Author Correction: Mianserin suppresses R-spondin 2-induced activation of Wnt/β-catenin signaling in chondrocytes and prevents cartilage degradation in a rat model of osteoarthritis

Correction to: Scientific Reports 10.1038/s41598-019-39393-x, published online 26 February 2019

This Article contains errors in the Reference list. Reference 21 is incorrectly listed as:

Masahiro N. et al. A genome-wide association study identifies susceptibility loci for ossification of the posterior longitudinal ligament of the spine. Nature Genetics 46(9), 1012–1016 (2014).

The correct reference is listed below as ref. 1.

Additionally, Reference 23 is incorrectly listed as:

Rita D. Hypertrophic differentiation of chondrocytes in osteoarthritis: the developmental aspect of degenerative joint disorders. Arthritis Research & Therapy 12(5), 216 (2010).

The correct reference is listed below as ref. 2.

Furthermore, Reference 25 is incorrectly listed as:

Marlies, B. et al. The Reason for Discontinuation of the First Tumor Necrosis Factor (TNF) Blocking Agent Does Not Influence the Effect of a Second TNF Blocking Agent in Patients with Rheumatoid Arthritis. The Journal of Rheumatology 36(10), 2171–2177 (2009).

The correct reference is listed below as ref. 3.

In addition, there is an error in the legend of Figure 4, where:

“The number of β-catenin-positive cells is divided by the number of DAPI signals to calculate the ratio of nuclear β-catenin-positive cells. The number of β-catenin-positive cells is divided by the number of DAPI signals to calculate the ratio of nuclear β-catenin-positive cells. *P < 0.05 by one-way ANOVA followed by Tukey’s post-hoc test. Scale = bar 200 μm (A), 100 µm (C), and 25 µm (D). (G) Model of Rspo2-mediated OA development, and the effect of mianserin.”

should read:

“The number of β-catenin-positive cells is divided by the number of DAPI signals to calculate the ratio of nuclear β-catenin-positive cells. *P < 0.05 by one-way ANOVA followed by Tukey’s post-hoc test. Scale bar = 200 μm (A), 100 µm (C), and 25 µm (D). (G) Model of Rspo2-mediated OA development, and the effect of mianserin.”