 17.9%) in trauma-related LPDS cases, and this verifies that LPDS cases are fractures that can be easily neglected. LPDS cases can be missed in the initial examination independently of aetiology despite radiological and clinical findings.

Table 2.

Etiological distribution of acute and chronic cases

LPDS Seizure n (%) Fall n (%) RTA n (%) Electrocution n (%) Sports injuries n (%)
Acute cases 34 (45.9) 23 (31.1) 11 (14.9) 4 (5.4) 2 (2.7)
Chronic cases 52 (66.7) 14 (17.9) 5 (6.4) 6 (7.7) 1 (1.3)
Undetailed case series 80 (39.8) 73 (36.31) 31 (15.42) 17 (8.45) 0
Total 166 (47.02) 110 (31.16) 47 (13.31) 27 (7.64) 3 (0.84)

RTA: road traffic accidents

The time from injury to surgery (TFIS) duration also affects the results of chronic LPDS treatment. However, whether the poor results are related to a time delay or the preferred method of treatment is not clear. The treatments and results of the cases included in the study are listed in Table 3. The cases in which two or more of the summarized treatment methods were applied together were named ‘combined procedures’ (e.g. open reduction and stabilization with modified McLaughlin procedure). In this study, the average TFIS was found to be 23.30 ± 41.68 weeks (3 to 344). Although LPDS cases with longer delay were reported in the literature, they were not included in the study since they did not fit our inclusion criteria. Of 104 late-diagnosed shoulders, 75 (72.11%) were diagnosed within 16 weeks of the injury.

Table 3.

Analysis of acute and chronic LPDS cases according to age, bilaterality, RHL, CMS and follow-up

Age Bilaterality n (%) RHL (%) CMS Follow-up
Acute cases 43.87 ± 13.82 13 (23.7) 33.68 ± 11.06 83.54 ± 12.08 34.67 ± 23.33
Chronic cases 45.18 ± 12.02 13 (20.3) 38.97 ± 9.91 75.96 ± 16.95 40.19 ± 17.04

The average CMS of the missed LPDS cases operated within weeks 3 and 16 was 79.09 ± 15.72 (mean 52) and the score of those that were operated after week 16 was 67.83 ± 18.72 (mean 20). There was a significant difference between both groups and this difference resulted from the group that was operated between weeks 3 and 16 (p = 0.022). Upon investigating the preferred treatment options and results in acute and chronic LPDS cases, arthroplasty cases affected the results negatively in both groups (Table 4). When different treatment methods in chronic LPDS cases were compared, while there was no significant difference between those to which arthroplasty was applied and those to which rotational osteotomy was applied (p = 0.134); a statistical difference was found between the arthroplasty group and other treatment methods (p < 0.05). There was a significant difference between chronic LPDS cases treated with open reduction and allograft/autograft and the group in which rotational osteotomy was applied (p = 0.023). When the treatment results in the acutely treated group were compared, the CMS of the cases treated with open reduction and allograft/autograft had significantly higher scores when compared with those to which anatomical reconstruction with a plate or K-wire was applied (p = 0.018). On the other hand, no significant difference was found between the cases in which closed reduction was applied in the acutely treated group and the cases anatomically reconstructed with a plate or K-wire (p = 0.26). PFDS cases to which only open reduction (without fixation) was applied in the acute LPDS group are summarized in Table 3. The results of this treatment, especially in dislocations with two- and three-part fractures, were reported to be excellent and satisfactory, but precise comparison could not be made since they had no CMS (Table 3).

Table 4.

The treatments and results of the cases included in the study

Acute
Chronic
n RHL (%)
Mean ± SD
Constant score
Mean ± SD
Min-max. CMS Follow-up (months) n TFIS (week) RHL (%)
Mean ± SD
Constant score
Mean ± SD
Min-max. CMS Follow-up (months) p-values
Arthroplasty (TSA/HA) 8 50 ± 8.16 59 ± 11.14 49-71 25.87 ± 24.99 17 65.59 ± 89.8 49.5 ± 5.5 60.61 ± 10.20 42-82 27.36 ± 14.49 0.885
Allograft/autograft fixation 10 37.5 ± 10.87 86.78 ± 3.60 83-95 48.2 ± 29.33 4 12.75 ± 8.62 40 ± 8.16 89.67 ± 3.51 86-93 35 ± 24.08 0.160
McLaughlin/modified McLaughlin procedure 5 29.4 ± 3.58 99.33 ± 1.15* 98-100 65.4 ± 26.84 28 15.61 ± 15.27 34.83 ± 5.42 78.40 ± 10.71 60-86 23.57 ± 10.92 0.023*
Modified McLaughlin procedure + graft fixation 0 7 8.43 ± 6 39.29 ± 6.73 83.5 ± 5.36 77-90 22.43 ± 4.96
Balloon expansion + PMMA injection 1 NR 84.5 84.5 24 2 3 30 79 ± 1.41 78-80 22
Glenoid augmentation + graft 0 8 21.37 ± 9.91 39.25 ± 11.27 77.5 ± 18.08 55-98 43 ± 8.7
Rotational osteotomy 0 9 21.56 ± 17.98 30.56 ± 7.26 71.67 ± 18.87 40-90 21.67 ± 6.95
Frozen spherical-shaped allograft 1 35 12 25 14.32 ± 9.08 40.8 ± 8.12 79.52 ± 18.90 40-100 81.72 ± 33.43
Combined procedures 3 NR 90.33 ± 6.35 83-94 24 1 12 30 69 69 36
Closed reduction (with or without pin fixation) 19 24.83 ± 4 85 ± 15.11 58-100 38.63 ± 27.67 2 6 ± 2.83 20 100 100 48
Anatomic reconstruction with plate or K-wire fixation 67 NR 82.6 ± 11.24 16-100 28.79 ± 15.26 1 8 75% NR 24
Bioabsorbable screw reconstruction 2 45 92.5 ± 3.54 90-95 26 0
Open reduction 8 NR 57.5 ± 33.18 0
Total 124 104
*

there is a significant difference between groups (p < 0.05)

improper result or outcome

TSA, total shoulder arthroplasty; HA, hemiarthroplasty; RHL, reverse Hill-Sachs lesion; SD, standard deviation; PMMA, polymethyl methacrylate; K, Kirschner; CMS, Constant-Murray score; TFIS, time from injury to surgery; NR, not reported

Fig. 1.

Fig. 1

Pre-operative axial and three-dimensional tomography scans (A1, A2) and post-operative radiographs of the both shoulders at 24 months (B1, B2 / R, right; L, left) of a 37-year-old male who sustained an LPDS during a seizure and was treated at the fifth week after injury.

There are common treatment methods recommended in both acute and chronic LPDS cases. These are the McLaughlin procedure, filling of the Hill-Sachs defect with allograft/autograft, balloon expansion + polymethyl methacrylate injection, closed reduction and arthroplasties. Of these treatment methods, only the McLaughlin procedure yields better results in acute cases (p = 0.023). Other treatment methods yield similar results in acute and chronic LPDS cases. Thirty-five acute LPDS cases treated with anatomical reconstruction fixed with a plate or K-wire were compared with 25 chronic LPDS cases treated with a spherical-shaped allograft obtained from the femoral head. No statistically significant difference was found between these two treatment groups (p = 0.869) (Table 5).

Table 5.

Shoulders with acute or delayed diagnosis and authors’ treatment procedures

Author Shoulder (N) Treatment option Diagnosis
Kokkalis et al14 6 Modified McLaughlin technique + allograft Delayed
Martinez et al15 6 Spherical-shaped femoral head allograft fixation Delayed
Aksekili et al10 7 Glenoid augmentation with autograft Delayed
Diklic et al2 13 Spherical-shaped femoral head allograft fixation Delayed
Keppler et al17 9 Rotational osteotomy Delayed
Shams et al7 11 Modified McLaughlin technique + grafting Delayed
Cheng et al16 7 Total shoulder arthroplasty Delayed
Abdel-Hameed et al9 3 Modified McLaughlin technique + grafting Delayed
Gavriilidis et al18 3 Shoulder arthroplasty Delayed
Gerber and Lambert19 4 Spherical-shaped femoral head allograft fixation Delayed
Elmali et al20 2 Spherical-shaped femoral head allograft fixation Delayed
Benhamida et al21 2 Modified McLaughlin technique + grafting Delayed
Amir et al22 2 McLaughlin technique Delayed
Jacquot et al13 2 Balloon expansion and PMMA injection Delayed
Rodia et al23 1 Allograft fixation Delayed
Ivkovic et al24 2 Autograft fixation/Hemiarthroplasty Delayed
Bock et al5 1 Allograft/Autograft fixation Delayed
Verma et al25 1 Closed reduction Delayed
Bekmezci and Altan26 1 Modified McLaughlin technique + grafting Delayed
Kumar et al27 1 Combined procedures* Delayed
Chalidis et al28 1 Modified McLaughlin technique + grafting Delayed
Takase et al29 1 Hemiarthroplasty Delayed
Karachalios et al30 1 Open reduction and posterior capsular reconstruction Delayed
Tellisi et al31 2 ORIF/CR Delayed
Dervin et al32 1 Modified McLaughlin technique + grafting Delayed
Aparicio et al33 2 Modified McLaughlin technique + grafting Delayed
Poyanli et al34 2 Hemiarthroplasty/Modified McLaughlin technique Delayed
Torrens et al35 2 Allograft/Autograft fixation/Hemiarthroplasty Delayed
Kılıçoğlu et al36 2 Hemiarthroplasty Delayed
Popelka37 1 Total shoulder arthroplasty Delayed
Delcogliano et al38 4 Modified McLaughlin/McLaughlin technique Delayed
Begin et al39 2 Allograft/Autograft fixation Acute
Khayal et al40 1 Allograft/Autograft fixation Acute
Altan et al6 1 Allograft/Autograft fixation (mosaicplasty) Acute
Duralde and Fogle41 4 Closed reduction Acute
Bock et al5 5 Allograft/Autograft fixation Acute
Cooke and Hackney42 2 Hemiarthroplasty Acute
Fukuda et al43 1 Anatomic reconstruction with plate or K-wire fixation Acute
Claro et al44 4 Anatomic reconstruction with plate or K-wire fixation
Hemiarthroplasty
Closed reduction and pin fixation
Acute
Miller and Lynch45 3 Modified McLaughlin technique + grafting Acute
Iosifidis et al46 2 Closed reduction Acute
Assom et al47 2 OR and bioabsorbable screw fixation Acute
De Wall et al48 3 Closed reduction and pin fixation Acute
Ide et al49 1 Anatomic reconstruction with plate or K-wire fixation Acute
Hayes et al50 1 Anatomic reconstruction with plate or K-wire fixation Acute
Altay et al51 10 Anatomic reconstruction with plate or K-wire fixation Acute
Soliman and Koptan12 21 Anatomic reconstruction with plate or K-wire fixation Acute
Fiorentino et al52 5 Anatomic reconstruction with plate or K-wire fixation
Combined procedures*
Acute
Robinson et al3 28 Anatomic reconstruction with plate or K-wire fixation Acute
Martens and Hessels53 2 Anatomic reconstruction with plate or K-wire fixation
Hemiarthroplasty
Acute
Finkelstein et al54 2 Modified McLaughlin technique Acute
Ogawa et al55 10 OR/CR Acute
Page et al56 2 Hemiarthroplasty Acute
Oakes and McAllister57 1 Anatomic reconstruction with plate or K-wire fixation Acute
Jacquot et al13 1 Balloon expansion and PMMA injection Acute
Ketenci et al58 2 CR Acute
Aparicio et al33 4 CR Acute
Ito et al59 1 Hemiarthroplasty Acute
Toker et al60 1 Allograft/Autograft fixation Acute
Riggenbach et al61 1 Hemiarthroplasty Acute
Mastrokalos et al62 1 Spherical-shaped femoral head allograft fixation Acute
*

combined procedures: open reduction and stabilization with modified McLaughlin procedure

OR, open reduction; CR, closed reduction; PMMA, polymethyl methacrylate; K, Kirschner

In chronic LPDS cases, no significant difference was observed between the results of the treatment performed using a graft and without using a graft in addition to the modified McLaughlin procedure (p = 0.460). Re-dislocation was observed in two cases of both groups. While avascular necrosis (AVN) is observed more frequently in acute cases despite being dependent on the treatment options, allograft flattening and arthritis were observed more in chronic cases.

Discussion

It is generally difficult to diagnose LPDS and it is frequently missed in the initial evaluation. Although the misdiagnosis rates were reported to be in the range of 60% to 80%, it was found to be 51% in our study.62 Shoulder dislocations can be divided into two: locked shoulder dislocations and unstable shoulders.64 Significant symptoms may occur such as swelling, pain and limitation of movement in the shoulder after injury. The most frequent reason for misdiagnosis is that anteroposterior (AP) radiographs are usually normal. Focusing on the seizure and not asking for the AP radiograph in PFDS cases with no direct trauma can cause misdiagnosis.

Seizures, falling/indirect trauma, traffic accidents, electric shocks and sports injuries, respectively, are the causes.62,64,65 Although seizure cases usually develop in an epileptic background, these seizures can rarely be seen in hypoglycaemic coma, vitamin D deficiency, aortic dissection, brain tumour and idiopathically.33,35,60,66,67 Higher rates of seizure-related chronic LPDS (66.7%) can be explained by the under-diagnosis of these cases. LPDS cases are a subset of posterior shoulder dislocations and they include cases with PFDS and isolated reverse Hill-Sachs defect. Although such a differentiation is not made in the literature, a major part (75.8%) of acute cases was made up of PFDS cases in our study. LPDS cases with PFDS and isolated RHL require different treatment procedures. No clear treatment algorithm has been suggested in PFDS cases when choosing the treatment according to the size of the defect in isolated RHL cases.

Closed reduction (with or without pin fixation), open reduction, open reduction and internal fixation (ORIF), ORIF + bone grafting and hemiarthroplasty are the preferred methods in the treatment of acute PFDS.5,11,40,41,54 Total shoulder prosthesis, McLaughlin procedure, spherical-shaped allograft fixation, glenoid augmentation and ORIF are certainly the preferred treatment methods in chronic PFDS;7,10,14-16 however, objective data for each method are not yet ensured since there are no large case series for each treatment method.

In this review, no difference was found between acute and chronic cases in which the RHL was filled with allograft or autograft independently of the percentage of the defect. The spherical femoral head allograft (SSFHA)-treated shoulders were also compared with allograft/autograft fixed group. The difficulties in supplying and the cost of frozen femoral head allografts are also a disadvantage. This analysis showed that there was no significant difference between those treated using the femoral head allograft and standard allo/autograft (p = 0.413). When acute and chronic cases treated with the McLaughlin procedure were compared, it was observed that the CM scores of acute LPDS cases were better (p = 0.023). According to this result, the success of the McLaughlin procedure decreases as TFIS increases. No difference was observed between acute and chronic groups in the case series treated using arthroplasty and allograft/autograft (Table 4).

The SSFHA was used in two different neglected LPDS case series by Diklic et al and Gerber and Lambert.2,18 Although the average CM scores of this method applied in cases with a defect in the range of 20% to 60% in reported studies are satisfactory, the complication rates are high. The SSFHA procedure may not be a good option considering its cost and complication rates.

There was no significant difference between chronic LPDS cases that are treated with rotational osteotomy or arthroplasty among the different treatment options (p = 0.134). Nine LPDS cases reported by Keppler et al were treated with rotational osteotomy.17 The average Rowe score of nine cases in total was found to be 71.67 ± 18.87. Although the average HSL (Hill-Sachs Lesion) is less when compared to other chronic LPDS treatment groups, rotational osteotomy results were found to be lower. It was considered that this treatment method is not a good treatment option for chronic LPDS cases, but it can be an alternative to hemiarthroplasty cases.

The acutely treated LPDS group was made up of PFDS cases at a rate of 75.8%. Cases treated with anatomical reconstruction and internal fixation in this group were three- and four-part fracture-dislocations. Cases treated with closed reduction (with/without pin fixation) mainly consisted of two- and three-part fracture-dislocations. No statistically significant difference was found between the treatment results of these two groups (p = 0.260). On the other hand, excellent and satisfactory results were obtained in the group including acute PFDS cases to which only open reduction was applied. The results of this group could not be compared with other groups since they were not scored over 100. Consequently, six humeral head AVNs that developed in acute LPDS cases were observed in the groups to which only open reduction was applied. The fact that AVN complication is observed more in cases to which open reduction is applied may be due to its containing three- and four-piece fractured dislocation cases and surgeon-related factors. Liu et al reported on a series of 18 patients with malunited chronic PFDS who were treated with anatomical reconstruction.69 They reconstructed two-part (lesser tubercle) malunited fracture dislocations, and after a mean follow-up of 38.1 ± 16.5, the mean CMS was 83.9 ± 8.3. In our study, no sufficient data were found on the treatment of malunited PFDS cases with anatomical reconstruction and internal fixation in the chronic LPDS group. However, it was reported that performing osteotomy, anatomical reconstruction and fixation suitable for the fracture line in malunited PFDS cases would not constitute risk of AVN (Table 6).68

Table 6.

Distribution of complications according to the groups

Acute LPDS Chronic LPDS
Avascular necrosis 6 2
Allograft collapse 1 3
Allograft flattening 1 10
Redislocation 2 2
Nonunion 1 -
Arthrosis/stiffness - 7
Total 11/124 (8.87%) 24/104 (23.07%)

The biggest challenge encountered in the planning of this review is that the treatment methods and assessment methods applied differ significantly. Forty-four different outcome scores in total, 22 of which were clinician-based and 21 of which were patient-based, were defined in the literature.69 The scoring methods in which scoring is performed over 100 were found to be CMS, ASES (American Shoulder and Elbow Surgeons), Rowe, Neer and Japanese Orthopaedic Association scores.1,15,50,70 The most widely used study results scored over 100 were included in this study.

When the treatment results of chronic LPDS cases are examined in detail, successful results are obtained in the cases operated in the first 16 weeks, while a decrease occurs in the CM scores of the cases treated after 16 weeks. In late diagnosed cases, it was reported that the shoulder can be anatomically reconstructed up to six months.5,8 In this study, we found the treatment results after four months to be 67.83 ± 18.72.

The modified McLaughlin technique is a frequently used technique for cases with an HSL in the range of 25% to 50%.7,9,13,34 The defect is filled by the osteotomized lesser tubercle with this technique. A graft can be added to the modified McLaughlin procedure according to the size of the defect.13 In the series of cases reported by Castagna et al, 16 cases with a defect in the range of 20% to 50% (41.9 years on average) were treated with the modified McLaughlin technique.8 The average times of delay of these cases were reported to be 5.7 months and the average CMS were reported to be 75.2. The results of this case series in which no complication is observed are lower when compared with our review data. In this technique, it is seen that joint ranges of motion theoretically decrease. The modified McLaughlin procedure may yield less successful results in PFDS cases. On the other hand, shoulder instability may develop in active individuals as a result of the changes in the tendon length in the long term, just as in the Magnuson-Stack procedure.71

Post-treatment complication rates were found to be 8.87% in acute LPDS cases and 23.07% in chronic LPDS cases. The most frequent complications of shoulder fracture-dislocations are AVN, secondary osteoarthritis and shoulder stiffness.72

Conclusions

LPDS has a complex injury pattern which includes PFDS, RHL and isolated PDSs. The number of bony fragments, time lapsed from injury to surgery, age, chosen treatment and the experience of the surgeon all affect the results. The strategy in delayed case series of reconstructing the shoulder joint in an anatomical way within a diagnostic delay period of up to 16 weeks instead of opting for a shoulder arthroplasty seems to be a promising one, knowing the outcome of shoulder arthroplasty. Reconstruction should be attempted to retain the humeral head and restore its shape primarily in delayed PFDS cases if potential signs of AVN are absent. Also, the surgeon should keep in mind that arthroplasty treatment as a salvage procedure has many pros and cons and should be selected very carefully.

Footnotes

ICMJE Conflict of interest statement: None declared.

Funding

No benefits in any form have been received or will be received from a commercial party related directly or indirectly to the subject of this article.

References

  • 1. Schliemann B, Muder D, Gessmann J, Schildhauer TA, Seybold D. Locked posterior shoulder dislocation: treatment options and clinical outcomes. Arch Orthop Trauma Surg 2011;131:1127-1134. [DOI] [PubMed] [Google Scholar]
  • 2. Diklic ID, Ganic ZD, Blagojevic ZD, Nho SJ, Romeo AA. Treatment of locked chronic posterior dislocation of the shoulder by reconstruction of the defect in the humeral head with an allograft. J Bone Joint Surg [Br] 2010;92:71-76. [DOI] [PubMed] [Google Scholar]
  • 3. Robinson CM, Seah M, Akhtar MA. The epidemiology, risk of recurrence, and functional outcome after an acute traumatic posterior dislocation of the shoulder. J Bone Joint Surg [Am] 2011;93:1605-1613. [DOI] [PubMed] [Google Scholar]
  • 4. Robinson CM, Akhtar A, Mitchell M, Beavis C. Complex posterior fracture-dislocation of the shoulder. Epidemiology, injury patterns, and results of operative treatment. J Bone Joint Surg [Am] 2007;89:1454-1466. [DOI] [PubMed] [Google Scholar]
  • 5. Bock P, Kluger R, Hintermann B. Anatomical reconstruction for Reverse Hill-Sachs lesions after posterior locked shoulder dislocation fracture: a case series of six patients. Arch Orthop Trauma Surg 2007;127:543-548. [DOI] [PubMed] [Google Scholar]
  • 6. Altan E, Senaran H, Acar MA, Aydin K, Ozbaydar MU. Mozaicplasty technique for treatment of reverse Hill-Sachs lesion. Tech Shoulder Elbow Surg 2013;14:1-4. [Google Scholar]
  • 7. Shams A, El-Sayed M, Gamal O, ElSawy M, Azzam W. Modified technique for reconstructing reverse Hill-Sachs lesion in locked chronic posterior shoulder dislocation. Eur J Orthop Surg Traumatol 2016;26:843-849. [DOI] [PubMed] [Google Scholar]
  • 8. Castagna A, Delle Rose G, Borroni M, et al. Modified MacLaughlin procedure in the treatment of neglected posterior dislocation of the shoulder. Chir Organi Mov 2009;93(S1):S1-S5. [DOI] [PubMed] [Google Scholar]
  • 9. Abdel-Hameed SK, Alzalabany A-KAI, Abdel-Aal MA, Soltan A-A. Reconstruction of humeral head defect in locked posterior dislocation shoulder. A case series of nine patients. Open Journal of Orthopedics 2015;5:25-33. [Google Scholar]
  • 10. Aksekili MA, Uğurlu M, Işık Ç, et al. Posterior bone block of chronic locked posterior shoulder dislocations with glenoid augmentation: a retrospective evaluation of ten shoulders. Int Orthop 2016;40:813-820. [DOI] [PubMed] [Google Scholar]
  • 11. Neer CS., II Displaced proximal humeral fractures. II. Treatment of three-part and four-part displacement. J Bone Joint Surg [Am] 1970;52:1090-1103. [PubMed] [Google Scholar]
  • 12. Soliman OA, Koptan WM. Four-part fracture dislocations of the proximal humerus in young adults: results of fixation. Injury 2013;44:442-447. [DOI] [PubMed] [Google Scholar]
  • 13. Jacquot F, Costil V, Werther JR, et al. Balloon treatment of posterior shoulder dislocation with reverse Hill-Sachs injury: description of a new technique. Int Orthop 2013;37:1291-1295. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 14. Kokkalis ZT, Mavrogenis AF, Ballas EG, Papanastasiou J, Papagelopoulos PJ. Modified McLaughlin technique for neglected locked posterior dislocation of the shoulder. Orthopedics 2013;36:e912-e916. [DOI] [PubMed] [Google Scholar]
  • 15. Martinez AA, Navarro E, Iglesias D, et al. Long-term follow-up of allograft reconstruction of segmental defects of the humeral head associated with posterior dislocation of the shoulder. Injury 2013;44:488-491. [DOI] [PubMed] [Google Scholar]
  • 16. Cheng SL, Mackay MB, Richards RR. Treatment of locked posterior fracture-dislocations of the shoulder by total shoulder arthroplasty. J Shoulder Elbow Surg 1997;6:11-17. [DOI] [PubMed] [Google Scholar]
  • 17. Keppler P, Holz U, Thielemann FW, Meinig R. Locked posterior dislocation of the shoulder: treatment using rotational osteotomy of the humerus. J Orthop Trauma 1994;8:286-292. [DOI] [PubMed] [Google Scholar]
  • 18. Gavriilidis I, Magosch P, Lichtenberg S, Habermeyer P, Kircher J. Chronic locked posterior shoulder dislocation with severe head involvement. Int Orthop 2010;34:79-84. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 19. Gerber C, Lambert SM. Allograft reconstruction of segmental defects of the humeral head for the treatment of chronic locked posterior dislocation of the shoulder. J Bone Joint Surg [Am] 1996;78:376-382. [DOI] [PubMed] [Google Scholar]
  • 20. Elmalı N, Taşdemir Z, Sağlam F, Gülabi D, Baysal Ö. One-stage surgical treatment of neglected simultaneous bilateral locked posterior dislocation of shoulder: a case report and literature review. Eklem Hastalik Cerrahisi 2015;26:175-180. [DOI] [PubMed] [Google Scholar]
  • 21. Benhamida MK, Ouertatani M, Hasayri I, et al. Locked posterior dislocation of the shoulder: A report of three cases. Chir Main 2015;34:98-101. [DOI] [PubMed] [Google Scholar]
  • 22. Amir MA, Alenazi B, Wyse RK, et al. Neglected bilateral posterior shoulder fracture dislocation in an uncontrolled seizure patient. Pak J Med Sci 2015;31:1018-1020. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 23. Rodia F, Ventura A, Touloupakis G, Theodorakis E, Ceretti M. Missed posterior shoulder dislocation and McLaughlin lesion after an electrocution accident. Chin J Traumatol 2012;15:376-378. [PubMed] [Google Scholar]
  • 24. Ivkovic A, Boric I, Cicak N. One-stage operation for locked bilateral posterior dislocation of the shoulder. J Bone Joint Surg [Br] 2007;89:825-828. [DOI] [PubMed] [Google Scholar]
  • 25. Verma NN, Sellards RA, Romeo AA. Arthroscopic reduction and repair of a locked posterior shoulder dislocation. Arthroscopy 2006;22:e1251-1255. [DOI] [PubMed] [Google Scholar]
  • 26. Bekmezci T, Altan E. Management and prognostic factors for delayed reconstruction of neglected posterior shoulder fracture-dislocation. Arch Trauma Res 2015;4:e29903. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 27. Kumar S, Chopra RK, Kashyap A, Arora S. Missed posterior shoulder dislocation with malunited proximal humerus fracture. Chin J Traumatol 2013;16:375-378. [PubMed] [Google Scholar]
  • 28. Chalidis BE, Papadopoulos PP, Dimitriou CG. Reconstruction of a missed posterior locked shoulder fracture-dislocation with bone graft and lesser tuberosity transfer: a case report. J Med Case Reports 2008;2:260. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 29. Takase K, Watanabe A, Yamamoto K. Chronic posterior dislocation of the glenohumeral joint complicated by a fractured proximal humerus: a case report. J Orthop Surg (Hong Kong) 2006;14:204-207. [DOI] [PubMed] [Google Scholar]
  • 30. Karachalios T, Bargiotas K, Papachristos A, Malizos KN. Reconstruction of a neglected posterior dislocation of the shoulder through a limited posterior deltoid-splitting approach. A case report. J Bone Joint Surg Am 2005;87:630-634. [DOI] [PubMed] [Google Scholar]
  • 31. Tellisi NK, Abusitta GR, Fernandes RJ. Bilateral posterior fracture dislocation of the shoulders following seizure. Saudi Med J 2004;25:1727-1729. [PubMed] [Google Scholar]
  • 32. Dervin GF, Brunet JA, Healey DC. A modification of the McLaughlin procedure as salvage for missed locked posterior fracture-dislocation of the humeral head: a case report. J Bone Joint Surg Am 2002;84-A:804-807. [DOI] [PubMed] [Google Scholar]
  • 33. Aparicio G, Calvo E, Bonilla L, Espejo L, Box R. Neglected traumatic posterior dislocations of the shoulder: controversies on indications for treatment and new CT scan findings. J Orthop Sci 2000;5:37-42. [DOI] [PubMed] [Google Scholar]
  • 34. Poyanli O, Gokcen B, Unay K, Akan K, Esenkaya I. Bilateral posterior shoulder dislocation with defect secondary to hypoglycemic coma. J Orthop Sci 2011;16:125-128. [DOI] [PubMed] [Google Scholar]
  • 35. Torrens C, Santana F, Melendo E, Marlet V, Caceres E. Osteochondral autograft and hemiarthroplasty for bilateral locked posterior dislocation of the shoulder. Am J Orthop (Belle Mead NJ) 2012;41:362-364. [PubMed] [Google Scholar]
  • 36. Kilicoglu O, Demirhan M, Yavuzer Y, Alturfan A. Bilateral posterior fracture-dislocation of the shoulder revealing unsuspected brain tumor: case presentation. J Shoulder Elbow Surg 2001;10:95-96. [DOI] [PubMed] [Google Scholar]
  • 37. Popelka V. [Chronic locked posterior shoulder dislocation treated by anatomical total shoulder arthroplasty]. Acta Chir Orthop Traumatol Cech 2016;83:55-61. [PubMed] [Google Scholar]
  • 38. Delcogliano A, Caporaso A, Chiossi S, et al. Surgical management of chronic, unreduced posterior dislocation of the shoulder. Knee Surg Sports Traumatol Arthrosc 2005;13:151-155. [DOI] [PubMed] [Google Scholar]
  • 39. Begin M, Gagey O, Soubeyrand M. Acute bilateral posterior dislocation of the shoulder: one-stage reconstruction of both humeral heads with cancellous autograft and cartilage preservation. Chir Main 2012;31:34-37. [DOI] [PubMed] [Google Scholar]
  • 40. Khayal T, Wild M, Windolf J. Reconstruction of the articular surface of the humeral head after locked posterior shoulder dislocation: a case report. Arch Orthop Trauma Surg 2009;129:515-519. [DOI] [PubMed] [Google Scholar]
  • 41. Duralde XA, Fogle EF. The success of closed reduction in acute locked posterior fracture-dislocations of the shoulder. J Shoulder Elbow Surg 2006;15:701-706. [DOI] [PubMed] [Google Scholar]
  • 42. Cooke SJ, Hackney RG. Bilateral posterior four-part fracture–dislocations of the shoulders following electric shock: A case report and literature review. Inj Extra 2005;36:90-95. [Google Scholar]
  • 43. Fukuda A, Nishimura A, Kato K, Sudo A. Arthroscopically assisted minimally invasive plate osteosynthesis for posterior fracture-dislocation of the shoulder. J Orthop Sci 2014;19:194-197. [DOI] [PubMed] [Google Scholar]
  • 44. Claro R, Sousa R, Massada M, Ramos J, Lourenço JM. Bilateral posterior fracture-dislocation of the shoulder: report of two cases. Int J Shoulder Surg 2009;3:41-45. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 45. Miller BG, Lynch B. Excellent long-term results for acute operative management of locked posterior shoulder dislocation. ANZ J Surg 2007;77:95. [DOI] [PubMed] [Google Scholar]
  • 46. Iosifidis MI, Giannoulis I, Traios S, Giantsis G. Simultaneous bilateral posterior dislocation of the shoulder: diagnostic problems and management. A case report. Knee Surg Sports Traumatol Arthrosc 2006;14:766-770. [DOI] [PubMed] [Google Scholar]
  • 47. Assom M, Castoldi F, Rossi R, Blonna D, Rossi P. Humeral head impression fracture in acute posterior shoulder dislocation: new surgical technique. Knee Surg Sports Traumatol Arthrosc 2006;14:668-672. [DOI] [PubMed] [Google Scholar]
  • 48. De Wall M, Lervick G, Marsh JL. Posterior fracture-dislocation of the proximal humerus: treatment by closed reduction and limited fixation: a report of four cases. J Orthop Trauma 2005;19:48-51. [DOI] [PubMed] [Google Scholar]
  • 49. Ide J, Honda K, Takagi K. Posterior dislocation of the shoulder associated with fracture of the humeral anatomical neck with 11-year follow-up after early open reduction and internal fixation. Arch Orthop Trauma Surg 2003;123:118-120. [DOI] [PubMed] [Google Scholar]
  • 50. Hayes PR, Klepps S, Bishop J, Cleeman E, Flatow EL. Posterior shoulder dislocation with lesser tuberosity and scapular spine fractures. J Shoulder Elbow Surg 2003;12:524-527. [DOI] [PubMed] [Google Scholar]
  • 51. Altay T, Oztürk H, Us RM, Günal I. Four-part posterior fracture–dislocations of the shoulder. Treatment by limited open reduction and percutaneous stabilization. Arch Orthop Trauma Surg 1999;119:35-38. [DOI] [PubMed] [Google Scholar]
  • 52. Fiorentino G, Cepparulo R, Lunini E, et al. Posterior shoulder fracture-dislocation: double approach treatment. Our experience. Acta Biomed 2016;87:184-190. [PubMed] [Google Scholar]
  • 53. Martens C, Hessels G. Bilateral posterior four-part fracture-dislocation of the shoulder. Acta Orthop Belg 1995;61:249-254. [PubMed] [Google Scholar]
  • 54. Finkelstein JA, Waddell JP, O’Driscoll SW, Vincent G. Acute posterior fracture dislocations of the shoulder treated with the Neer modification of the McLaughlin procedure. J Orthop Trauma 1995;9:190-193. [PubMed] [Google Scholar]
  • 55. Ogawa K, Yoshida A, Inokuchi W. Posterior shoulder dislocation associated with fracture of the humeral anatomic neck: treatment guidelines and long-term outcome. J Trauma 1999;46:318-323. [DOI] [PubMed] [Google Scholar]
  • 56. Page AE, Meinhard BP, Schulz E, Toledano B. Bilateral posterior fracture-dislocation of the shoulders: management by bilateral shoulder hemiarthroplasties. J Orthop Trauma 1995;9:526-529. [DOI] [PubMed] [Google Scholar]
  • 57. Oakes DA, McAllister DR. An atypical appearance of a posterior dislocation of the shoulder with a fracture of the proximal humerus. J Shoulder Elbow Surg 2001;10:182-185. [DOI] [PubMed] [Google Scholar]
  • 58. Ketenci IE, Duymus TM, Ulusoy A, et al. Bilateral posterior shoulder dislocation after electrical shock: A case report. Ann Med Surg (Lond) 2015;4:417-421. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 59. Ito H, Takayama A, Shirai Y. Posterior dislocation of the shoulder with a large fracture segment: a case report. J Shoulder Elbow Surg 2000;9:238-241. [PubMed] [Google Scholar]
  • 60. Toker G, Ozan F, Bora OA. Treatment of reverse Hill-Sachs lesion by autograft reconstruction. Acta Orthop Traumatol Turc 2012;46:398-402. [DOI] [PubMed] [Google Scholar]
  • 61. Riggenbach MD, Najarian RG, Bishop JY. Recurrent, locked posterior glenohumeral dislocation requiring hemiarthroplasty and posterior bone block with humeral head autograft. Orthopedics. 2012;35:e277-e282. [DOI] [PubMed] [Google Scholar]
  • 62. Mastrokalos DS, Panagopoulos GN, Galanopoulos IP, Papagelopoulos PJ. Posterior shoulder dislocation with a reverse Hill-Sachs lesion treated with frozen femoral head bone allograft combined with osteochondral autograft transfer. Knee Surg Sports Traumatol Arthrosc 2017;25:3285-3288. [DOI] [PubMed] [Google Scholar]
  • 63. Xu W, Huang L-X, Guo JJ, et al. Neglected posterior dislocation of the shoulder: A systematic literature review. J Orthop Translat 2015;3:89-94. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 64. Mouzopoulos G. The “Mouzopoulos” sign: a radiographic sign of posterior shoulder dislocation. Emerg Radiol 2010;17:317-320. [DOI] [PubMed] [Google Scholar]
  • 65. Robinson CM, Aderinto J. Posterior shoulder dislocations and fracture-dislocations. J Bone Joint Surg [Am] 2005;87:639-650. [DOI] [PubMed] [Google Scholar]
  • 66. Diklić I, Ganić Z, Stevanović V, Crnobarić A, Blagojević Z. [Treatment of unreduced posterior dislocations of the shoulder]. Acta Chir Iugosl 2006;53:63-67. [DOI] [PubMed] [Google Scholar]
  • 67. O’Neill D, Nair JR, Binymin KA. Simultaneous bilateral posterior fracture dislocation of the shoulders in a young man with unexpected severe vitamin D deficiency. Int J Gen Med 2012;5:399-402. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 68. Agarwal M, Khan WS, Trehan R, Syed AA, Giannoudis PV. Bilateral posterior fracture-dislocation of the shoulder presenting as a dissecting aneurysm of the thoracic aorta: an uncommon presentation of a rare injury. J Emerg Med 2008;35:265-268. [DOI] [PubMed] [Google Scholar]
  • 69. Liu X, Zhu Y, Lu Y, et al. Locked posterior shoulder dislocation associated with isolated fractures of the lesser tuberosity: a clinical study of 22 cases with a minimum of 2-year follow-up. J Orthop Trauma 2015;29:271-275. [DOI] [PubMed] [Google Scholar]
  • 70. Harvie P, Pollard TCB, Chennagiri RJ, Carr AJ. The use of outcome scores in surgery of the shoulder. J Bone Joint Surg [Br] 2005;87-B:151-154. [DOI] [PubMed] [Google Scholar]
  • 71. Fukuda A, Nishimura A, Kato K, Sudo A. Arthroscopically assisted minimally invasive plate osteosynthesis for posterior fracture-dislocation of the shoulder. J Orthop Sci 2014;19:194-197. [DOI] [PubMed] [Google Scholar]
  • 72. Ahmad CS, Wang VM, Sugalski MT, Levine WN, Bigliani LU. Biomechanics of shoulder capsulorrhaphy procedures. J Shoulder Elbow Surg 2005;14(S1):12S-18S. [DOI] [PubMed] [Google Scholar]

Articles from EFORT Open Reviews are provided here courtesy of Bioscientifica Ltd.

RESOURCES