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. 2020 Apr 29;9(4):42. doi: 10.1038/s41389-020-0226-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Western blotting showed the expression levels of ACSL4, c-Myc, FBW7, SKP2 after treatment Huh7-shACSL4 cells with FBW7 or SKP2 siRNA. b, c CCK-8 and colony-formation assays for indicated Huh7 cells were evaluated. d Western blotting showed the suppression efficiency after treatment Bel-7402-pcDNA-ACSL4 and PLC/PRF5-pcDNA-ACSL4 cells with c-Myc siRNA or a specific c-Myc inhibitor 10058-F4 (20 μM). e, f CCK-8 and colony-formation assays for indicated Bel-7402 and PLC/PRF5 cells were evaluated. Data are presented as the mean ± SD (n = 3 biological replicates). ***p < 0.001. The data were analyzed using Student’s t-test.