Correction to: Genome Biol 22, 297 (2021).
10.1186/s13059-021-02513-w
Following publication of the original article [1], the authors identified an error in the author affiliations presented in additional file 1. The additional file has been updated and published in this correction.
The original article [1] has been corrected.
Supplementary Information
Additional file 1: Fig. S1. Construction of CRISPR genome-wide and surfaceome libraries. Fig. S2. Quality analyses of constructed genome-wide and surfaceome CRISPR libraries. Fig. S3. Evaluation of surfaceome and genome-wide CRISPR libraries. Fig. S4. Validation of the screening results. Fig. S5. Determination of gene modification efficiency. Fig. S6. Validation of the top 10 hits from surfaceome and genome-wide screens. Fig. S7. Construction and validation of single clones of ICAM-1−/−, RAB5C−/−, OLFML3−/−, SLC4A7−/− and ATP6AP1−/+ H1-Hela cells. Fig. S8. Validation of the effects of ICAM-1, RAB5C and OLFML3 on RV infection, related to Fig. 3. Fig. S9. Dissection of the functions of RAB5C and OLFML3 in RV infection. Fig. S10. RNA-seq analyses of the effects of RAB5C knockout on RV infection. Fig. S11. RNA-Seq analyses of the effects of OLFML3 on RV infection (related to Fig. 4). Fig. S12. Bar plots showing RT-qPCR quantification of ISG expression in mock and OLML3−/− cells at 24 h post infection of RV-B14 (a) and RV-A16 (b) at an MOI of 2
Footnotes
Publisher’s Note
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Hong Mei, Zhao Zha and Wei Wang contributed equally to this work.
Contributor Information
Jieming Qu, Email: jmqu0906@163.com.
Jia Liu, Email: liujia@shanghaitech.edu.cn.
Reference
- 1.Mei H, Zha Z, Wang W, Xie Y, Huang Y, Li W, Wei D, Zhang X, Qu J, Liu J. Surfaceome CRISPR screen identifies OLFML3 as a rhinovirus-inducible IFN antagonist. Genome Biol. 2021;22(1):297. doi: 10.1186/s13059-021-02513-w. [DOI] [PMC free article] [PubMed] [Google Scholar]
Associated Data
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Supplementary Materials
Additional file 1: Fig. S1. Construction of CRISPR genome-wide and surfaceome libraries. Fig. S2. Quality analyses of constructed genome-wide and surfaceome CRISPR libraries. Fig. S3. Evaluation of surfaceome and genome-wide CRISPR libraries. Fig. S4. Validation of the screening results. Fig. S5. Determination of gene modification efficiency. Fig. S6. Validation of the top 10 hits from surfaceome and genome-wide screens. Fig. S7. Construction and validation of single clones of ICAM-1−/−, RAB5C−/−, OLFML3−/−, SLC4A7−/− and ATP6AP1−/+ H1-Hela cells. Fig. S8. Validation of the effects of ICAM-1, RAB5C and OLFML3 on RV infection, related to Fig. 3. Fig. S9. Dissection of the functions of RAB5C and OLFML3 in RV infection. Fig. S10. RNA-seq analyses of the effects of RAB5C knockout on RV infection. Fig. S11. RNA-Seq analyses of the effects of OLFML3 on RV infection (related to Fig. 4). Fig. S12. Bar plots showing RT-qPCR quantification of ISG expression in mock and OLML3−/− cells at 24 h post infection of RV-B14 (a) and RV-A16 (b) at an MOI of 2