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. 2019 Mar 1;19:189. doi: 10.1186/s12885-019-5378-x

Correction to: miR-18a reactivates the Epstein-Barr virus through defective DNA damage response and promotes genomic instability in EBV-associated lymphomas

Pengfei Cao 1,2,#, Meili Zhang 1,2,5,#, Lujuan Wang 1,2,3, Buqing Sai 1,2,3, Jiuqi Tang 2, Zhaohui Luo 2, Cijun Shuai 4, Liyang Zhang 5, Zheng Li 1,2,3, Yanjin Wang 6, Guiyuan Li 1,2,3, Juanjuan Xiang 1,2,3,
PMCID: PMC6396492  PMID: 30823908

Following publication of the original article [1], the authors reported that they had supplied the incorrect figure for publication.

Cao et al. BMC Cancer (2018) 18:1293.

https://doi.org/10.1186/s12885-018-5205-9

Following publication of the original article [1], the authors reported that they had supplied the incorrect figure for publication. The correct figure is displayed below. The authors apologise for the error.

Fig. 2.

Fig. 2

miR-18a promotes cell proliferation in EBV-positive lymphoma cells. a miR-18a promoted tumor cell growth in vitro in EBV-positive lymphoma cell lines. CCK-8 cell viability assay was performed after the transfection of miR-18a mimics or miR-18a inhibitor into EBV-positive lymphoma cell lines (P3HR-1, Raji, EBV infected BJAB) and the EBV-negative lymphoma cell line BJAB. The data represented the mean values of five repeats. The data are shown as the means±SD (Student T-test, *p < 0.05). b Flow cytometry analysis of the cell cycle after the transfection of miR-18a. c Cell cycle distribution of cells in S phase. The data were presented as the means±SD of four replicates (Student T-test, *p < 0.05; **p < 0.01)

Reference

  • 1.Cao et al. BMC Cancer (2018) .miR-18a reactivates the Epstein-Barr virus through defective DNA damage response and promotes genomic instability in EBV-associated lymphomas. BMC Cancer 18:1293. 10.1186/s12885-018-5205-9 [DOI] [PMC free article] [PubMed]

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