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. 2020 Apr 22;106(2):354. doi: 10.1016/j.neuron.2020.04.006

ALS/FTD-Linked Mutation in FUS Suppresses Intra-axonal Protein Synthesis and Drives Disease Without Nuclear Loss-of-Function of FUS

Jone López-Erauskin, Takahiro Tadokoro, Michael W Baughn, Brian Myers, Melissa McAlonis-Downes, Carlos Chillon-Marinas, Joshua N Asiaban, Jonathan Artates, Anh T Bui, Anne P Vetto, Sandra K Lee, Ai Vy Le, Ying Sun, Mélanie Jambeau, Jihane Boubaker, Deborah Swing, Jinsong Qiu, Geoffrey G Hicks, Zhengyu Ouyang, Xiang-Dong Fu, Lino Tessarollo, Shuo-Chien Ling, Philippe A Parone, Christopher E Shaw, Martin Marsala, Clotilde Lagier-Tourenne, Don W Cleveland ∗, Sandrine Da Cruz ∗∗
PMCID: PMC7184958  PMID: 32325059

(Neuron 100, 816–830.e1–e7; November 21, 2018)

In the original publication of this paper, the concentration of the puromycin was incorrectly reported. In both panel A of Figure 7 and the “In vivo protein synthesis labeling” section of the STAR Methods, the amount was listed as 10 μg/kg. The correct concentration should be 10 mg/kg. The authors apologize for this error.

Contributor Information

Don W. Cleveland, Email: dcleveland@ucsd.edu.

Sandrine Da Cruz, Email: sdacruz@ucsd.edu.

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