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. 2018 Apr 11;49(5):1223–1231. doi: 10.1161/STROKEAHA.117.019687

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© 2018 The Authors.

Stroke is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 1. — Multiphasic pattern of mitochondrial respiratory decline after ischemia/reperfusion (I/R). A, Representative traces of mitochondrial oxygen consumption from sham (red) and 35 minute ischemia (black) in whole tissue homogenates. Addition of 1 mM cyanide (KCN) almost fully inhibited respiration in homogenates. B, Effect of I/R injury on ADP-stimulated oxygen consumption (sham: n=19; different time points n as indicated by the dots; Kruskal–Wallis test with Dunn multiple comparisons test; C) citrate synthase (C) and NADH oxidase (D) activity in the same preparations.