Author Correction: Increased Ca²⁺ content of the sarcoplasmic reticulum provides arrhythmogenic trigger source in swimming-induced rat athlete’s heart model

Correction to: Scientific Reports 10.1038/s41598-020-76496-2, published online 11 November 2020

This Article contains errors in the Results section under subheading ‘The I_Ca,L, SR Ca²⁺ content and Ca²⁺ transient measurements’.

“We analyzed the integral of caffeine-induced NCX currents as an indicator of the SR Ca²⁺ content and found that SR Ca²⁺ content was significantly increased in the trained group compared to controls (− 1.84 ± 0.4 (pA*s)/pF vs − 1.25 ± 0.5 (pA*s)/pF n = 8/5 and 8/4, respectively, p < 0.05; Fig. 6b).”

should read:

“We analyzed the integral of caffeine-induced NCX currents as an indicator of the SR Ca²⁺ content and found that SR Ca²⁺ content was significantly increased in the trained group compared to controls (− 1.84 ± 0.4 (pA*s)/pF vs − 1.25 ± 0.5 (pA*s)/pF n = 8/5 and 8/5, respectively, p < 0.05; Fig. 6b).”

Secondly, this Article contains an error in Figure 6 where the experimental numbers of the control columns are incorrect (8/4) in panel (b). The correct Figure 6 appears below as Figure 1.

Figure 1.

Assessment of Ca²⁺ handling on isolated cells. Panel (a) shows identical current–voltage relationship of L-type Ca²⁺ current between groups. Panel (b) illustrates significantly larger inward current as a response of 10 mM caffeine application. Panel (c,d) reports larger Ca²⁺ transient amplitude in the case of trained rats (red trace) compared to control (black trace). Panel (e) indicates faster transient relaxation kinetics in the case of trained animals.