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. 2021 Mar 8;11:5866. doi: 10.1038/s41598-021-85556-0

Author Correction: Cigarette smoke induces endoplasmic reticulum stress and suppresses efferocytosis through the activation of RhoA

Hiroyuki Ito 1,2, Yoshiro Yamashita 1, Takeshi Tanaka 1, Masahiro Takaki 1, Minh Nhat Le 3, Lay-Myint Yoshida 3, Konosuke Morimoto 1,2,
PMCID: PMC7940424  PMID: 33686105

Correction to: Scientific Reports 10.1038/s41598-020-69610-x, puplished online 28 July 2020

This Article contains an error in Figure 6 where the PERK inhibitor ‘GSK2606414’ is incorrectly referred to as ‘GSK2656157’. As a result, the Figure legend,

“A model of the effect of the CSE-induced ER stress on efferocytosis. ER stress and oxidative stress induced by smoking activate the PERK-eIF2α pathway. The results of the experiments using TUDCA, GSK2656157 and salubrinal highlight the therapeutic potential of TUDCA in the impairment of efferocytosis by cigarette smoke.”

should read:

“A model of the effect of the CSE-induced ER stress on efferocytosis. ER stress and oxidative stress induced by smoking activate the PERK-eIF2α pathway. The results of the experiments using TUDCA, GSK2606414 and salubrinal highlight the therapeutic potential of TUDCA in the impairment of efferocytosis by cigarette smoke.”

The correct Figure 6 appears below as Figure 1.

Figure 1.

Figure 1

A model of the effect of the CSE-induced ER stress on efferocytosis. ER stress and oxidative stress induced by smoking activate the PERK-eIF2α pathway. The results of the experiments using TUDCA, GSK2606414 and salubrinal highlight the therapeutic potential of TUDCA in the impairment of efferocytosis by cigarette smoke.


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