Skip to main content
Food Science and Biotechnology logoLink to Food Science and Biotechnology
. 2017 Apr 30;26(2):501–505. doi: 10.1007/s10068-017-0069-3

Comparison of trans-fatty acids on proliferation and migration of vascular smooth muscle cells

Haewon Kim 1, Chang Hee Jeong 1, Hong Gu Lee 2, Sung Gu Han 1,
PMCID: PMC6049441  PMID: 30263571

Abstract

Consumption of trans-fatty acids has been linked to an increased risk of cardiovascular diseases, such as atherosclerosis. Milk and dairy products contain trans-fatty acids, such as transvaccenic acid (TVA) and conjugated linoleic acid (CLA). Although artificially hydrogenated trans-fatty acids (e.g., elaidic acid (EA)) are known to induce atherosclerosis, it is unclear whether ruminant trans-fats, such as TVA, are associated with such diseases. Therefore, we investigated the effects of TVA on vascular smooth muscle cells (VSMCs). VSMCs were treated with TVA, CLA, and EA at 0-100 μM for 24 h. Cell proliferation and migration increased upon treatment with EA, not with TVA and CLA. EA increased protein expression of proliferation-associated proteins (cyclin-dependent kinase 4 (CDK4) and cyclin D1), while TVA and CLA decreased CDK4 expression. These results suggest that TVA is not as risky as other trans-fatty acids such as EA in the vascular system.

Keywords: trans-vaccenic acid, elaidic acid, vascular smooth muscle cell, proliferation, migration

References

  • 1.Aluganti Narasimhulu C, Fernandez-Ruiz I, Selvarajan K, Jiang X, Sengupta B, Riad A, Parthasarathy S. Atherosclerosis—do we know enough already to prevent it? Curr. Opin. Pharmacol. 2016;27:92–102. doi: 10.1016/j.coph.2016.02.006. [DOI] [PubMed] [Google Scholar]
  • 2.Lacolley P, Regnault V, Nicoletti A, Li Z, Michel JB. The vascular smooth muscle cell in arterial pathology: A cell that can take on multiple roles. Cardiovasc. Res. 2012;95:194–204. doi: 10.1093/cvr/cvs135. [DOI] [PubMed] [Google Scholar]
  • 3.Glass CK, Witztum JL. Atherosclerosis: The road ahead. Cell. 2001;104:503–516. doi: 10.1016/S0092-8674(01)00238-0. [DOI] [PubMed] [Google Scholar]
  • 4.Gomez D, Owens GK. Smooth muscle cell phenotypic switching in atherosclerosis. Cardiovasc. Res. 2012;95:156–164. doi: 10.1093/cvr/cvs115. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 5.Tanner FC, Greutert H, Barandier C, Frischknecht K, Luscher TF. Different cell cycle regulation of vascular smooth muscle in genetic hypertension. Hypertension. 2003;42:184–188. doi: 10.1161/01.HYP.0000082360.65547.7C. [DOI] [PubMed] [Google Scholar]
  • 6.Lim S, Kaldis P. Cdks, cyclins and CKIs: Roles beyond cell cycle regulation. Development. 2013;140:3079–3093. doi: 10.1242/dev.091744. [DOI] [PubMed] [Google Scholar]
  • 7.Kim TJ, Lim Y, Kim DW, Kwon JS, Son JH, Jin YR, Son DJ, Jung JC, Avery MA, Hong JT, Yun YP. Epothilone D, a microtubule-stabilizing compound, inhibits neointimal hyperplasia after rat carotid artery injury by cell cycle arrest via regulation of G1-checkpoint proteins. Vasc. Pharmacol. 2007;47:229–237. doi: 10.1016/j.vph.2007.06.009. [DOI] [PubMed] [Google Scholar]
  • 8.de Caterina R. n-3 fatty acids in cardiovascular disease. New Engl. J. Med. 2011;364:2439–2450. doi: 10.1056/NEJMra1008153. [DOI] [PubMed] [Google Scholar]
  • 9.Gebauer SK, Destaillats F, Mouloungui Z, Candy L, Bezelgues JB, Dionisi F, Baer DJ. Effect of trans fatty acid isomers from ruminant sources on risk factors of cardiovascular disease: Study design and rationale. Contemp. Clin. Trials. 2011;32:569–576. doi: 10.1016/j.cct.2011.03.012. [DOI] [PubMed] [Google Scholar]
  • 10.Wang T, Lee HG. Advances in research on cis-9, trans-11 conjugated linoleic acid: A major functional conjugated linoleic acid isomer. Crit. Rev. Food Sci. 2015;55:720–731. doi: 10.1080/10408398.2012.674071. [DOI] [PubMed] [Google Scholar]
  • 11.Bassett CM, Edel AL, Patenaude AF, McCullough RS, Blackwood DP, Chouinard PY, Paquin P, Lamarche B, Pierce GN. Dietary vaccenic acid has antiatherogenic effects in LDLr-/-mice. J. Nutr. 2010;140:18–24. doi: 10.3945/jn.109.105163. [DOI] [PubMed] [Google Scholar]
  • 12.Iwata NG, Pham M, Rizzo NO, Cheng AM, Maloney E, Kim F. Trans fatty acids induce vascular inflammation and reduce vascular nitric oxide production in endothelial cells. PLoS ONE. 2011;6:e29600. doi: 10.1371/journal.pone.0029600. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 13.Abdelmagid SA, Clarke SE, Wong J, Roke K, Nielsen D, Badawi A, El-Sohemy A, Mutch DM, Ma DW. Plasma concentration of cis9trans11 CLA in males and females is influenced by SCD1 genetic variations and hormonal contraceptives: A cross-sectional study. Nutr. Metabolism. 2013;10:50. doi: 10.1186/1743-7075-10-50. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 14.Song J, Kwon N, Lee MH, Ko YG, Lee JH, Kim OY. Association of serum phospholipid PUFAs with cardiometabolic risk: Beneficial effect of DHA on the suppression of vascular proliferation/inflammation. Clin. Biochem. 2014;47:361–368. doi: 10.1016/j.clinbiochem.2014.01.005. [DOI] [PubMed] [Google Scholar]
  • 15.Yun MR, Lee JY, Park HS, Heo HJ, Park JY, Bae SS, Hong KW, Sung SM, Kim CD. Oleic acid enhances vascular smooth muscle cell proliferation via phosphatidylinositol 3-kinase/Akt signaling pathway. Pharmacol. Res. 2006;54:97–102. doi: 10.1016/j.phrs.2006.03.001. [DOI] [PubMed] [Google Scholar]
  • 16.Wang P, Henning SM, Heber D. Limitations of MTT and MTS-based assays for measurement of antiproliferative activity of green tea polyphenols. PLoS ONE. 2010;5:e10202. doi: 10.1371/journal.pone.0010202. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 17.Delbosc S, Glorian M L, Port AS, Bereziat G, Andreani M, Limon I. The benefit of docosahexanoic acid on the migration of vascular smooth muscle cells is partially dependent on Notch regulation of MMP-2/-9. Am. J. Pathol. 2008;172:1430–1440. doi: 10.2353/ajpath.2008.070951. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 18.Zhang Y, Liu C, Zhu L, Jiang X, Chen X, Qi X, Liang X, Jin S, Zhang P, Li Q, Wang D, Liu X, Zeng K, Zhang J, Xiang Y, Zhang CY. PGC-1alpha inhibits oleic acid induced proliferation and migration of rat vascular smooth muscle cells. PLoS ONE. 2007;2:e1137. doi: 10.1371/journal.pone.0001137. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 19.Ferguson JE, Patterson C. Break the cycle: The role of cell-cycle modulation in the prevention of vasculoproliferative diseases. Cell Cycle. 2003;2:211–219. doi: 10.4161/cc.2.3.366. [DOI] [PubMed] [Google Scholar]
  • 20.Casimiro MC, Velasco-Velazquez M, Aguirre-Alvarado C, Pestell RG. Overview of cyclins D1 function in cancer and the CDK inhibitor landscape: Past and present. Expert Opin. Inv. Drug. 2014;23:295–304. doi: 10.1517/13543784.2014.867017. [DOI] [PubMed] [Google Scholar]
  • 21.Park SL, Won SY, Song JH, Kambe T, Nagao M, Kim WJ, Moon SK. EPO gene expression promotes proliferation, migration and invasion via the p38MAPK/AP-1/MMP-9 pathway by p21WAF1 expression in vascular smooth muscle cells. Cell. Signal. 2015;27:470–478. doi: 10.1016/j.cellsig.2014.12.001. [DOI] [PubMed] [Google Scholar]
  • 22.Park ES, Lee KP, Jung SH, Lee DY, Won KJ, Yun YP, Kim B. Compound K, an intestinal metabolite of ginsenosides, inhibits PDGF-BB-induced VSMC proliferation and migration through G1 arrest and attenuates neointimal hyperplasia after arterial injury. Atherosclerosis. 2013;228:53–60. doi: 10.1016/j.atherosclerosis.2013.02.002. [DOI] [PubMed] [Google Scholar]
  • 23.Ringseis R, Gahler S, Eder K. Conjugated linoleic acid isomers inhibit plateletderived growth factor-induced NF-kappaB transactivation and collagen formation in human vascular smooth muscle cells. Eur. J. Nutr. 2008;47:59–67. doi: 10.1007/s00394-008-0697-8. [DOI] [PubMed] [Google Scholar]
  • 24.Suh SJ, Jin UH, Kim SH, Chang HW, Son JK, Lee SH, Son KH, Kim CH. Ochnaflavone inhibits TNF-alpha-induced human VSMC proliferation via regulation of cell cycle, ERK1/2, and MMP-9. J. Cell. Biochem. 2006;99:1298–1307. doi: 10.1002/jcb.20912. [DOI] [PubMed] [Google Scholar]
  • 25.Bryk D, Zapolska-Downar D, Malecki M, Hajdukiewicz K, Sitkiewicz D. Trans fatty acids induce a proinflammatory response in endothelial cells through ROS-dependent nuclear factor-kappaB activation. J. Physiol. Pharmacol. 2011;62:229–238. [PubMed] [Google Scholar]

Articles from Food Science and Biotechnology are provided here courtesy of Springer

RESOURCES