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Clinical Cardiology logoLink to Clinical Cardiology
. 2006 Dec 5;29(4):155–160. doi: 10.1002/clc.4960290406

Amount of left ventricular hypertrophy determines the plasma N‐terminal pro‐brain natriuretic peptide level in patients with hypertrophic cardiomyopathy and normal left ventricular ejection fraction

Seon Woon Kim 1, Seung Woo Park 1,, Seong‐Hoon Lim 1, Sung Uk Kwon 1, Yu Jeong Hoi 1, Man Ki Park 1, Sang‐Chol Lee 1, Sang Hoon Lee 1, Jeong Euy Park 1, Eun‐Seok Jeon 1
PMCID: PMC6654112  PMID: 16649724

Abstract

Background: N‐terminal pro‐brain natriuretic peptide (NT‐proBNP) is increased in patients with hypertrophic cardiomyopathy (HCM); however, the determinants of NT‐proBNP level have not been clarified in HCM.

Hypothesis: This study was performed to determine the relationship between NT‐proBNP levels and various echocardiographic variables of patients with HCM and normal left ventricular ejection fraction (LVEF).

Methods: We assessed plasma NT‐proBNP levels and echocardiographic variables of 36 patients (19 men, 58 ± 14 years) with HCM and an LVEF of ≥ 55%. Echocardiographic variables measured were LV wall thickness, end‐diastolic LV internal dimension (LVIDd) and volume (LVEDV), LV mass, and LV mass index (LV mass/body surface area, LVMI). Left ventricular outflow tract pressure gradient, transmitral E and A velocities, deceleration time (DT) of the transmitral E wave, and septal annular E' velocity were measured by Doppler technique. The relationship between echocardiographic variables and plasma NT‐proBNP level was analyzed.

Results: The plasma NT‐proBNP level was 775.2 ± 994.2 pg/ml (range 33.1‐729.0 pg/ml). It showed positive correlations with LV end‐diastolic septal thickness (r = 0.39, p = 0.010) and LVMI (r = 0.27, p = 0.050), while it revealed negative correlations with LVIDd (r = −0.44, p = 0.004), LVEDV (r= −0.44, p = 0.004) and DT(r= −0.31, p = 0.034). The NT‐proBNP level was higher in the patients with than in those without LV diastolic dysfunction (p = 0.03 3) and was independently related to LVIDd (p = 0.001), LVMI (p = 0.006) and DT (p = 0.031) by multivariate analysis.

Conclusion: In patients with HCM and normal LVEF, the amount of LV hypertrophy and LV diastolic dysfunction may exert a significant role in determining plasma NT‐proBNP level.

Keywords: hypertrophic cardiomyopathy, natriuretic peptide, hypertrophy, diastolic dysfunction

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