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. 2017 Sep 23;113(14):1797. doi: 10.1093/cvr/cvx184

Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent

PMCID: PMC7190929  PMID: 29036473

Cardiovasc Res 2016;111(4):410–421]

‘We described in the Methods section that BDM was used at 100 mM. However, this was an error. In our experimental setting, BDM was always used at the concentration of 15 mM as previously described [14]. We sincerely apologize for this inconsistency and the inconvenience it may have caused.'

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