Skip to main content
JAMA Network logoLink to JAMA Network
. 2019 Nov 22;2(11):e1916018. doi: 10.1001/jamanetworkopen.2019.16018

Association Between Parental Educational Attainment and Youth Outcomes and Role of Race/Ethnicity

Shervin Assari 1,, Cleopatra H Caldwell 2,3, Mohsen Bazargan 1,4
PMCID: PMC6902825  PMID: 31755951

Key Points

Question

Is parental educational attainment associated with the same amount of health benefits for various ethnic groups?

Findings

In this cross-sectional study of 10 619 youth aged 12 to 17 years, parental educational attainment was associated with fewer health benefits for Hispanic youth compared with their non-Hispanic counterparts.

Meaning

These findings suggest that elimination of ethnic health disparities requires more than equalizing socioeconomic resources across ethnic groups.


This cross-sectional study uses data from the Population Assessment of Tobacco and Health survey to explore racial and ethnic differences in the associations between parental educational attainment and youth outcomes among US adolescents.

Abstract

Importance

The concept of minorities’ diminished returns refers to the smaller protective effects of educational attainment for racial and ethnic minority groups compared with those for majority groups.

Objective

To explore racial and ethnic differences in the associations between parental educational attainment and youth outcomes among US adolescents.

Design, Setting, and Participants

A cross-sectional study was performed of 10 619 youth aged 12 to 17 years who were participants at wave 1 of the Population Assessment of Tobacco and Health (PATH) study, a nationally representative survey, in 2013. Data analysis was performed from August to October 2019.

Main Outcomes and Measures

The dependent variables were youth tobacco dependence, aggression, school performance, psychological distress, and chronic medical conditions. The independent variable was parental educational attainment. Age and sex of the adolescents and marital status of the parents were the covariates. Race and ethnicity were the moderating variables. Logistic regression was used for data analysis.

Results

Among the participants, 5412 (51.0%) were aged 12 to 15 years, and 5207 (49.0%) were aged 16 to 17 years; 5480 (51.7%) were male. For non-Hispanic white youth, as parental educational attainment increased, there were stepwise reductions in the prevalence of tobacco dependence (13.2% vs 6.9% vs 2.7%), aggression (37.9% vs 34.8% vs 26.1%), low grade point average (84.2% vs 75.6% vs 53.3%), and chronic medical conditions (51.7% vs 50.8% vs 43.9%), but there was not such a trend for psychological distress (43.7% vs 48.6% vs 41.0%). Interactions were significant between Hispanic ethnicity and parental education on tobacco dependence (OR, 3.37 [95% CI, 2.00-5.69] for high school graduation; OR, 5.40 [95% CI, 2.52-11.56] for college graduation; P < .001 for both), aggression (OR, 1.41 [95% CI, 1.09-1.81]; P = .008 for high school graduation; OR, 1.59 [95% CI, 1.14-2.21]; P = .006 for college graduation), and psychological distress (OR, 1.50 [95% CI, 1.05-2.13]; P = .03). Black race showed an interaction with college graduation on poor school performance (OR, 2.00 [95% CI, 1.26-3.17]; P = .003) and chronic medical conditions (OR, 1.56 [95% CI, 1.14-2.14]; P = .005). All these findings suggest that the protective associations between high parental educational attainment and youth development might be systemically smaller for Hispanic and black youth than for non-Hispanic youth.

Conclusions and Relevance

Although high parental educational attainment is associated with better outcomes for youth, this association is systemically less significant for Hispanic and black than non-Hispanic white youth. The result is an increased health risk in youth from middle class black and Hispanic families. Given the systemic pattern for outcomes across domains, the diminishing returns of parental educational attainment may be due to upstream social processes that hinder ethnic minority families from translating their capital and human resources into health outcomes.

Introduction

Race- and ethnicity-associated health disparities among youth are a public health concern in the United States.1,2,3 The worse health outcomes of black and Hispanic youth compared with non-Hispanic youth are attributed, at least in part, to lower socioeconomic status (SES), particularly parental educational attainment.4,5 Outcomes tend to be worse for black and Hispanic youth than for non-Hispanic white youth, possibly because of high levels of socioeconomic adversity.6 These disparities can be seen across domains, such as health risk behaviors,7,8 aggression,7,8 school performance,5,7 psychological well-being,9,10 and physical health.2 For example, chronic medical conditions (CMCs), such as asthma11 and diabetes,12,13 are more common among black and Hispanic than non-Hispanic white youth. Disparities in school achievement are also well described in the United States.14

Because race and ethnicity closely overlap with SES indicators (eg, educational attainment),2,15 at least some of the racial and ethnic disparities in youth outcomes traditionally have been attributed to SES inequalities across racial and ethnic groups.16,17,18 Recent research,19,20 however, has introduced minorities’ diminished returns (MDRs) as an overlooked mechanism behind racial and ethnic health disparities. The concept of MDR refers to weaker-than-expected associations between SES indicators, particularly educational attainment, and tangible health outcomes for racial and ethnic minority groups compared with the majority group.19,20 Minorities’ diminished returns emphasize that some of the racial and ethnic disparities are beyond group differences in access to SES resources but instead are due to the group differences in their outcomes. As such, MDRs provide a paradigm shift for understanding the underlying mechanisms behind the racial and ethnic disparities within the middle class.19,20 Minorities’ diminished returns are also of interest because they provide an explanation for why racial and ethnic disparities have persisted despite enormous investments seeking to eliminate them.21,22,23

A growing body of research24,25,26,27,28,29 has suggested that high parental educational attainment is associated with smaller health gains among non-Hispanic black youth than among non-Hispanic white youth. Diminished returns of parental SES on self-rated health,25 depression,30,31 anxiety,32 impulse control,27 school performance,33 school attainment,24 school bonding,34 obesity,29,35 asthma,36 attention-deficit/hyperactivity disorder,37 health care use,26 and smoking38 are shown for non-Hispanic black compared with non-Hispanic white families. All these results, however, are derived from studies comparing non-Hispanic black and non-Hispanic white youth, and very limited knowledge exists on MDRs for Hispanic youth. Thus, there is a need to extend the MDR literature from black to Hispanic families.

Building on prior research on MDRs19,20 and using a nationally representative sample of youth in the United States,39,40 this study compares racial and ethnic groups of youth for the association between parental educational attainment and the following 5 youth outcomes: tobacco dependence, aggression, low grade point average (GPA), psychological distress, and CMCs. The present study expands the MDRs literature from studies of black adults,41,42,43 black youth,27,28,29 and Hispanic adults44,45,46 to Hispanic youth, and it tests whether MDRs are robust regardless of ethnicity and outcome. If MDRs are due to upstream social phenomena (eg, marginalization and differential treatment), we should expect a general pattern of MDRs across outcomes among both black and Hispanic individuals.

Methods

Design and Settings

This cross-sectional study is a secondary analysis of data from wave 1 of the Population Assessment of Tobacco and Health (PATH) study.39,40 Funded by the National Institutes of Health and the Food and Drug Administration, PATH is a state-of-the-art study on tobacco use of US youth. PATH has enrolled 13 650 people aged 12 to 17 years who are representative of US youth. Wave 1 of PATH youth data were collected in 2013 to 2014. The analytical sample for this study was limited to 10 619 youth who were either Hispanic or non-Hispanic black or white. Any individual of another racial or ethnic group, such as Asian American, Native American/Alaska Native, mixed race, or unknown race/ethnicity, was excluded.

All youth participants in the PATH study provided written assent. Their parents or guardians also provided written informed consent. The institutional review board of Westat approved the PATH study protocol. Charles Drew University of Medicine and Science institutional review board exempted the current secondary analysis from a full review. This study follows the Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) reporting guideline.47

Sample and Sampling

The PATH study’s youth samples in wave 1 were the civilian, noninstitutionalized, US population aged 12 to 17 years. The PATH study used a 4-stage stratified probability sampling to recruit participants. At first, 156 primary sampling units were selected. Primary sampling units were counties or a group of counties. The second stage formed and sampled smaller geographical segments in each primary sampling unit. The third stage involved sampling of residential addresses, using the US Postal Service Computerized Delivery Sequence Files. The fourth stage was the selection of one participating youth from the household with an eligible youth. The response rate of the participants at wave 1 of the PATH study was 78.4%.

Study Variables

The study variable roles are as follows: race and ethnicity as the moderators, parental educational attainment as the independent variable, youth outcomes as the dependent variables, and demographic factors (age, sex, and family structure) as the covariates. All the study variables were measured at the individual level. Publicly available data do not provide information on zip code; thus, we could not geocode our data in order to use higher-level SES indicators.

Race

Race (1, black vs 0, white) was self-identified. Race was a dichotomous variable.

Ethnicity

Ethnicity (1, Hispanic vs 0, non-Hispanic) was self-identified. Ethnicity was a dichotomous variable.

Parental Educational Attainment

Parents reported their educational attainment. Parental educational attainment was a 3-level nominal variable: less than high school graduate, high school graduate or more but not college graduate, and college graduate.

Demographic Factors

Age, sex, and family structure were demographic covariates. Age was a dichotomous variable (0, 12 to 15 years old vs 1, 16 to 17 years old). Family structure was a dichotomous variable (1, married vs 0, unmarried) and calculated according to parents’ marital status.

Tobacco Dependence

Tobacco dependence was calculated using the following 3 items: time to wanting to use first tobacco product after waking, ever having strong cravings to use tobacco, and ever feeling that one really needed to use tobacco. Responses to these items ranged from 1 (low) to 5 (high). We operationalized tobacco dependence as a dichotomous outcome (1, any level of dependence vs 0, no dependence). This measure is a standard measure and is used to model tobacco dependence in youth.48

Aggressive Behavior

Two items were used to measure aggressive behaviors. Participants were asked “When was the last time you did the following 2 or more times: were a bully or threatened other people and started physical fights with other people?” Items were positively correlated (r = 0.30). Item responses ranged from 1 to 4: 1, never; 2, over a year ago; 3, 2 to 12 months ago; and 4, past month. We calculated a sum score that ranged from 2 to 8, with a higher score indicating more aggressive behaviors. These items are taken from the externalizing behaviors component of the Global Appraisal of Individual Needs–Short Screener.49 This measure predicts future risk of arrest or incarceration within the last 12 months. This variable has been previously used as a categorical variable reflecting high-risk individuals, with very good predictive validity.50 Studies have shown that the Global Appraisal of Individual Needs–Short Screener instrument has adequate internal consistency, as well as overall and subscale construct validity.49

Low Grade Point Average

Participants were asked “What is your current overall GPA?” The answers were “mostly A’s,” “A’s and B’s,” “mostly B’s,” “B’s and C’s,” “mostly C’s,” “C’s and D’s,” “mostly D’s,” “D’s and F’s,” and “mostly F’s.” Low GPA was treated as a binary outcome (0, mostly As vs 1, any other status). Self-reported GPA and grades are well-established outcomes and are commonly used to measure school performance. Validation studies have shown that, overall, the self-reported GPA has very low absolute over- and underreporting, which emphasizes its validity across grade levels and subject areas.51

Psychological Distress

Psychological distress was measured using the following 4 items. “When was the last time you had significant problems with the following: feeling very trapped, lonely, sad, blue, depressed, or hopeless about the future; sleep trouble, such as bad dreams, sleeping restlessly, or falling asleep during the day; feeling very anxious, nervous, tense, scared, panicked, or like something bad was going to happen; and becoming very distressed and upset when something reminded you of the past?” Item responses ranged from 1 to 4 as follows: 1, never; 2, over a year ago; 3, two to twelve months ago; and 4, past month.52 We calculated a sum score that ranged from 4 to 16, with a higher score indicating more psychological distress (Cronbach α = 0.82). These items are taken from the internalizing behavioral component of the Global Appraisal of Individual Needs–Short Screener instrument,49 which was previously used to categorize youth as low vs high risk. This simplified classification has good predictive strength,50 as well as internal consistency and overall and subscale construct validity.49

Chronic Medical Conditions

To measure CMCs, parents were asked whether any physician has ever told them that their child has any of the following 7 CMCs: hypertension, high cholesterol, asthma, bronchitis or pneumonia, attention-deficit/hyperactivity disorder, dental problem, and diabetes. Parent reports provide valid information regarding CMCs, although some bias due to self-report measurement is expected. We treated CMCs as a binary outcome, where 1 equals 1 or more CMCs regardless of their type, and 0 equals no CMCs.

Statistical Analysis

We analyzed the data using SPSS statistical software version 23.0 (IBM). To analyze the PATH data, survey weights that are due to the sampling design (clustered stratified sampling) were applied. Because survey weights were considered, the results are representative of the US youth population. Taylor series linearization was used to re-estimate the variance of the variables. For our data analysis, we first examined the distribution of our variables. We did not perform linear regression because 3 of our variables were not linearly distributed (ie, tobacco dependence, aggressive behaviors, and number of CMCs). We also tested the assumption of homoscedasticity (eg, random distribution of error terms), which failed for tobacco dependence, aggressive behaviors, and number of CMCs. Because we had 5 outcomes with different distributions, because it was essential for us to use the very same multivariable modeling approach to all outcomes (for comparability of MDRs across outcomes), and because we could not perform linear regression for 3 of our outcomes, we chose to operationalize all our outcomes as dichotomous. Thus, for multivariable analysis, we applied binary logistic regressions to all our outcomes (ie, tobacco dependence, aggression, low GPA, psychological distress, and CMCs). We ran 2 logistic regression models per outcome, all in the pooled sample that included white, black, Hispanic, and non-Hispanic participants. The first model did not have any interaction terms. The second model included 4 interaction terms between race and ethnicity with parental educational attainment (high school and college graduation). From our logistic regression models, we reported odds ratios (ORs), 95% CIs, and P values (2-sided χ2 tests). P < .05 was considered statistically significant. Data analysis was performed from August to October 2019.

Results

Descriptive Statistics of the Participants

Table 1 summarizes descriptive statistics for the overall sample. This study included 10 619 US youth who were aged 12 to 17 years. The sample was almost half male (5480 [51.7%]) and half female (5113 [48.3%]), and also half 12 to 15 years old (5412 [51.0%]) and half 16 to 17 years old (5207 [49.0%]). In total, 1982 youth (18.7%) were black and 8637 (81.3%) were white. Among all participants, 8236 were non-Hispanic (77.6%) and 2383 were Hispanic (22.4%).

Table 1. Descriptive Statistics in the Overall Sample and by Race and Ethnicity.

Variable All Participants, No. (%) (N = 10 619) Ethnicity, No. (%) P Valuea Race, No. (%) P Valuea
Non-Hispanic (n = 8236) Hispanic (n = 2383) White (n = 8637) Black (n = 1982)
Sex
Female 5113 (48.3) 3967 (48.3) 1146 (48.3) ≥.05 4166 (48.3) 947 (48.0) ≥.05
Male 5480 (51.7) 4252 (51.7) 1228 (51.7) 4456 (51.7) 1024 (52.0)
Age, y
12-15 5412 (51.0) 4108 (49.9) 1304 (54.7) ≥.05 4399 (50.9) 1013 (51.1) ≥.05
16-17 5207 (49.0) 4128 (50.1) 1079 (45.3) 4238 (49.1) 969 (48.9)
Family structure
Nonmarried parents 3820 (36.0) 2867 (34.8) 953 (40.0) <.05 2618 (30.3) 1202 (60.6) <.05
Married parents 6792 (64.0) 5365 (65.2) 1427 (60.0) 6012 (69.7) 780 (39.4)
Parental educational attainment
Less than high school graduate 1963 (18.5) 1026 (12.5) 937 (39.3) <.05 1531 (17.7) 432 (21.8) <.05
High school graduate 5530 (52.1) 4422 (53.7) 1108 (46.5) <.05 4373 (50.6) 1157 (58.4) <.05
College graduate 3126 (29.4) 2788 (33.9) 338(14.2) <.05 2733 (31.6) 393 (19.8) <.05
Tobacco dependence, any 542 (5.1) 441 (5.4) 101 (4.2) ≥.05 476 (5.5) 66 (3.3) <.05
Aggression 3372 (32.1) 2687 (33.0) 685 (29.1) <.05 2657 (31.1) 715 (36.6) <.05
Low grade point average 7853 (74.5) 5902 (72.2) 1951 (82.3) <.05 6141 (71.6) 1712 (86.8) <.05
Psychological distress 3570 (44.9) 2801 (45.0) 769 (44.6) ≥.05 2949 (45.2) 621 (43.6) <.05
Chronic medical condition 5027 (47.5) 3983 (48.5) 1044 (44.0) <.05 4046 (47.0) 981 (49.6) <.05
a

P for comparison of Hispanic and non-Hispanic or white and black youth, with P < .05 denoting statistical significance.

Outcomes Based on the Intersections of Race/Ethnicity and Educational Attainment

Table 2 presents the distributions of our binary outcomes based on the intersections of race, ethnicity, and educational attainment. This table shows how the prevalence of our undesired youth outcomes change as a function of educational attainment for each racial/ethnic group.

Table 2. Prevalence of Undesired Youth Outcomes by the Intersection of Race/Ethnicity and Educational Level.

Outcome Educational Level
Non-Hispanic Hispanic
White, No. (%) P Valuea Black, No. (%) P Valuea White, No. (%) P Valuea Black, No. (%) P Valuea
Low Mid High Low Mid High Low Mid High Low Mid High
Tobacco dependence 87 (13.2) 231 (6.9) 65 (2.7) <.05 17 (4.6) 33 (3.1) 8 (2.2) <.05 29 (3.3) 52 (5.1) 12 (4.0) ≥.05 2 (3.0) 0 (0.0) 0 (0.0) ≥.05
Aggression 248 (37.9) 1159 (34.8) 629 (26.1) <.05 150 (41.7) 373 (35.6) 128 (36.3) ≥.05 225 (26.1) 318 (31.7) 78 (26.2) ≥.05 22 (34.4) 31 (32.3) 11 (32.4) ≥.05
Grade point average 548 (84.2) 2524 (75.6) 1285 (53.3) <.05 333 (91.2) 927 (87.8) 285 (79.8) <.05 759 (87.7) 820 (81.3) 205 (67.7) <.05 56 (86.2) 83 (86.5) 28 (84.8) ≥.05
Psychological distress 206 (43.7) 1265 (48.6) 759 (41.0) ≥.05 117 (45.2) 343 (44.7) 111 (41.3) <.05 243 (40.8) 363 (47.6) 113 (47.1) ≥.05 15 (34.9) 27 (41.5) 8 (40.0) ≥.05
Chronic medical condition 340 (51.7) 1703 (50.8) 1063 (43.9) <.05 168 (46.0) 519 (49.0) 190 (53.2) ≥.05 337 (38.8) 466 (46.1) 137 (45.7) ≥.05 30 (46.2) 56 (58.3) 18 (51.4) ≥.05
a

P for trend for comparison of percentage outcome across educational levels within ethnic group, with P < .05 denoting statistical significance.

As educational attainment increased, we observed a statistically significant stepwise reduction (P < .05 for trend) in the prevalence of tobacco dependence (13.2% vs 6.9% vs 2.7%), aggression (37.9% vs 34.8% vs 26.1%), low GPA (84.2% vs 75.6% vs 53.3%), and CMCs (51.7% vs 50.8% vs 43.9%) for non-Hispanic whites. For whites, psychological distress did not show such a trend (43.7% vs 48.6% vs 41.0%; P ≥ .05 for trend). For Hispanic white youth, such a significant stepwise reduction was only seen for low GPA (87.7% vs 81.3% vs 67.7%). For non-Hispanic black youth, tobacco use (4.6% vs 3.1% vs 2.2%), low GPA (91.2% vs 87.8% vs 79.8%), and psychological distress (45.2% vs 44.7% vs 41.3%) showed such a significant stepwise reduction. The same stepwise reduction could not be seen for other outcomes in other racial/ethnic groups (P ≥ .05 for trend).

Multivariable Models

Table 3 presents the summary of 10 logistic regression models in the pooled sample. In all these models, parental educational attainment was the main independent variable. For each outcome, 2 models were run. Although model 1 only entered the main effects of parental educational attainment, race, ethnicity, and covariates, model 2 also included 4 statistical interaction terms between race and ethnicity and parental educational levels (ie, high school graduation and college graduation).

Table 3. Summary of 2 Series of Logistic Regressions on the Interactive Effects of Race, Ethnicity, and Parental Educational Attainment on Youth Outcomes.

Characteristic Model 1: Main Effects Model 2: Model 1 Plus Interactions
OR (95% CI) P Value OR (95% CI) P Value
Tobacco dependence (any)
Ethnicity (Hispanic) 0.59 (0.46-0.75) <.001 0.25 (0.16-0.38) <.001
Race (black) 0.41 (0.31-0.54) <.001 0.30 (0.18-0.50) <.001
Male 1.42 (1.19-1.70) <.001 1.40 (1.17-1.68) <.001
Age 5.11 (4.08-6.40) <.001 5.14 (4.11-6.44) <.001
Parents married 0.53 (0.44-0.64) <.001 0.54 (0.45-0.65) <.001
Parental education <.001 <.001
High school graduate 0.74 (0.59-0.92) .007 0.50 (0.38-0.66) <.001
College graduate 0.34 (0.25-0.46) <.001 0.22 (0.16-0.31) <.001
Ethnicity (Hispanic) × high school graduate NAa NAa 3.37 (2.00-5.69) <.001
Ethnicity (Hispanic) × college graduate NAa NAa 5.40 (2.52-11.56) <.001
Race (black) × high school graduate NAa NAa 1.38 (0.74-2.58) .31
Race (black) × college graduate NAa NAa 1.93 (0.78-4.78) .15
Intercept 0.04 <.001 0.06 <.001
Aggression
Ethnicity (Hispanic) 0.79 (0.71-0.88) <.001 0.61 (0.49-0.74) <.001
Race (black) 1.12 (1.00-1.25) .04 1.18 (0.93-1.50) .175
Male 1.53 (1.41-1.66) <.001 1.53 (1.41-1.66) <.001
Age 1.07 (0.98-1.16) .12 1.07 (0.98-1.16) .13
Parents married 0.81 (0.74-0.89) <.001 0.81 (0.74-0.89) <.001
Parental education <.001 <.001
High school graduate 1.00 (0.89-1.12) .98 0.92 (0.77-1.09) .32
College graduate 0.74 (0.64-0.84) <.001 0.62 (0.52-0.75) <.001
Ethnicity (Hispanic) × high school graduate NAa NAa 1.41 (1.09-1.81) .008
Ethnicity (Hispanic) × college graduate NAa NAa 1.59 (1.14-2.21) .006
Race (black) × high school graduate NAa NAa 0.82 (0.62-1.09) .17
Race (black) × college graduate NAa NAa 1.26 (0.90-1.75) .18
Intercept 0.47 <.001 0.52 <.001
Low grade point average (other than As)
Ethnicity (Hispanic) 1.47 (1.30-1.67) <.001 1.29 (0.97-1.71) .08
Race (black) 2.20 (1.90-2.55) <.001 1.54 (1.06-2.24) .03
Male 2.06 (1.88-2.27) <.001 2.07 (1.88-2.27) <.001
Age 1.31 (1.20-1.44) <.001 1.31 (1.20-1.44) <.001
Parents married 0.61 (0.55-0.68) <.001 0.61 (0.55-0.68) <.001
Parental education <.001 <.001
High school graduate 0.64 (0.55-0.75) <.001 0.59 (0.47-0.74) <.001
College graduate 0.26 (0.23-0.31) <.001 0.22 (0.18-0.28) <.001
Ethnicity (Hispanic) × high school graduate NAa NAa 1.05 (0.75-1.46) .78
Ethnicity (Hispanic) × college graduate NAa NAa 1.41 (0.97-2.06) .07
Race (black) × high school graduate NAa NAa 1.28 (0.84-1.95) .23
Race (black) × college graduate NAa NAa 2.00 (1.26-3.17) .003
Intercept 4.13 <.001 4.64 <.001
Psychological distress
Ethnicity (Hispanic) 0.98 (0.88-1.11) .79 0.84 (0.66-1.06) .14
Race (black) 0.87 (0.77-0.99) .03 0.95 (0.72-1.26) .75
Male 0.49 (0.45-0.54) <.001 0.49 (0.45-0.54) <.001
Age 1.41 (1.29-1.55) <.001 1.41 (1.29-1.55) <.001
Parents married 0.84 (0.76-0.93) .001 0.84 (0.76-0.93) .001
Parental education <.001 <.001
High school graduate 1.27 (1.11-1.44) <.001 1.22 (1.00-1.48) .05
College graduate 1.02 (0.88-1.18) .80 0.93 (0.76-1.14) .48
Ethnicity (Hispanic) × high school graduate NAa NAa 1.17 (0.89-1.56) .26
Ethnicity (Hispanic) × college graduate NAa NAa 1.50 (1.05-2.13) .03
Race (black) × high school graduate NAa NAa 0.85 (0.61-1.17) .31
Race (black) × college graduate NAa NAa 1.00 (0.68-1.45) .98
Intercept 0.96 .63 1.02 .86
Chronic medical conditions (any)
Ethnicity (Hispanic) 0.83 (0.75-0.91) <.001 0.64 (0.53-0.78) <.001
Race (black) 0.97 (0.87-1.08) .55 0.83 (0.66-1.05) .13
Male 1.43 (1.32-1.55) <.001 1.43 (1.32-1.54) <.001
Age 1.05 (0.97-1.13) .23 1.05 (0.97-1.13) .25
Parents married 0.73 (0.68-0.80) <.001 0.74 (0.68-0.80) <.001
Parental education <.001 <.001
High school graduate 1.19 (1.07-1.33) .001 1.04 (0.88-1.22) .66
College graduate 1.03 (0.91-1.16) .68 0.83 (0.70-0.98) .03
Ethnicity (Hispanic) × high school graduate NAa NAa 1.32 (1.05-1.68) .02
Ethnicity (Hispanic) × college graduate NAa NAa 1.62 (1.20-2.18) .002
Race (black) × high school graduate NAa NAa 1.06 (0.82-1.39) .65
Race (black) × college graduate NAa NAa 1.56 (1.14-2.14) .005
Intercept 0.85 .01 0.98 .85

Abbreviations: NA, not applicable; OR, odds ratio.

a

Interaction term was not entered into the model.

According to model 1, parental educational attainment was associated with all youth outcomes, namely tobacco dependence (OR, 0.74 [95% CI, 0.59-0.92]; P = .007 for high school graduation; OR, 0.34 [95% CI, 0.25-0.46]; P < .001 for college graduation), aggression (OR, 0.74 [95% CI, 0.64-0.84]; P < .001 for college graduation), low GPA (OR, 0.64 [95% CI, 0.55-0.75] for high school graduation; OR, 0.26 [95% CI, 0.23-0.31] for college graduation; P < .001 for both), psychological distress (OR, 1.27 [95% CI, [1.11-1.44]; P < .001 for high school graduation), and CMCs (OR, 1.19 [95% CI, 1.07-1.33]; P = .001 for high school graduation). As the direction of these ORs shows, for 3 outcomes, higher parental educational attainment was negatively associated with the undesired youth outcomes (tobacco dependence, aggression, and low GPA), and for 2 outcomes, high parental educational attainment was positively associated with undesired outcome (psychological distress and CMCs).

Model 2 showed statistically significant interactions between race and ethnicity with parental educational attainment on all youth outcomes. Interactions were significant between Hispanic ethnicity and education on tobacco dependence (OR, 3.37 [95% CI, 2.00-5.69] for high school graduation; OR, 5.40 [95% CI, 2.52-11.56] for college graduation; P < .001 for both), aggressive behaviors (OR, 1.41 [95% CI, 1.09-1.81]; P = .008 for high school graduation; OR, 1.59 [95% CI, 1.14-2.21]; P = .006 for college graduation), and psychological distress (OR, 1.50 [95% CI, 1.05-2.13]; P = .03). Race showed an interaction with college graduation on low GPA (OR, 2.00 [95% CI, 1.26-3.17]; P = .003) and CMCs (OR, 1.56 [95% CI, 1.14-2.14]; P = .005). All these findings suggest that the protective effect of high parental educational attainment on youth development might be systemically smaller for Hispanic and black than for non-Hispanic youth. These findings suggested that high parental educational attainment has a less significant association with positive youth outcomes for black and Hispanic families than for non-Hispanic families.

Discussion

The current study showed significant interactions between race and ethnicity with parental educational attainment on youth outcomes. Overall, the associations between high parental educational attainment and youth outcomes were found to be less significant for ethnic minority families than for non-Hispanic white families. However, this pattern was heterogeneous and inconsistent across ethnic groups and level of education that showed the interaction.

Because MDRs could be observed across outcomes and ethnic minorities (ie, blacks and Hispanics), we hypothesize that the probable causes for the observed MDRs of parental educational attainment are some upstream social forces. This is because proximal or downstream determinants of tobacco dependence, aggression, low GPA, psychological distress, and CMCs vary to a large degree. These outcomes, however, overlap in terms of upstream distal social determinants.53,54,55

This study confirmed what was previously found for black adults,41,42,43 black youth,27,28,29 and Hispanic adults.44,45,46 Compared with non-Hispanic white youth, black and Hispanic youth remain at high risk of asthma,36 attention-deficit/hyperactivity disorder,37 mental health problems,25 depression,30,31 obesity,29,35 dental health problems,56 poor health care use,26 low GPA,33 poor school attainment,24 poor school bonding,34 impulse control,27 and cigarette smoking,38 all of which are disproportionate to their high parental education.

These patterns are not limited to youth but carry over into adulthood. These patterns also are not limited to educational attainment25 and are seen for income,42 employment,57 and marital status.32 Minorities’ diminished returns are well described for obesity,29,35 substance use,44 depression,42 happiness,58 affect,58 anxiety,32 self-rated health,59 CMCs,37 oral health,26 and mortality.57 This study suggests that the same patterns also apply to Hispanic youth.

Because of the existing MDRs, we should observe worse-than-expected outcomes in Hispanic youth from middle-class families. Thus, ethnic health disparities are not limited to the low SES sections of the society because other social mechanisms are at work to diminish the health return of parental education for ethnic minority families.

In this study, Hispanic youth showed less aggression than white youth. This seemingly counterintuitive result could be understood in the context of the Hispanic health paradox60,61 and the healthy immigrant effect.62 Similarly, black youth had less tobacco dependence than white youth. Some better mental health outcomes of blacks compared with whites have been attributed to resilience, which might be a function of adversity.63

As mentioned already, despite the general trend, some inconsistencies were observed. We found differences in MDRs based on race and ethnic minority group, educational level, and the outcome. We need to conduct more studies to understand the circumstances in which a particular educational level does promote an outcome for one but not another ethnic group. Despite these heterogeneities and nuances, black and Hispanic youth tended to be at an overall disadvantage compared with non-Hispanic white youth.

Future Research

There is a need to study structural and societal factors that result in MDRs of SES indicators for ethnic minority groups. Because diminished returns hold across health outcomes and the health outcomes are universally affected, regardless of the domain, it would appear that some upstream social processes may be interfering with the translation of human capital into health. For example, residential segregation, stressful neighborhoods, unequal quality of schooling, discrimination, and poor access to healthy food options may be mechanisms hindering the development of youth from middle-class ethnic minority families. The high psychological costs of upward social mobility may be another mechanism causing diminished returns of parental educational attainment for ethnic minority youth. At the same time, some mechanisms may be relevant to one but not to other outcomes. For example, predatory tobacco marketing practices may play a role in explaining ethnic differences in returns of SES in the form of tobacco use disparities. However, because these patterns are independent of the outcome, we argue that racism and colorism may be deeper causes. However, testing this hypothesis needs additional research. Again, previous studies31,64,65 that have proposed other mechanisms have all been limited to black participants. There is, thus, an urgent need to study the mechanisms of MDRs in Hispanic families. Another unanswered question is the actual magnitude of these MDRs. Some of these interactions were significant but with a small effect. We argue that their upstream nature will have large trickle-down effects, because many outcomes are shaped by more distal social determinants of health. In addition, even though the effect sizes of MDRs may be small, when multiplied throughout the total population larger differences may show up at the population level.66

Policy Implications

These results suggest that specific types of policies and public health programs may be needed to reduce ethnic health disparities. Because disparities are shaped by the differential outcomes of SES, there is a need to go beyond policies focusing exclusively on health to policies addressing the broader social processes placing ethnic minorities at a relative disadvantage. There is a need for innovative national and local policies that reduce disparities due to differential SES and also those that happen at identical SES levels (ie, MDRs).20,26,27,29,32,41,44,56,67 There is a need to discover policy and program solutions that equalize a minority population’s abilities to leverage its available educational attainment.19,20 Future research should explore the role of discrimination in the labor market,41 the potential outcomes of enforcement of existing antidiscrimination laws, and the diminished returns of educational attainment among black and Hispanic families. Communities where ethnic minorities reside may benefit from additional investment aimed at education quality, a strategy that may boost the translation of educational attainment into health outcomes.68 Programs that help families secure high-paying jobs are essential. Overall, to undo MDRs of parental educational attainment among Hispanic families, we need to minimize the societal and environmental barriers in the daily lives of ethnic minorities. We should also continuously invest in educational programs aimed at society at large to reduce the unfair treatment of highly educated ethnic minority people.

Policy makers should be aware that inequalities persist among highly educated individuals through diminished returns of human capital among ethnic minority people. This view of health disparities is in line with the view that it is race/ethnicity and class, not race/ethnicity or class, that affect health disparities.69,70 This view may alter how we allocate resources to eliminate racial and ethnic health disparities. Instead of a reductionist view that attributes ethnic health disparities solely to SES inequalities, health gaps must be addressed across SES levels. However, this study suggests that simply reallocating resources is not sufficient. We must empower ethnic minorities to efficiently translate their human capital into positive health outcomes, thereby realizing their full health potential.

Limitations

This study had some methodological limitations. The cross-sectional design prevented us from making any causal inferences. The sample size was imbalanced across racial and ethnic groups. We only included black, Hispanic, and non-Hispanic white youth. Other ethnic minorities such as Asian populations should be included in future studies. We only studied the differential outcome of educational attainment. Other family SES indicators, such as income, employment, and wealth, were not available. These important components of SES are not accounted for in our analyses, and they may confound the associations between education, ethnicity, and youth outcomes. The degree to which disparities remain beyond SES should be determined in the studies that can control for a wide range of SES components. We did not have data on area-level SES factors. This study did not include variation by zip code or geographic location. Despite these limitations, we believe this study still contributes to extending the existing literature on MDRs among US youth. A large sample size, a random sampling, generalizable results to the United States, and the multiple outcomes studied are among the strengths of this study.

Conclusions

In the United States, black and Hispanic youth are at a relative disadvantage compared with their non-Hispanic white counterparts regarding the magnitude of the association between educational attainment of their parents and youth outcomes.

References

  • 1.Guglielmo D, Gazmararian JA, Chung J, Rogers AE, Hale L. Racial/ethnic sleep disparities in US school-aged children and adolescents: a review of the literature. Sleep Health. 2018;4(1):-. doi: 10.1016/j.sleh.2017.09.005 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 2.Butler AM. Social determinants of health and racial/ethnic disparities in type 2 diabetes in youth. Curr Diab Rep. 2017;17(8):60. doi: 10.1007/s11892-017-0885-0 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 3.Messiah SE, Arheart KL, Lopez-Mitnik G, Lipshultz SE, Miller TL. Ethnic group differences in cardiometabolic disease risk factors independent of body mass index among American youth. Obesity (Silver Spring). 2013;21(3):424-428. doi: 10.1002/oby.20343 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 4.Albrecht SS, Gordon-Larsen P. Ethnic differences in body mass index trajectories from adolescence to adulthood: a focus on Hispanic and Asian subgroups in the United States. PLoS One. 2013;8(9):e72983. doi: 10.1371/journal.pone.0072983 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 5.Arellano CM, Chavez EL, Deffenbacher JL. Alcohol use and academic status among Mexican American and White non-Hispanic adolescents. Adolescence. 1998;33(132):751-760. [PubMed] [Google Scholar]
  • 6.Benjet C, Borges G, Medina-Mora ME, Zambrano J, Cruz C, Méndez E. Descriptive epidemiology of chronic childhood adversity in Mexican adolescents. J Adolesc Health. 2009;45(5):483-489. doi: 10.1016/j.jadohealth.2009.03.002 [DOI] [PubMed] [Google Scholar]
  • 7.Basch CE. Aggression and violence and the achievement gap among urban minority youth. J Sch Health. 2011;81(10):619-625. doi: 10.1111/j.1746-1561.2011.00636.x [DOI] [PubMed] [Google Scholar]
  • 8.Wallace JM Jr, Vaughn MG, Bachman JG, O’Malley PM, Johnston LD, Schulenberg JE. Race/ethnicity, socioeconomic factors, and smoking among early adolescent girls in the United States. Drug Alcohol Depend. 2009;104(1)(suppl):S42-S49. doi: 10.1016/j.drugalcdep.2009.06.007 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 9.Assari S, Caldwell CH. Mental health service utilization among black youth: psychosocial determinants in a national sample. Children (Basel). 2017;4(5):E40. doi: 10.3390/children4050040 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 10.Malhotra K, Shim R, Baltrus P, Heiman HJ, Adekeye O, Rust G. Racial/ethnic disparities in mental health service utilization among youth participating in negative externalizing behaviors. Ethn Dis. 2015;25(2):123-129. [PubMed] [Google Scholar]
  • 11.Forno E, Celedon JC. Asthma and ethnic minorities: socioeconomic status and beyond. Curr Opin Allergy Clin Immunol. 2009;9(2):154-160. doi: 10.1097/ACI.0b013e3283292207 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 12.Lado JJ, Lipman TH. Racial and ethnic disparities in the incidence, treatment, and outcomes of youth with type 1 diabetes. Endocrinol Metab Clin North Am. 2016;45(2):453-461. doi: 10.1016/j.ecl.2016.01.002 [DOI] [PubMed] [Google Scholar]
  • 13.El-Toukhy S, Sabado M, Choi K. Trends in susceptibility to smoking by race and ethnicity. Pediatrics. 2016;138(5):e20161254. doi: 10.1542/peds.2016-1254 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 14.Chavous TM, Rivas-Drake D, Smalls C, Griffin T, Cogburn C. Gender matters, too: the influences of school racial discrimination and racial identity on academic engagement outcomes among African American adolescents. Dev Psychol. 2008;44(3):637-654. doi: 10.1037/0012-1649.44.3.637 [DOI] [PubMed] [Google Scholar]
  • 15.Carlo G, Crockett LJ, Carranza MA, Martinez MM. Understanding ethnic/racial health disparities in youth and families in the US. Nebr Symp Motiv. 2011;57:1-11. doi: 10.1007/978-1-4419-7092-3_1 [DOI] [PubMed] [Google Scholar]
  • 16.Rossen LM, Schoendorf KC. Measuring health disparities: trends in racial-ethnic and socioeconomic disparities in obesity among 2- to 18-year old youth in the United States, 2001-2010. Ann Epidemiol. 2012;22(10):698-704. doi: 10.1016/j.annepidem.2012.07.005 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 17.Lau M, Lin H, Flores G. Racial/ethnic disparities in health and health care among U.S. adolescents. Health Serv Res. 2012;47(5):2031-2059. doi: 10.1111/j.1475-6773.2012.01394.x [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 18.Jones A. Race, socioeconomic status, and health during childhood: a longitudinal examination of racial/ethnic differences in parental socioeconomic timing and child obesity risk. Int J Environ Res Public Health. 2018;15(4):E728. doi: 10.3390/ijerph15040728 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 19.Assari S. Health disparities due to diminished return among black Americans: public policy solutions. Soc Issues Policy Rev. 2018;12(1):112-145. doi: 10.1111/sipr.12042 [DOI] [Google Scholar]
  • 20.Assari S. Unequal gain of equal resources across racial groups. Int J Health Policy Manag. 2018;7(1):1-9. doi: 10.15171/ijhpm.2017.90 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 21.Dankwa-Mullan I, Rhee KB, Williams K, et al. . The science of eliminating health disparities: summary and analysis of the NIH summit recommendations. Am J Public Health. 2010;100(1)(suppl):S12-S18. doi: 10.2105/AJPH.2010.191619 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 22.Apter AJ, Casillas AM. Eliminating health disparities: what have we done and what do we do next? J Allergy Clin Immunol. 2009;123(6):1237-1239. doi: 10.1016/j.jaci.2009.04.028 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 23.Yearwood EL. NIH summit: the science of eliminating health disparities. J Child Adolesc Psychiatr Nurs. 2009;22(2):109-110. doi: 10.1111/j.1744-6171.2009.00188.x [DOI] [PubMed] [Google Scholar]
  • 24.Assari S. Parental education attainment and educational upward mobility; role of race and gender. Behav Sci (Basel). 2018;8(11):E107. doi: 10.3390/bs8110107 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 25.Assari S. Parental educational attainment and mental well-being of college students: diminished returns of Blacks. Brain Sci. 2018;8(11):E193. doi: 10.3390/brainsci8110193 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 26.Assari S, Hani N. Household income and children's unmet dental care need; blacks' diminished return. Dent J (Basel). 2018;6(2):E17. doi: 10.3390/dj6020017 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 27.Assari S, Caldwell CH, Mincy R. Family socioeconomic status at birth and youth impulsivity at age 15; blacks' diminished return. Children (Basel). 2018;5(5):E58. doi: 10.3390/children5050058. [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 28.Assari S, Caldwell CH, Mincy RB. Maternal educational attainment at birth promotes future self-rated health of white but not black youth: a 15-year cohort of a national sample. J Clin Med. 2018;7(5):E93. doi: 10.3390/jcm7050093 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 29.Assari S, Thomas A, Caldwell CH, Mincy RB. Blacks’ diminished health return of family structure and socioeconomic status: 15 years of follow-up of a national urban sample of youth. J Urban Health. 2018;95(1):21-35. doi: 10.1007/s11524-017-0217-3 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 30.Assari S, Caldwell CH. High risk of depression in high-income African American boys. J Racial Ethn Health Disparities. 2018;5(4):808-819. doi: 10.1007/s40615-017-0426-1 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 31.Assari S, Lankarani MM, Caldwell CH. Does discrimination explain high risk of depression among high-income African American men? Behav Sci (Basel). 2018;8(4):E40. doi: 10.3390/bs8040040 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 32.Assari S, Caldwell CH, Zimmerman MA. Family structure and subsequent anxiety symptoms: minorities' diminished return. Brain Sci. 2018;8(6):E97. doi: 10.3390/brainsci8060097 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 33.Assari S. Parental educational attainment and academic performance of American college students: blacks’ diminished returns. J Health Econ Dev. 2019;1(1):21-31. [PMC free article] [PubMed] [Google Scholar]
  • 34.Assari S. Family socioeconomic position at birth and school bonding at age 15: blacks’ diminished returns. Behav Sci (Basel). 2019;9(3):E26. doi: 10.3390/bs9030026 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 35.Assari S. Family income reduces risk of obesity for white but not black children. Children (Basel). 2018;5(6):E73. doi: 10.3390/children5060073 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 36.Assari S, Moghani Lankarani M. Poverty status and childhood asthma in white and black families: national survey of children’s health. Healthcare (Basel). 2018;6(2):E62. doi: 10.3390/healthcare6020062 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 37.Assari S, Caldwell CH. Family income at birth and risk of attention deficit hyperactivity disorder at age 15: racial differences. Children (Basel). 2019;6(1):E10. doi: 10.3390/children6010010 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 38.Bachman JG, O’Malley PM, Johnston LD, Schulenberg JE, Wallace JM. Racial/ethnic differences in the relationship between parental education and substance use among U.S. 8th-, 10th-, and 12th-grade students: findings from the Monitoring the Future project. J Stud Alcohol Drugs. 2011;72(2):279-285. doi: 10.15288/jsad.2011.72.279 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 39.Tourangeau R, Yan T, Sun H, Hyland A, Stanton CA. Population Assessment of Tobacco and Health (PATH) reliability and validity study: selected reliability and validity estimates. Tob Control. 2018. October 8. doi: 10.1136/tobaccocontrol-2018-054561 [DOI] [PubMed] [Google Scholar]
  • 40.Hyland A, Ambrose BK, Conway KP, et al. . Design and methods of the Population Assessment of Tobacco and Health (PATH) Study. Tob Control. 2017;26(4):371-378. doi: 10.1136/tobaccocontrol-2016-052934 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 41.Assari S. Blacks’ diminished return of education attainment on subjective health: mediating effect of income. Brain Sci. 2018;8(9):E176. doi: 10.3390/brainsci8090176 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 42.Assari S. High income protects whites but not African Americans against risk of depression. Healthcare (Basel). 2018;6(2):E37. doi: 10.3390/healthcare6020037 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 43.Assari S. The benefits of higher income in protecting against chronic medical conditions are smaller for African Americans than whites. Healthcare (Basel). 2018;6(1):E2. doi: 10.3390/healthcare6010002 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 44.Assari S, Farokhnia M, Mistry R. Education attainment and alcohol binge drinking: diminished returns of Hispanics in Los Angeles. Behav Sci (Basel). 2019;9(1):E9. doi: 10.3390/bs9010009 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 45.Assari S, Mistry R. Diminished return of employment on ever smoking among Hispanic whites in Los Angeles. Health Equity. 2019;3(1):138-144. doi: 10.1089/heq.2018.0070 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 46.Assari S. Socioeconomic determinants of systolic blood pressure: minorities’ diminished returns. J Health Econ Dev. 2019;1(1):1-11. [PubMed] [Google Scholar]
  • 47.Ebrahim S, Clarke M. STROBE: new standards for reporting observational epidemiology, a chance to improve. Int J Epidemiol. 2007;36(5):946-948. doi: 10.1093/ije/dym185 [DOI] [PubMed] [Google Scholar]
  • 48.Liu G, Wasserman E, Kong L, Foulds J. A comparison of nicotine dependence among exclusive E-cigarette and cigarette users in the PATH study. Prev Med. 2017;104:86-91. doi: 10.1016/j.ypmed.2017.04.001 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 49.McDonell MG, Comtois KA, Voss WD, Morgan AH, Ries RK. Global Appraisal of Individual Needs Short Screener (GSS): psychometric properties and performance as a screening measure in adolescents. Am J Drug Alcohol Abuse. 2009;35(3):157-160. doi: 10.1080/00952990902825421 [DOI] [PubMed] [Google Scholar]
  • 50.Garner BR, Belur VK, Dennis ML. The GAIN Short Screener (GSS) as a predictor of future arrest or incarceration among youth presenting to substance use disorder (SUD) treatment. Subst Abuse. 2013;7:199-208. doi: 10.4137/SART.S13152 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 51.Sticca F, Goetz T, Bieg M, Hall NC, Eberle F, Haag L. Examining the accuracy of students’ self-reported academic grades from a correlational and a discrepancy perspective: evidence from a longitudinal study. PLoS One. 2017;12(11):e0187367. doi: 10.1371/journal.pone.0187367 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 52.Sawdey MD, Day HR, Coleman B, et al. . Associations of risk factors of e-cigarette and cigarette use and susceptibility to use among baseline PATH study youth participants (2013-2014). Addict Behav. 2019;91:51-60. doi: 10.1016/j.addbeh.2018.11.027 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 53.Mirowsky J, Ross CE. Education, health, and the default American lifestyle. J Health Soc Behav. 2015;56(3):297-306. doi: 10.1177/0022146515594814 [DOI] [PubMed] [Google Scholar]
  • 54.Ross CE, Mirowsky J. Refining the association between education and health: the effects of quantity, credential, and selectivity. Demography. 1999;36(4):445-460. doi: 10.2307/2648083 [DOI] [PubMed] [Google Scholar]
  • 55.Ross CE, Mirowsky J. The interaction of personal and parental education on health. Soc Sci Med. 2011;72(4):591-599. doi: 10.1016/j.socscimed.2010.11.028 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 56.Assari S. Socioeconomic status and self-rated oral health: diminished return among Hispanic whites. Dent J (Basel). 2018;6(2):E11. doi: 10.3390/dj6020011 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 57.Assari S. Life expectancy gain due to employment status depends on race, gender, education, and their intersections. J Racial Ethn Health Disparities. 2018;5(2):375-386. doi: 10.1007/s40615-017-0381-x [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 58.Assari S, Preiser B, Kelly M. Education and income predict future emotional well-being of whites but not blacks: a ten-year cohort. Brain Sci. 2018;8(7):E122. doi: 10.3390/brainsci8070122 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 59.Assari S, Lapeyrouse LM, Neighbors HW. Income and self-rated mental health: diminished returns for high income black Americans. Behav Sci (Basel). 2018;8(5):E50. doi: 10.3390/bs8050050 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 60.Giuntella O. The Hispanic health paradox: new evidence from longitudinal data on second and third-generation birth outcomes. SSM Popul Health. 2016;2:84-89. doi: 10.1016/j.ssmph.2016.02.013 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 61.Vega WA, Sribney WM. Understanding the Hispanic health paradox through a multi-generation lens: a focus on behavior disorders. Nebr Symp Motiv. 2011;57:151-168. doi: 10.1007/978-1-4419-7092-3_7 [DOI] [PubMed] [Google Scholar]
  • 62.Constant AF, García-Muñoz T, Neuman S, Neuman T. A “healthy immigrant effect” or a “sick immigrant effect”? selection and policies matter. Eur J Health Econ. 2018;19(1):103-121. doi: 10.1007/s10198-017-0870-1 [DOI] [PubMed] [Google Scholar]
  • 63.Assari S, Lankarani MM. Association between stressful life events and depression: intersection of race and gender. J Racial Ethn Health Disparities. 2016;3(2):349-356. doi: 10.1007/s40615-015-0160-5 [DOI] [PubMed] [Google Scholar]
  • 64.Assari S. Does school racial composition explain why high income black youth perceive more discrimination? a gender analysis. Brain Sci. 2018;8(8):E140. doi: 10.3390/brainsci8080140 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 65.Assari S, Moghani Lankarani M. Workplace racial composition explains high perceived discrimination of high socioeconomic status African American men. Brain Sci. 2018;8(8):E139. doi: 10.3390/brainsci8080139 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 66.Lantz PM. The medicalization of population health: who will stay upstream? Milbank Q. 2019;97(1):36-39. doi: 10.1111/1468-0009.12363 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 67.Assari S, Mistry R. Educational attainment and smoking status in a national sample of American adults: evidence for the blacks’ diminished return [published correction appears in Int J Environ Res Public Health. 2018;15(10):E2084]. Int J Environ Res Public Health. 2018;15(4):E763. doi: 10.3390/ijerph15040763 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 68.Hazelzet E, Picco E, Houkes I, Bosma H, de Rijk A. Effectiveness of interventions to promote sustainable employability: a systematic review. Int J Environ Res Public Health. 2019;16(11):E1985. doi: 10.3390/ijerph16111985 [DOI] [PMC free article] [PubMed] [Google Scholar]
  • 69.Navarro V. Race or class, or race and class. Int J Health Serv. 1989;19(2):311-314. doi: 10.2190/CNUH-67T0-RLBT-FMCA [DOI] [PubMed] [Google Scholar]
  • 70.Navarro V. Race or class versus race and class: mortality differentials in the United States. Lancet. 1990;336(8725):1238-1240. doi: 10.1016/0140-6736(90)92846-A [DOI] [PubMed] [Google Scholar]

Articles from JAMA Network Open are provided here courtesy of American Medical Association

RESOURCES