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. 2018 Dec 28;9(102):37807. doi: 10.18632/oncotarget.26547

Correction: Aryl hydrocarbon receptor regulates histone deacetylase 8 expression to repress tumor suppressive activity in hepatocellular carcinoma

Li-Ting Wang 1, Shyh-Shin Chiou 2,3, Chee-Yin Chai 4, Edward Hsi 5, Shen-Nien Wang 1,6,7, Shau-Ku Huang 8,9, Shih-Hsien Hsu 1,9,10
PMCID: PMC6340884  PMID: 30701034

This article has been corrected: Figures 2A and 3B were displayed incorrectly. The proper figure images are given below. The authors declare that these corrections do not change the results or conclusions of this paper.

Figure 2. Ectopic HDAC8 expression showed a high correlation with AHR in hepatocellular carcinoma (HCC).

Figure 2

A. Overexpression of HDAC8 protein was detected in HCC patients with high AHR mRNA expression.

Figure 3. AHR directly activated cellular HDAC8 expression via the AHR–ARNT complex in hepatoma cells.

Figure 3

B. AHR activation induced by TCDD increased cellular CYP1B1 and HDAC8 protein levels in hepatoma cells.

Original article: Oncotarget. 2017; 8:7489-7501. https://doi.org/10.18632/oncotarget.9841


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