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. 2018 Nov 28;39:377–387. doi: 10.1016/j.ebiom.2018.11.040

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© 2018 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 1 — GSK3β regulates GluA1 phosphorylation and HCN4 expression in the mice brain. Hippocampi from wildtype (n = 6) and GSK3β[S9A] (n = 5) mice were isolated and lysed. (a) Western blot was used to determine the levels of GluA1, P-GluA1 (S845), P-GluA1 (S831), GluA2, GluN1, EEAT2, ErbB4, TrkB, HCN4, and GAD67. Tubulin is shown as a loading control. (b) Quantitative Western blot analysis results. Error bars indicate SEM. *p < .05, **p < .01, ***p < .001 (two-way ANOVA significant interaction effect, p = 0.0001, ***; two-way ANOVA with Bonferroni correction).