I noticed that the article did not mention that (sometimes severe) polyneuropathies can occur even in stages of prediabetes (prediabetes, subclinical diabetes, and impaired glucose tolerance) (1). So what counts here is the diabetic diathesis, the predisposition, rather than the current blood sugar level. Every practicing neurologist knows this, and no recent research has shown a relationship in type 2 diabetes between good blood glucose control and neurological damage or symptoms. This differs from other sequelae of diabetes, such as vascular damage, for which blood sugar levels are very important. Why then is obtaining good blood glucose control still being repeatedly recommended (especially from internists) for the treatment of polyneuropathy? Ut aliquid fiat?
Footnotes
Conflict of interest statement
The author declares that no conflict of interest exists.
References
- 1.Sommer C, Geber C, Young P, Forst R, Birklein F, Schoser B. Polyneuropathies—etiology, diagnosis, and treatment options. Dtsch Arztebl Int. 2018;115:83–90. doi: 10.3238/arztebl.2018.083. [DOI] [PMC free article] [PubMed] [Google Scholar]
