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. 2018 Nov 7;13(11):e0207325. doi: 10.1371/journal.pone.0207325

Correction: Biologically anchored knowledge expansion approach uncovers KLF4 as a novel insulin signaling regulator

Annamalai Muthiah, Morgan S Angulo, Natalie N Walker, Susanna R Keller, Jae K Lee
PMCID: PMC6221346  PMID: 30403750

S2 Table is incomplete. The bottom part of S2 Table is missing. The full S2 Table can be viewed below.

Supporting information

S2 Table. L0 and L1 genes.

L0 represents genes that were differentially expressed between DW16 and DC16 adipocytes. L1 represents genes in L0 for which expression profiles significantly correlated with expression of insulin signaling pathway genes (Lpath) in adipocytes using data for all four conditions DC8, DW8, DC16 and DW16 (marked L1 in table).

(PDF)

Click here for additional data file. (1.2MB, pdf)

Reference

  • 1.Muthiah A, Angulo MS, Walker NN, Keller SR, Lee JK (2018) Biologically anchored knowledge expansion approach uncovers KLF4 as a novel insulin signaling regulator. PLoS ONE 13(9): e0204100 10.1371/journal.pone.0204100 [DOI] [PMC free article] [PubMed] [Google Scholar]

Associated Data

This section collects any data citations, data availability statements, or supplementary materials included in this article.

Supplementary Materials

S2 Table. L0 and L1 genes.

L0 represents genes that were differentially expressed between DW16 and DC16 adipocytes. L1 represents genes in L0 for which expression profiles significantly correlated with expression of insulin signaling pathway genes (Lpath) in adipocytes using data for all four conditions DC8, DW8, DC16 and DW16 (marked L1 in table).

(PDF)

Click here for additional data file. (1.2MB, pdf)

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