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. 2018 Apr 20;9(30):21495–21511. doi: 10.18632/oncotarget.25138

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Copyright: © 2018 Paulmurugan et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Therapeutic response of Ln229 cells stably expressing split-Renilla luciferase complementation sensor with different p53 variants (p53^wt, p53^Y220C, p53^G245S, p53^R282W) in response to the treatment of (A) Doxorubicin (1 μM), (B) Temozolomide (250 μM), (C) PhiKan083 (125 μM) and (D) SCH529074 (2.5 μM), for the change in cell viability and induced apoptosis.