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Journal of Clinical Laboratory Analysis logoLink to Journal of Clinical Laboratory Analysis
. 2004 Sep 3;18(5):276–279. doi: 10.1002/jcla.20037

Urokinase gene 3′‐UTR T/C polymorphism is associated with oral cancer

Ming‐Hsui Tsai 1, Wen‐Chi Chen 2,3, Huey‐Yi Chen 2,4, Fuu‐Jen Tsai 2,5,
PMCID: PMC6808149  PMID: 15356878

Abstract

Urokinase is thought to be involved in the formation of oral cancer, although there is a lack of genetic evidence. Our aim was to study single nucleotide polymorphisms in order to investigate the possibility. A total of 130 oral cancer patients and 105 controls were studied. Polymerase chain reaction (PCR) based restriction analysis was used to identify the C/T polymorphism of the urokinase gene, which is located on the 3′‐untranslated region (3′‐UTR) of chromosome 10. There was a significant difference in the distribution of the urokinase gene 3′‐UTR C/T polymorphism frequency between cancer patients and the normal control group (P<0.05). The “T” allele was prominent in the cancer group. The odds ratio for the risk of the “T” allele in cancer patients was 2.71 (95% CI=1.325∼5.562). The cancer patients were further categorized according to gender and whether or not they were habitual smokers or betel nut chewers. These clinical parameters were then compared with tumor cell differentiation and tumor progression. No significant differences were found. Therefore, the urokinase gene 3′‐UTR “T” allele is associated with oral cancer and may play a role in oral cancer formation. However, we did not find the relationship between tumor progression and this polymorphism. J. Clin. Lab. Anal. 18:276–279, 2004. © 2004 Wiley‐Liss, Inc.

Keywords: urokinase, oral cancer, single nucleotide polymorphism

REFERENCES

  • 1. Clayman G, Wang SW, Nicolson GL, et al. Regulation of urokinase‐type plasminogen activator expression in squamous‐cell carcinoma of the oral cavity. Int J Cancer 1993;54:73–80. [DOI] [PubMed] [Google Scholar]
  • 2. Ljungner H, Bjorlin G, Astedt B. Immunological identification of plasminogen activators in normal and malignant tissues of the oral cavity in man. Int J Oral Surg 1984;13:334–338. [DOI] [PubMed] [Google Scholar]
  • 3. Nozaki S, Endo Y, Kawashiri S, et al. Immunohistochemical localization of a urokinase‐type plasminogen activator system in squamous cell carcinoma of the oral cavity: association with mode of invasion and lymph node metastasis. Oral Oncol 1998;34:58–62. [DOI] [PubMed] [Google Scholar]
  • 4. Ko YC, Huang YL, Lee CH, Chen MJ, Lin LM, Tsai CC. Betel quid chewing, cigarette smoking, and alcohol consumption related to oral cancer in Taiwan. J Oral Pathol Med 1995;24:450–453. [DOI] [PubMed] [Google Scholar]
  • 5. Jayant K, Balakrishnan V, Sanghvi LD, Jussawala DJ. Quantification of the role of smoking and chewing habits in oral, pharynx, and esophageal cancer. Br J Cancer 1977;35:232–235. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 6. Jeng JH, Kuo ML, Hahn LJ, Kuo MYP. Genotoxic and non‐genotoxic effects of betel quid ingredients on oral mucosal fibroblasts in vitro. J Dent Res 1994;73:1043–1049. [DOI] [PubMed] [Google Scholar]
  • 7. Das BR, Nagpal JK. Understanding the biology of oral cancer. Med Sci Monit 2002;8:RA258–RA267. [PubMed] [Google Scholar]
  • 8. Tripputi P, Blasi F, Verde P, Cannizzaro LA, Emanuel BS, Croce CM. Human urokinase gene is located on the long arm of chromosome 10. Proc Nat Acad Sci USA 1985;82:4448–4452. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 9. Conne B, Berczy M, Belin D. Detection of polymorphism in the human urokinase‐type plasminogen activator gene. Thromb Hemost 1997;77:434–435. [PubMed] [Google Scholar]
  • 10. Yasuda T, Sakata Y, Kitamura K, Morita M, Ishida T. Localization of plasminogen activators and their inhibitor in squamous cell carcinomas of the head and neck. Head Neck 1997;19:611–616. [DOI] [PubMed] [Google Scholar]
  • 11. Barlow Y, Southam JC. Plasminogen activators in normal and malignant oral epithelium in vivo and in vitro. Arch Oral Biol 1992;37:749–756. [DOI] [PubMed] [Google Scholar]
  • 12. Epstein JB, Zhang L, Rosin M. Advances in the diagnosis of oral premalignant and malignant lesions. J Can Dent Assoc 2002;68:617–621. [PubMed] [Google Scholar]
  • 13. Sundqvist K, Liu Y, Nair J, Bartsch H, Arvidson K, Grafstrom RC. Cytotoxic and genotoxic effects of areca nut‐related compounds in cultured human buccal epithelial cells. Cancer Res 1989;49:5294–5298. [PubMed] [Google Scholar]
  • 14. Chang YC, Hu CC, Tseng TH, Tai KW, Lii CK, Chou MY. Synergistic effects of nicotine on arecholine‐induced cytoxicity in human bucosal fibroblasts. J Oral Pathol Med 2001;30:458–464. [DOI] [PubMed] [Google Scholar]

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