In the original article, there was a mistake in Figures 3, 5 and 7 as published. The marked symbols “+” and “-”in Figures 3B, C, Figures 5C, D and Figure 7C were misplaced. The corrected Figures 3, 5 and 7 appear below.
Figure 3.
Ginsenoside Re attenuated HG-induced RF/6A cell injury and oxidative stress. (A) ROS levels were monitored using a fluorescence microscope. (B) Statistical analysis of ROS fluorescence intensity. The enzymatic activities of LDH (C), MDA (D), CAT (E), and GSH-Px (F) were detected by spectrophotometry. The data are presented as the mean ± standard error of the mean (n = 5). ##P < 0.01 versus the control group; *P < 0.05, **P < 0.01 versus the HG group. Scale bar, 50 μm.
Figure 5.
Effects of Ginsenoside Re on HG-triggered apoptosis in RF/6A cells. (A) Distribution map of apoptotic cells detected by annexin V/PI double staining. (B) Representative images captured with fluorescence microscopy showing TUNEL-stained RF/6A cells. (C) Quantitative analysis of the ratio of annexin V/PI-positive cells to total cells. (D) The ratio of TUNEL-positive cells. The results are expressed as the mean ± SE of the mean (n = 5). ##P < 0.01 versus the control group; **P < 0.01 versus the HG group. Scale bar, 50 μm.
Figure 7.
Re protects RF/6A cells via regulation of the PI3K/Akt pathway. (A) Akt and p-AKT expression detected by western blot. (B) The changes of related proteins after LY294002 (PI3K inhibitor) incubation. (C) Analysis of Akt and p-Akt expression. (D–H) Statistic analysis of related protein levels. The results are presented as the mean ± SEM percentage of the control from three independent tests. ##P < 0.01 versus the control group; *P < 0.05, **P < 0.01 versus the HG group; $$P < 0.01 versus the HG+Re group.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.