Annals of Neurology reference number: ANA‐18‐1177.
PMID: 30723964.
Addition to the end of the Acknowledgements section of the paper.
The dataset(s) used for the analyses described in this manuscript were obtained from the Age‐Related Eye Disease Study (AREDS) Database found at https://www.nei.nih.gov/research/clinical‐trials/age‐related‐eye‐disease‐study‐areds through dbGaP accession number phs000001.v3.p1. Funding support for AREDS was provided by the National Eye Institute (N01‐EY‐0‐2127). We would like to thank the AREDS participants and the AREDS Research Group for their valuable contribution to this research. The Framingham Heart Study is conducted and supported by the National Heart, Lung, and Blood Institute (NHLBI) in collaboration with Boston University (Contract No. N01‐HC‐25195 and HHSN268201500001I). This manuscript was not prepared in collaboration with investigators of the Framingham Heart Study and does not necessarily reflect the opinions or views of the Framingham Heart Study, Boston University, or NHLBI. Funding to support the Omni cohort recruitment, retention and examination was provided by NHLBI Contract N01‐HC‐25195 and HHSN268201500001I, as well as NHLBI grants R01‐HL070100, R01‐HL076784, R01‐HL‐49869, and U01‐HL‐053941.Research support to collect data and develop an application to support this project was provided by 3P50CA093459, 5P50CA097007, 5R01ES011740, and 5R01CA133996. The WHI program is funded by the National Heart, Lung, and Blood Institute, National Institutes of Health, U.S. Department of Health and Human Services through contracts HHSN268201600018C, HHSN268201600001C, HHSN268201600002C, HHSN268201600003C, and HHSN268201600004C. This manuscript was not prepared in collaboration with investigators of the WHI, has not been reviewed and/or approved by the Women's Health Initiative (WHI), and does not necessarily reflect the opinions of the WHI investigators or the NHLBI. Funding support for WHI GARNET was provided through the NHGRI Genomics and Randomized Trials Network (GARNET) (Grant Number U01 HG005152). Assistance with phenotype harmonization and genotype cleaning, as well as with general study coordination, was provided by the GARNET Coordinating Center (U01 HG005157). Assistance with data cleaning was provided by the National Center for Biotechnology Information. Funding support for genotyping, which was performed at the Broad Institute of MIT and Harvard, was provided by the NIH Genes, Environment and Health Initiative [GEI] (U01 HG004424). The datasets used for the analyses described in this manuscript were obtained from dbGaP at http://www.ncbi.nlm.nih.gov/sites/entrez?db=gap through dbGaP accession phs000196, phs000292 phs000372, phs000394, phs000397, phs000428, phs000615, phs000315 and phs000675. The authors acknowledge the contribution of data from Hepatitis C Pathogenesis and the Human Genome supported by 1X01HG005271–01 and R01DA013324 and accessed through dbGAP to the analysis presented in this publication. Funding support for the Genes and Blood Clotting Study was provided through the NIH/NHLBI (R37 HL039693). The Genes and Blood Clotting Study is one of the Phase 3 studies as part of the Gene Environment Association Studies (GENEVA) under GEI. Assistance with genotype cleaning was provided by the GENEVA Coordinating Center (U01 HG004446). Funding support for DNA extraction and genotyping, which was performed at the Broad Institute, was provided by NIH/NHLBI (R37 HL039693). Additional support was provided by the Howard Hughes Medical Institute. The dataset(s) used for the analyses described in this manuscript were obtained from the database of Genotype and Phenotype (dbGaP) found at http://www.ncbi.nlm.nih.gov/gap through dbGaP accession number phs000368. Samples and associated phenotype data for the Genome‐Wide Association Scan [GWAS] of Polycystic Ovary Syndrome Phenotypes were provided by Andrea Dunaif, M.D. The authors acknowledge the contribution of data from Genetic Architecture of Smoking and Smoking Cessation accessed through dbGAP. Funding support for genotyping, which was performed at the Center for Inherited Disease Research (CIDR), was provided by 1 X01 HG005274‐01. CIDR is fully funded through a federal contract from the National Institutes of Health to The Johns Hopkins University, contract number HHSN268200782096C. Assistance with genotype cleaning, as well as with general study coordination, was provided by the Gene Environment Association Studies (GENEVA) Coordinating Center (U01 HG004446). Funding support for collection of datasets and samples was provided by the Collaborative Genetic Study of Nicotine Dependence (COGEND; P01 CA089392) and the University of Wisconsin Transdisciplinary Tobacco Use Research Center (P50 DA019706, P50 CA084724). The dataset(s) used for the analyses described in this manuscript were obtained from the Genetics of Fuchs' Endothelial Corneal Dystrophy (FECD) Study through dbGaP accession number phs000421. The grants that have funded the enrollment of the cases and controls to be used in this GWAS are:R01EY016514 (DUEC, PI: Gordon Klintworth), R01EY016482 (CWRU, PI: Sudha Iyengar), and 1X01HG006619–01 (PI: Sudha Iyengar, Natalie Afshari). We would like to thank the FECD participants and the FECD Research Group for their valuable contribution to this research. The authors acknowledge the contribution of data from CIDR‐NIDA Study of HIV Host Genetics accessed through dbGAP. Funding support for genotyping, which was performed at the Center for Inherited Disease Research (CIDR), was provided by 1 X01 HG005275‐01A1. Funding support for collection of datasets and samples was provided by NIDA grants R01DA026141(Johnson); R01DA004212 (Watters); U01DA006908 (Watters); R01DA009532 (Bluthenthal); as well as the San Francisco Department of Public Health; SAMHSA; and HRSA. The Genome‐Wide Association Study (GWAS) of Non‐Hodgkin Lymphoma (NHL) project was supported by the intramural program of the Division of Cancer Epidemiology and Genetics (DCEG), National Cancer Institute (NCI), National Institutes of Health (NIH). The datasets have been accessed through the NIH database for Genotypes and Phenotypes (dbGaP) under accession # phs000801. A full list of acknowledgements can be found in the supplementary note (Berndt SI et al., Nature Genet., 2013, PMID: 23770605). This study made use of data generated by investigators in the BEACON consortium through a grant funded by the US National Institutes of Health (NIH) (RO1CA136725) to Thomas L. Vaughan and David C. Whiteman (multiple PIs). In support of this work, T.L.V. was also supported by NIH grant KO5CA124911 and D.C.W. by a Future Fellowship grant FT0990987 from the Australia Research Council. Additional collaborators, sources of support and origin of the data and biospecimens are listed in the following publication: Levine DM, Ek WE, Zhang R, Liu X, Onstad L, Sather C, et al. A genome‐wide association study identifies new susceptibility loci for esophageal adenocarcinoma and Barrett's esophagus. Nat Genet. 2013 Dec;45(12):1487–93.