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. 2019 Mar 27;42(4):1287–1300. doi: 10.1007/s10753-019-00988-1

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© The Author(s) 2019

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 2 — The expression of nephrin, podocin, FKN, wnt-4, and β-catenin protein in the kidneys of mice. Renal tissue sections of mice were stained by IHC (original magnification, × 400). a Nephrin and podocin; b FKN and wnt-4; c β-catenin. a Control, b LPS-induced, c anti-FKN, d XAV, e LPS + anti-FKN, f LPS + XAV, and g LPS + anti-FKN + XAV mice.

Fig. 2 — The expression of nephrin, podocin, FKN, wnt-4, and β-catenin protein in the kidneys of mice. Renal tissue sections of mice were stained by IHC (original magnification, × 400). a Nephrin and podocin; b FKN and wnt-4; c β-catenin. a Control, b LPS-induced, c anti-FKN, d XAV, e LPS + anti-FKN, f LPS + XAV, and g LPS + anti-FKN + XAV mice.