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American Journal of Cancer Research logoLink to American Journal of Cancer Research
. 2021 Sep 15;11(9):4638–4640.

Cilostazol eliminates radiation-resistant glioblastoma by re-evoking big conductance calcium-activated potassium channel activity

Chan-Chuan Liu 1, Cheng-Lin Wu 3, I-Chun Yeh 6, Sheng-Nan Wu 2, Chun-I Sze 1,3,*, Po-Wu Gean 1,4,5,*
PMCID: PMC8493387  PMID: 34659911

In this article, we found the mistakes in Figures 12A and 13. These mistakes did not affect the results, the findings, and conclusion of this study. Therefore, we would like to correct the mistakes and replaced the images. The correct Figures 12A and 13 are as follows.

Figure 12.

Figure 12

Cilostazol inhibits radiation-resistant 1306MG 3.5GR6 growth in an intracranial tumor model. A. Lucexpressing radiation-resistant 1306MG 3.5GR6 cells (5 × 105 cells) were injected intracranially into the striatum of NOD-SCID mice. Tumor growth was monitored using an IVIS-200 imaging system; B. The histogram indicating tumor growth by evaluating bioluminescent photons. Tumor growth was slower in the Cilostazol-treated mice than in the vehicle-control mice; means ± SEM; n = 3 in vehicle and Cilostazol groups; * means comparison with vehicle control; **, p<0.01; C. The histogram indicating the changes of body weight. Cilostazol did not alter the body weight.

Figure 13.

Figure 13

Summary of the alternation of BK channel activity in developing radiation resistance in GBM.


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