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[Preprint]. 2020 Oct 6:2020.09.25.20201863. Originally published 2020 Sep 27. [Version 2] doi: 10.1101/2020.09.25.20201863

Deep Immune Profiling of MIS-C demonstrates marked but transient immune activation compared to adult and pediatric COVID-19

Laura Vella, Josephine R Giles, Amy E Baxter, Derek A Oldridge, Caroline Diorio, Leticia Kuri-Cervantes, Cecile Alanio, Maria Betina Pampena, Jennifer E Wu, Zeyu Chen, Yinghui Jane Huang, Elizabeth M Anderson, Sigrid Gouma, Kevin O McNerney, Julie Chase, Chakkapong Burudpakdee, Jessica H Lee, Sokratis A Apostolidis, Alexander C Huang, Divij Mathew, Oliva Kuthuru, Eileen C Goodwin, Madison E Weirick, Marcus J Bolton, Claudia P Arevalo, Andre Ramos, Cristina Jasen, Heather M Giannini, Kurt DAndrea, Nuala J Meyer, Edward M Behrens, Hamid Bassiri, Scott E Hensley, Sarah E Henrickson, David T Teachey, Michael R Betts, E John Wherry
PMCID: PMC7523167  PMID: 32995826

Abstract

Pediatric COVID-19 following SARS-CoV-2 infection is associated with fewer hospitalizations and often milder disease than in adults. A subset of children, however, present with Multisystem Inflammatory Syndrome in Children (MIS-C) that can lead to vascular complications and shock, but rarely death. The immune features of MIS-C compared to pediatric COVID-19 or adult disease remain poorly understood. We analyzed peripheral blood immune responses in hospitalized SARS-CoV-2 infected pediatric patients (pediatric COVID-19) and patients with MIS-C. MIS-C patients had patterns of T cell-biased lymphopenia and T cell activation similar to severely ill adults, and all patients with MIS-C had SARS-CoV-2 spike-specific antibodies at admission. A distinct feature of MIS-C patients was robust activation of vascular patrolling CX3CR1+ CD8 T cells that correlated with use of vasoactive medication. Finally, whereas pediatric COVID-19 patients with acute respiratory distress syndrome (ARDS) had sustained immune activation, MIS-C patients displayed clinical improvement over time, concomitant with decreasing immune activation. Thus, non-MIS-C versus MIS-C SARS-CoV-2 associated illnesses are characterized by divergent immune signatures that are temporally distinct and implicate CD8 T cells in clinical presentation and trajectory of MIS-C.

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