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. Author manuscript; available in PMC: 2018 Oct 13.
Published in final edited form as: Cell Rep. 2018 Sep 4;24(10):2795–2797. doi: 10.1016/j.celrep.2018.08.050

Loss of Abhd5 Promotes Colorectal Tumor Development and Progression by Inducing Aerobic Glycolysis and Epithelial-Mesenchymal Transition

Juanjuan Ou, Hongming Miao, Yinyan Ma, Feng Guo, Jia Deng, Xing Wei, Jie Zhou, Ganfeng Xie, Hang Shi, Bingzhong Xue, Houjie Liang *, Liqing Yu *
PMCID: PMC6186214  NIHMSID: NIHMS988145  PMID: 30184511

In the originally published version of this article, Figures 1C and 5A included incorrect immunoblots for GAPDH, and Figure 7F included an incorrect immunoblot for Snail. Figure 6K contained an incorrectly labeled internal control—the label for the internal control should be GAPDH, not β-actin. These errors have been corrected in the figures shown below. These corrections do not change the conclusions of the study.

Figure 1.

Figure 1.

Abhd5 Suppresses EMT and Growth Advantage of CRC Cells

Figure 5.

Figure 5.

Abhd5 Deficiency Induces EMT via Inactivating p53

Figure 7.

Figure 7.

Aerobic Glycolysis Induces p53 Suppression via Inactivating AMPK in Abhd5-Deficient Cells

Figure 6.

Figure 6.

Abhd5 Deficiency-Induced EMT in CRC Cells Depends on Glucose Uptake and Aerobic Glycolysis

The authors regret these errors.

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