Skip to main content
. 2020 Feb 21;8:e8650. doi: 10.7717/peerj.8650

Table 2. Comparison of pathological characteristics between the two groups.

APLA2R-ab (−) (N = 59) APLA2R-ab (+) (N = 170) P value
Pathological stage (%)
I 45/59 (76.27%) 112/170 (65.88%) 0.454
II 14/59 (23.72%) 55/170 (32.35%) 0.321
III + IV 0 2/170 (1.18%) 0.293
Tissue IgG 2.46 ± 0.83 2.37 ± 0. 89 0.486
Tissue IgM 0.36 ± 0.79 0.38 ± 0.69 0.810
Tissue IgA 0.12 ± 0.50 0.23 ± 0.61 0.182
Tissue C3 1.09 ± 0.87 1.19 ± 0.94 0.459
Tissue Fib 0.08 ± 0.39 0.15 ± 0.52 0.264
Tissue C1q 0.53 ± 0.80 0.50 ± 0.71 0.796
Pathological characteristics
Global sclerosis 35/59 (59.32%) 88/170 (51.76%) 0.337
Glomerular mesangial hyperplasia 16/59 (27.12%) 64/170 (37.65%) 0.124
Crescents 2/59 (3.38%) 0 0.159
Hyperplasia endothelialitis 4/59 (6.78%) 7/170 (4.12%) 0.418
Acute renal tubular lesions 2/59 (3.39%) 20/170 (11.76%) 0.015*
Chronic renal tubular lesions 2/59 (3.39%) 21/170 (13.53%) 0.010**
Inflammatory cell infiltration 3/59 (5.08%) 23/170 (13.53%) 0.031*
Vascular disease 21/59 (35.59%) 57/170 (33.53%) 0.796

Notes.

Compared with the PLa2R− group and the PLA2R+ group, P < 0.05, P∗∗ < 0.01.