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. 2021 Sep 1;40(17):e109067. doi: 10.15252/embj.2021109067

Inactive variants of death receptor p75NTR reduce Alzheimer’s neuropathology by interfering with APP internalization

Chenju Yi, Ket Yin Goh, Lik‐Wei Wong, Ajeena Ramanujan, Kazuhiro Tanaka, Sreedharan Sajikumar, Carlos F Ibáñez
PMCID: PMC8408598  PMID: 34467558

Abstract

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Correction to: The EMBO Journal (2021) 40: e104450. DOI 10.15252/embj.2020104450 | Published online 1 December 2020

The authors correct Figure 6A of this paper. During the revision process, images from p75NTR‐expressing mice were inadvertently used in place of p75NTR knock‐out neurons. The corrected figure, showing lack of p75NTR labeling in knock‐out neurons, along with their corresponding internalized APP, is shown here. This error only concerns the images used to illustrate the quantitative data. It does not affect the analysis itself nor the conclusions derived from it. The authors apologize for this oversight and agree with this corrigendum; no response could be obtained from KT.

Figure 6A. Original.

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Figure 6A. Corrected.

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Correction to: The EMBO Journal (2021) 40: e104450. DOI 10.15252/embj.2020104450 | Published online 1 December 2020


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