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. 2020 Aug 20;39(39):6172–6189. doi: 10.1038/s41388-020-01429-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Bar graph comparing expression of AR in 22RV1 treated with scramble siRNA or siRNA against AR. b Bar graph showing expression of AAK1, SYNE4, and MAN1A1 in LNCaP cells treated with siRNA against AR in comparison to scramble siRNA. c Bar graph showing total number of rMATS predicted splicing events in MDA-PCa-2b cells treated with siRNA against AR or scrambled control. d Upset plot comparing the genes predicted to undergo alternative splicing by rMATS or HTA-2.0 analysis in prostate cancer cells treated with casodex or enzalutamide in comparison to DHT or siRNA against AR in comparison to scrambled siRNA. e The heatmap comparing PSI for prostate cancer genes across MDA-PCa-2b cells treated with scrambled or siRNA against AR. f Bar graph revealing the GO pathway enriched for genes under undergoing ASE modulated by genomic inhibition of AR in MDA-PCa-2b.