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. 2009 Feb 3;20(4):341–344. doi: 10.1002/clc.4960200408

Factors influencing heart rate variability in patients with severe aortic valve disease

Jens Jung 1,, Armin Heisel 1, Bernhard Butz 1, Roland Fries 1, Hermann Schieffer 1, Dietmar Tscholl 2, Hans‐Joachim Schäfers 2
PMCID: PMC6655940  PMID: 9098592

Abstract

Background and hypothesis: Heart rate variability (HRV) is an accepted tool for the assessment of cardiovascular autonomic tone. There are no sufficient data concerning its application to patients with severe aortic valve disease (AVD) requiring cardiac surgery.

Methods: It was the aim of this study to examine HRV and its physiologic correlates in patients with severe aortic valve disease requiring cardiac surgery. The correlates of time domain indices of HRV obtained from 24‐h Holter electrocardiographic recordings were analyzed in 36 consecutive patients (23 men and 13 women, mean age 62 ± 11 years) with AVD prior to cardiac surgery (aortic stenosis: 17 patients, aortic valve regurgitation: 3 patients, combined aortic valve disease: 16 patients).

Results: Low values of HRV were found in the entire study group: SDNN 96.8 ± 30.9 ms, SDNNI 39.3 ± 14.4 ms, SDANN 86 ± 28.9 ms, and RMSSD 30 ± 18.1 ms. In a univariate analysis, there was no significant correlation between the time domain measures of HRV and age, gender, medication, left ventricular ejection fraction, peak aortic pressure gradient, fraction of aortic valve regurgitation, and left ventricular mass assessed by echocardiography. Patients in advanced functional classes of heart failure [New York Heart Association (NYHA) III or IV] had significantly lower values for SDNN (83.8 ± 33.6 vs. 107.3 ± 24.7 ms; p<0.05) and SDANN (72.7 ± 29.4 vs. 96.6 ± 24.3 ms; p<0.05) than patients in NYHA class I or II. Reassessment of HRV 1 week after aortic valve replacement was performed in 17 patients and showed a significant further decrease of SDNN (102.4 ± 29.7 vs. 61.5 ± 23.5 ms; p<0.001), SDNNI (40.7 ± 13.6 vs. 23.4 ± 12.4 ms; p<0.001) and SDANN (91.8 ±29.2 vs. 54.2 ± 22.8 ms;p<0.001).

Conclusion: Patients with AVD requiring cardiac surgery reveal reduced time domain indices of HRV. This observation is pronounced in patients with a progressed clinical class of heart failure, whereas hemodynamic and echocardiographic parameters seem to have no significant influence on HRV parameters in this population. In addition, there is evidence of a further reduction of HRV time domain indices 1 week after uncomplicated aortic valve replacement.

Keywords: autonomic nervous system, heart rate variability, aortic valve disease

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References

  • 1. Kleiger RE, Miller JP, Bigger JT, Moss AJ, and the Multicenter Post‐Infarction Research Group : Decreased heart rate variability and its association with increased mortality after acute myocardial infarction. Am J Cardiol 1987; 59: 256–262. [DOI] [PubMed] [Google Scholar]
  • 2. Odemuyiwa O, Malik M, Farrell T, Bashir Y, Poliniecki J, Camm AJ: Comparison of the predictive characteristics of heart rate variability index and left ventricular ejection fraction for all‐cause mortality, arrhythmic events and sudden death after acute myocardial infarction. Am J Cardiol 1991; 688: 434–439. [DOI] [PubMed] [Google Scholar]
  • 3. Casolo G, Balli E, Taddei T, Amuhasi J, Gori C: Decreased spontaneous heart rate variability on congestive heart failure. Am J Cardiol 1989; 64: 1162–1167. [DOI] [PubMed] [Google Scholar]
  • 4. Kienzle MG, Ferguson DW, Birkett CL, Myers GA, Berg WJ, Mariano DJ: Clinical hemodynamic and sympathetic neural correlates of heart rate variability in congestive heart failure. Am J Cardiol 1992; 69: 482–485. [DOI] [PubMed] [Google Scholar]
  • 5. Panina G, Khot UN, Nunziata E, Cody RJ, Binkley PF: Role of spectral measures of heart rate variability as markers of disease progression in patients with chronic congestive heart failure not treated with angiotensin‐converting enzyme inhibitors. Am Heart J 1996; 131: 153–157. [DOI] [PubMed] [Google Scholar]
  • 6. Santinga JT, Kirsh MM, Flora JD, Brymer JF: Factors relating to sudden death in patients having aortic valve replacement. Ann Thorac Surg 1980; 29: 249–253. [DOI] [PubMed] [Google Scholar]
  • 7. Devereux RB, Alonso DR, Lutas EM, Gottlieb GJ, Campo E, Sachs I, Reichek N: Echocardiographic assessment of left ventricular hypertrophy: Comparison to necropsy findings. Am J Cardiol 1986; 7: 450–458. [DOI] [PubMed] [Google Scholar]
  • 8. Coumel P, Hermida JS, Wennerblöm B, Leenhardt A, Maison‐Blanche P, Cauchemez B: Heart rate variability on left ventricular hypertrophy and heart failure, and the effects of beta‐blockade. Eur Heart J 1991; 12: 412–422. [DOI] [PubMed] [Google Scholar]
  • 9. Stein KM, Borer JS, Hochreiter C, Okin PM, Herrold EM, Devereux RB, Kligfield P: Prognostic value and physiological correlates of heart rate variability in chronic severe mitral regurgitation. Circulation 1993; 88: 127–135. [DOI] [PubMed] [Google Scholar]
  • 10. Binkley PF, Haas GJ, Starling RC, Nunziata E, Hatton PA, Leier CV, Cody RJ: Sustained augmentation of parasympathetic tone with angiotensin‐converting enzyme inhibition in patients with congestive heart failure. J Am Coll Cardiol 1993; 21: 655–661. [DOI] [PubMed] [Google Scholar]
  • 11. Hogue CW, Stein PK, Apostolidou I, Lappas DG, Kleiger RE: Alterations in temporal patterns of heart rate variability after coronary artery bypass graft surgery. Anesthesiology 1994; 81: 1356–1364. [DOI] [PubMed] [Google Scholar]

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