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. 2019 Mar 28;8(6):e1583547. doi: 10.1080/2162402X.2019.1583547

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© 2019 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited, and is not altered, transformed, or built upon in any way.

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Figure 2. — Pathological comparison of APC and non-APC HB.

(a) Histological component distribution assessed by pathological reviewing of APC and non-APC HB. (b) Representative HES and IHC staining of β-catenin (β-cat) and GS within the tumors of APC and non-APC HB patients (high magnification). Lymphocytes without staining are seen in APC-HB. (c) Levels of AXIN2 and LGR5 mRNAs assessed by qRT-PCR in APC and non-APC HB as well as in adult and pediatric non-tumor liver (NTL), expressed as a fold-change compared to adult NTL. The non-APC and non-CTNNB1-mutated HB sample is marked in red. * P < .05 (Wilcoxon test, without taking into account the non-APC CTNNB1 NM tumor).