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. 2019 Jun 12;12:149. doi: 10.3389/fnmol.2019.00149

Corrigendum: Gap Junctions in A8 Amacrine Cells Are Made of Connexin36 but Are Differently Regulated Than Gap Junctions in AII Amacrine Cells

Shubhash C Yadav 1, Stephan Tetenborg 1, Karin Dedek 1,2,*
PMCID: PMC6582223  PMID: 31249510

In the original article, there was a mistake in Figure 5 as published. The line scans in Figures 5E,F depicting the channel intensity of the respective ROIs in Figures 5B,C, were swapped by mistake. The corrected Figure 5 appears below.

Figure 5.

Figure 5

Co-localization of A8 gap junctions with bipolar cell terminals in vertical sections. (A–C) Single retinal slices of Ier5-EGFP (A8 cell) mouse stained with Cx36 and bipolar cell markers: VGluT1 (A), secretagogin [(B), SCGN], and synaptotagmin-2 [(C), Syt2]. Square white boxes in (A–C) are the selected ROIs shown in (A'–C””). Arrows denote co-localization of all the three channels which is also represented in the normalized intensity plots (D–F). (D–F) Intensity plot for three channels, corresponding to (A–C). The respective inset represents the single scan overlay of the three channels. The plot denotes normalized pixel intensity of three channels in y-axis, and the x-axis represents the relative distance of peak intensities of the three individual channels. Scale bar: (A–C), 10 μm; (A'–C””), 2.5 μm.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.


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