In the original article, there was an error. The original article read: Recent study shows that host ROS can be sensed as a chemorepellent in H. pylori by the chemoreceptor TlpD, which initiates chemotaxis to promote gastric gland colonization (Collins et al., 2018; Perkins et al., 2019).
A correction has been made to the section, Thrive Under ROS Conditions by Metabolic Remodeling. The corrected sentence is below:
Recent studies showed that ROS could be sensed in H. pylori by the chemoreceptor TlpD. Host oxidants hypochlorous acid (HOCl) could act as a chemoattractant by reversibly oxidizing TlpD that inactivates the chemotransduction signaling complex (Perkins et al., 2019). While H2O2 could act as a chemorepellent which initiates chemotaxis through TlpD to promote gastric gland colonization (Collins et al., 2018).
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
References
- Collins K. D., Hu S., Grasberger H., Kao J. Y., Ottemann K. M. (2018). Chemotaxis allows bacteria to overcome host-generated reactive oxygen species that constrain gland colonization. Infect. Immun. 86:e00878-17. [DOI] [PMC free article] [PubMed] [Google Scholar]
- Perkins A., Tudorica D. A., Amieva M. R., Remington S. J., Guillemin K. (2019). Helicobacter pylori senses bleach (HOCl) as a chemoattractant using a cytosolic chemoreceptor. PLoS Biol. 17:e3000395. 10.1371/journal.pbio.3000395 [DOI] [PMC free article] [PubMed] [Google Scholar]