Correction: Loss of ZNF32 augments the regeneration of nervous lateral line system through negative regulation of SOX2 transcription

This article has been corrected: In Figure 6F, the images of P-1-169 group from Figure 5D were accidentally duplicated. The corrected Figure 6 is shown below. The authors declare that these corrections do not change the results or conclusions of this paper.

Original article: Oncotarget. 2016; 7:70420–70436. 70420-70436. https://doi.org/10.18632/oncotarget.11895

Figure 6. Identification of NLSs in ZNF32 and the localization of NLS mutants.

(A, B) ZNF32 NLS 1 (Aa 170-185). (A) Schematic representation of recombinant, GFP-tagged ZNF32 mutant proteins. Lines represent the deleted sequences in the proteins. (B) The subcellular localization of ZNF32 and NLS 1 mutant proteins. Recombinant proteins are shown in green (GFP), and cell nuclei are shown in blue (DAPI). (C, D) ZNF32 NLS 2 (Aa 186-199). The basic amino acids Lys and Arg were replaced with Ala in NLS 2 mutant. (C) Schematic representation of the recombinant, GFP-tagged ZNF32 mutants. Lines represent the deleted sequences in the proteins. (D) The subcellular localization of ZNF32 and NLS 2 mutant. Recombinant proteins are shown in green (GFP), and cell nuclei are shown in blue (DAPI). (E, F) ZNF32 NLS 3 (Aa 227-239). (E) Schematic representation of the recombinant, GFP-tagged ZNF32 mutants. Lines represent the deleted sequences in the proteins. (F) The subcellular localization of ZNF32 and NLS 3 mutant. Recombinant proteins are shown in green (GFP), and cell nuclei are shown in blue (DAPI). Scale bar = 50 μm.