In the article titled “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating Treg/Th17 Expression” [1], reference [4], J. A. Carlsson et al. [2], was incorrectly included in the reference list and should be replaced with M. F. Roelofs et al. [3].
References
- 1.Dai Q., Li J., Yun Y., Wang J. Toll-like receptor 4–myeloid differentiation primary response gene 88 pathway is involved in the shikonin treatment of CIA by regulating Treg/Th17 expression. Evidence-Based Complementary and Alternative Medicine. 2018;2018:9. doi: 10.1155/2018/2428546.2428546 [DOI] [PMC free article] [PubMed] [Google Scholar]
- 2.Carlsson J. A., Wold A. E., Sandberg A.-S., Östman S. M. The polyunsaturated fatty acids arachidonic acid and docosahexaenoic acid induce mouse dendritic cells maturation but reduce T-cell responses in vitro. PLoS ONE. 2015;10(11) doi: 10.1371/journal.pone.0143741.e0143741 [DOI] [PMC free article] [PubMed] [Google Scholar]
- 3.Roelofs M. F., Joosten L. A. B., Abdollahi-Roodsaz S., et al. The expression of toll-like receptors 3 and 7 in rheumatoid arthritis synovium is increased and costimulation of toll-like receptors 3, 4, and 7/8 results in synergistic cytokine production by dendritic cells. Arthritis & Rheumatology. 2005;52(8):2313–2322. doi: 10.1002/art.21278. [DOI] [PubMed] [Google Scholar]
