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Elsevier - PMC COVID-19 Collection logoLink to Elsevier - PMC COVID-19 Collection
. 2021 Dec 30;33:105103. doi: 10.1016/j.rinp.2021.105103

Modeling the dynamics of COVID-19 using fractal-fractional operator with a case study

Jian-Cun Zhou a,b, Soheil Salahshour c,, Ali Ahmadian d,e, Norazak Senu f
PMCID: PMC8716155  PMID: 34980997

Abstract

This research study consists of a newly proposed Atangana–Baleanu derivative for transmission dynamics of the coronavirus (COVID-19) epidemic. Taking the advantage of non-local Atangana–Baleanu fractional-derivative approach, the dynamics of the well-known COVID-19 have been examined and analyzed with the induction of various infection phases and multiple routes of transmissions. For this purpose, an attempt is made to present a novel approach that initially formulates the proposed model using classical integer-order differential equations, followed by application of the fractal fractional derivative for obtaining the fractional COVID-19 model having arbitrary order Ψ and the fractal dimension Ξ. With this motive, some basic properties of the model that include equilibria and reproduction number are presented as well. Then, the stability of the equilibrium points is examined. Furthermore, a novel numerical method is introduced based on Adams–Bashforth fractal-fractional approach for the derivation of an iterative scheme of the fractal-fractional ABC model. This in turns, has helped us to obtained detailed graphical representation for several values of fractional and fractal orders Ψ and Ξ, respectively. In the end, graphical results and numerical simulation are presented for comprehending the impacts of the different model parameters and fractional order on the disease dynamics and the control. The outcomes of this research would provide strong theoretical insights for understanding mechanism of the infectious diseases and help the worldwide practitioners in adopting controlling strategies.

Keywords: COVID-19 transmission, Fractal fractional order model, Deterministic stability analysis, Case study, Adams–Bashforth method, Newton polynomial

Introduction

The recent but well-studied COVID-19 outbreak has resulted large-scale sever unexpected consequences, that ultimately impacted the global community from human’s health as well as financial perspectives. For the apprehension of such contagious diseases, legislatures have sanctioned extraordinary measures, including quarantines, curfews, lockdowns, and restrictions on local and international movements. Indeed, lack of dependable information in concerning the sickness transmission has essentially prompted cautious reactions. These newly arose concerns, which have required significant choices dependent on figures, have exhibited more than any time in recent memory the requirement for solid instruments planned to demonstrate the spread of COVID-19 and other irresistible sicknesses [1], [2], [3]. An especially dire need is the geo-confinement of episodes, as this might permit a more inactive designation of clinical assets.

At present, the accessibility to legitimate and successful treatment is scars for a COVID-19 infected individuals with the exception of certain medications as Remdesivir which are endorsed by certain countries including Australia and the European Union [4]. According to the literature, and industrial experts, there exists no influential and approved antibody for this novel contamination albeit not many nations have guaranteed it. The best avoidance methodologies utilized in certain regions for the complete control are the successive tests to decide the infected people, detachment and lockdown, social separating, utilization of severe Standard Operating Procedures (SOPs), and so forth until successful medicines and antibody become accessible. The preventive measures have been proved to be one of the effective tools in controlling the faster transmission of the contagious diseases. For this purpose, the academic researchers, and the pharmaceutical experts are putting their considerable efforts. Several approaches have been used to investigate the transmission mechanism of these infectious diseases. For understanding the mechanism and theoretical implementation, numerical modeling has proved to be an effective tool for these diseases. Numerous epidemic models have also been introduced to investigate the dynamic of the COVID-19 to present various controlling strategies around the globe. For example, a model of COVID-19 with Lockdown is proposed in [5], and the effect of undetected cases by means of a numerical model is investigated in [6]. The effect of some preventive measure on the reducing the COVID-19 in Pakistan by means of another numerical model is introduced in [7], [8]. A transmission numerical model considering the ecological spread of the infection with a contextual analysis of Saudi Arabia is studied in [9].

A recent contribution to fractional calculus was made by Atangana and Baleanu, “who presented operators based on generalized Mittag-Leffler functions to solve fractional integrals and derivatives [10], as the Mittag-Leffler function is more suitable in expressing nature than power function. It can be recalled that the Mittag–Leffler function has been introduced to provide a response to the conventional question of complex analysis, in particular to portray the procedure of the analytic continuation of power-law series outside the disc of their convergence. Since 2016, the Atangana–Baleanu operators have inspired an explosion of new research in fractional calculus. This work is growing at a remarkable rate in the fields of mathematics, science, and engineering”. The Atangana–Baleanu derivative is a nonlocal fractional derivative with a nonsingular kernel that is connected with a variety of applications.

A powerful tools that described the real world situation in mathematical concept and terminology is known as mathematical modeling. “The different aspects for the majority of biological and general dynamics are well described via aforementioned techniques of mathematics. In this regards, the researchers use the tools of mathematical modeling to study the transmission and make further plan to prevent the mankind form the effects of mentioned infectious disease. In this regards, many researchers developed different mathematical models for the current COVID-19”, for detail see [11], [12], [13], [14].

A large portion of the mathematical models of COVID-19 are formulated in terms of the integer order derivatives which have a few restrictions to portray the realistic aspects of a phenomena under consideration. To manage those constraints, non-integer order derivatives provide a practical mean to the sickness dynamic and beneficial results that need to comprehend the models. non-integer order models have memory appropriateness and give a superior situation to depict an epidemic model. Many mathematical models on the elements various illnesses in term of non-integer order derivatives were proposed see for occasion [15], [16], [17], [18], [19], [20], [21], [22], [23], [24] and the literature referenced therein. Fractal fractional calculus is the generalization of classical calculus [25], [26], [27], [28], [29]. To get a better insight into a mathematical model and to deeply understand phenomena, non-integer order operators can be used.

For the ease of understanding this research is organized as follows: the mathematical model with fractal fractional-order derivative is formulated in Section “Fractional COVID-19 transmission models”. In Section “Equilibria and basic reproductive number (R0)”, the equilibrium points and basic reductive number R0 are presented. The local stability of the disease-free and endemic equilibria for the determinist version model are presented in Section “Stability analysis”. Furthermore, the parameters estimation is shown in Section “Case study”. The existence and qualitative analysis with Hyers–Ulam Stability of fractional-order model in the sense of ABC presented in Section “Qualitative analysis of the COVID-19 model”. The numerical schemes and discussions are presented in Section “Simulation results & numerical schemes”, and in the last section we presented the concluding remarks.

Fractional COVID-19 transmission models

A compartmental approach is used to develop the mathematical model for COVID-19 transmission dynamics. “The total population N is divided into six compartments named S,E,I,A,H,G, and R represent susceptible, exposed, symptomatically infected, asymptomatically infected, isolated, or hospitalized, and Recovered/immune cases respectively. In the mathematical model developed in this study, humans get into the suspected group S at the rate of G and infected with Coronavirus as a result of contact with individuals in the group of A or I.. The exposed group E gains population from infection induced by the Coronavirus. A proportion G3,0<G3<1 of the members of the group E advance to the asymptomatic group A and the remaining proportion 1G3 progresses to the symptomatic group I. People in the group I and A progress either to the Hospitalization group H or recovery group R at the rates indicated in Table 1. In the construction of the mathematical model, the exposed compartment E is included because people who are contracted with the virus do not get infectious immediately; there is an incubation period for the virus to get infectious. The groups I and A are included in the model, as people infected with Coronavirus are either symptomatic or asymptomatic. COVID-19 induced death rate G11 is also considered in the model. As a result, the authors are convinced that the model considered in this study named SEIAHR model incorporates all essential components of COVID-19 to study its transmission dynamics, in agreement with the definition of a mathematical model in” [30]. The mathematical model used in this study called SEIAHR model is shown in (1),

S˙(t)=GG1G2I(t)+AS(t)NG8S(t),E˙(t)=G1G2I(t)+AS(t)NG4+G8E(t),I˙(t)=1G3G4E(t)G5+G6+G8I(t),A˙(t)=G3G4E(t)G10+G7+G8A(t),H˙(t)=G5I(t)+G10A(t)G11+G9+G8H(t),R˙(t)=G6I(t)+G7A(t)+G11+G9H(t)G8R(t). (1)

with initial condition

S(0)0,E(0)0,I(0)0,A(0)0,H(0)0,R(0)0. (2)

The detail of the used unknown variables and parameters are given below in Table 1, Table 2 respectively:

Table 1.

Variables description.

Variables Description
S The class of susceptible individuals
E The class of exposed individuals
I The class of symptomatic infected individuals
A The class of asymptomatic infected individuals
H The class of Hospitalized individuals
R The class of Recovered individuals

Table 2.

Descriptions and numerical values of the parameters.

Symbols Description Values Ref.
G Influx rate 80.89 [30]
G1 Transmission rate from A to S group 0.25 [30]
G2 Transmission rate from I to S group 1 [30]
G3 The proportion of A cases 0.80 [30]
G4 The incubation period of Coronavirus 0.1923 [30]
G5 The rate at which I cases are transferred to H cases 0.6000 [30]
G6 The cure rate of I cases 0.05 [30]
G7 The cure rate of A cases 0.0714 [30]
G8 Natural mortality rate 0.0004563 [30]
G9 The rate at which H cases are transferred to R case 0.04255 [30]
G10 The rate at which A cases are transformed into H cases 0.03 [30]
G11 Coronavirus induced death rate 0.0018 [30]

Recently, it has been studied that the theory of fractional-calculus is rich for applications and researchers obtained more accurate results through fractional system rather than ordinary systems. Hence, we structured the above model (1) of COVID-19 infection in the framework of new fractal fractional derivative with a generalized Mittag-Leffler kernel as follows:

FFMD0,tΨ,Ξ[S(t)]=GG1G2I(t)+AS(t)NG8S(t),FFMD0,tΨ,Ξ[E(t)]=G1G2I(t)+AS(t)NG4+G8E(t),FFMD0,tΨ,Ξ[I(t)]=1G3G4E(t)G5+G6+G8I(t),FFMD0,tΨ,Ξ[A(t)]=G3G4E(t)G10+G7+G8A(t),FFMD0,tΨ,Ξ[H(t)]=G5I(t)+G10A(t)G11+G9+G8H(t),FFMD0,tΨ,Ξ[R(t)]=G6I(t)+G7A(t)+G11+G9H(t)G8R(t). (3)

With initial condition

S(0)0,E(0)0,I(0)0,A(0)0,H(0)0,R(0)0. (4)

Where the symbol FFMD0,tΨ,Ξ represents the fractal fractional order derivative with fractional order 0<Ψ1 and the fractal dimension Ξ>0. Now, applying the AB fractional integral to both sides of (3), we obtained the following system

S(t)S(0)=Ξ(1Ψ)tΞ1M(Ψ)F1(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F1(s,S,E,I,A,H,R)ds,E(t)E(0)=Ξ(1Ψ)tΞ1M(Ψ)F2(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F2(s,S,E,I,A,H,R)ds,I(t)I(0)=Ξ(1Ψ)tΞ1M(Ψ)F3(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F3(s,S,E,I,A,H,R)ds,A(t)A(0)=Ξ(1Ψ)tΞ1M(Ψ)F3(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F3(s,S,E,I,A,H,R)ds,H(t)H(0)=Ξ(1Ψ)tΞ1M(Ψ)F4(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F4(s,S,E,I,A,H,R)ds,R(t)R(0)=Ξ(1Ψ)tΞ1M(Ψ)F5(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F5(s,S,E,I,A,H,R)ds. (5)

Equilibria and basic reproductive number (R0)

In order to proceeds the dynamical behavior analysis, we firstly present some basic theoretical properties of the proposed model (1), including basic reproductive number, disease free and endemic equilibria. Additionally, an analytical expression for the important biological parameter termed as the basic reproductive number is provided. We obtained the following two equilibrium points for the proposed model (1):

Disease-free equilibrium point (DFE)

The proposed epidemiological model (1) of the COVID-19 is examined for the disease free equilibrium, for this purpose let N0 is the disease free equilibrium of the proposed model (1), then for analyzing this point the population under consideration is assumed to be infection free. Thus the system reported by N0=GG8,0,0,0,0,0. where S0=GG8.

Basic reproductive number R0

The endemic equilibrium (EE) of the COVID-19 vaccine model (1) denoted by To track down R0 for the above model (1), we utilize the next generation matrix technique [1], [31]. The Jacobian matrix around the DFE point N0 is given by:

J(E0)=G80G1G2G1000G4+G8G1G2G1000J320000G3G40G10+G7+G80000G5G10G11+G9+G8000G6G7G11+G9G8, (6)

where J32=1G3G4G5+G6+G8.

Now, we decompose the above matrix in the form of T and V such that M=TV, where

M=G4+G8G1G2G101G3G4G5+G6+G800G3G40G10+G7+G800G5G10G11+G9+G8. (7)
T=0G1G2G10000000000000, (8)

and

V1=1G4+a80001G3φG4+G8G5+G6+a81G5+G6+G800G3G4G4+G8G10+G9+G801G10+G7+G80V41G5G5+G6+G5G11+G9+G810G10+G7+G8G11+G9+G81G11+G9+G8 (9)

where, V41=G51G3G4G10+G7+G8+G10G3G4a5+G6+G8G4+G8G5+G6+G8G10+G7+G8G11+G9+G8. The dominant eigenvalue of ρTV1 is called the basic reproductive number, and is given by

R0=G1G21G3G4G4+G8G5+G6+G8+G1G3G4G4+G8G10+G7+G8. (10)

R0 can be written as R0=R1+R2,R1=G1G2Y2Y1Y3,R2=G1G3G4Y1Y4 where Yi,i=16 are as defined above.

Endemic equilibrium point (EE)

Let N is the endemic equilibrium of model (1), then the solution of the resultant algebraic equations will leads to the endemic equilibrium which define, N=S,E,I,A,H,R, where

S=GG8+Y1G8E,I=Y2Y3E,A=G3G4Y4E,H=1Y5G5Y2Y3+G10G4G3Y4E,R=1G8G6Y2Y3G7G3G4Y4+Y6Y5G5Y2Y3+G10G4G3Y4E, (11)

and E=m2m1=G8NGR0Y1R0, which is a solution of a quadratic equation m1E2+m2E=0, where,

m1=Y1Y1NG8R0,m2=Y1NG8GRoNG8,R0=R1+R2,R1=G1G2Y2Y1Y3,R2=G1G3G4Y1Y4,Y1=G4+G8,Y2=1G3G4,Y3=G5+G6+G8,Y4=G10+G7+G8,Y5=G11+G9+G8,Y6=G11+G9. (12)

Thus, for R0>1 a positive EE exists, with the assumption that G=G8N.

Stability analysis

The previous section presented the basic reproductive number, disease free and endemic equilibria of the proposed model (1). This analysis provides a clue for suggesting a better analysis of the dynamical behavior of the model. Thus regarding the local as well as global analysis of the proposed model we have the following stability results.

Stability analysis of DFE

Theorem 1

The proposed model (1) is locally asymptotically stable (LAS) at DFE, if |arg(λ)|>π2M for all roots λ of the following associated equation,

detdiagFMGFMGFMGFMGFMGFMGJN0=0. (13)

Proof

The jacobian matrix of system (3) around the DFE is given above (6), has the following characteristic equation:

FM+G82FM+G8+G9+G11(F3M+a2F2M+a1FM+a0)=0, (14)

where

a2=Y1+Y3+Y4.a1=Y1Y3+Y1Y4+Y3Y4G1G4G3G1G2Y2,a0=Y1Y3Y4+G1G4G3Y3+G1G2Y2Y4, (15)

where Yi,i=16 are as defined above in Eq. (12). From (14), we have a2>0 and a1=Y1Y31R1+Y1Y41R2+Y3Y4>0 for R0<1 as R1 and R2 are positive and R1+R2=R0 Besides, a0=Y1Y3Y4+G1G4G3Y3+G1G2Y2Y4=Y1Y3Y4R01>0 for R0<1, since Y1Y3Y4<0. Further more, the eigenvalues of the equation

Ψ(F)=F3M+a2F2M+a1FM+a0=0, have a negative real part, if the Routh–Hurwitz stability condition a0a2a1<0 and a0,a2,a1>0 are satisfied. That is,

a0a1a2=Y1Y3Y4R01Y1+Y3+Y4Y1Y31R1+Y1Y41R2+Y3Y4,=2Y1Y3Y4+Y1+Y3Y1Y31R1+Y1+Y4Y1Y41R2+Y4+Y3Y4Y3<0. (16)

For R1<1,R2<1,R0<1. The argument of the root of equations

F1M+G8=0,
F2M+G8=0,
F3M+G8+G9+G11=0,

are similar, that is:

[|arg(δk)|>πm+k2πm>πM>π2M],

where k=0,1,2,3,,(m1).

Similarly, we can find the arguments of the roots of the equation

F3M+a2F2M+a1FM+a0=0.

are all greater than π2M if R0<1, having an argument less than π2M for R0>1. Thus, for R0<1 the DFE N0 is LAS.

Stability analysis of EE

Theorem 2

If R0>1 , then the EE of model (1) LAS.

Proof

Since, we know that for R0>1 the EEP exists. Further the Jacobian matrix J at EEP is given by:

JEEP=Y7G80G2Y8Y800Y7Y1G2Y8Y8000Y2Y30000G3G40Y40000G5G10Y5000G6G7Y6G8, (17)

where Y1=G4+G8,Y2=1G3G4,Y3=G5+G6+G8,Y4=G10+G7+G8

Y5=G11+G9+G8,Y6=G11+G9,Y7=G1G2I+AN,Y8=G1SN.

The two eigenvalues λ1=G8 and λ2=Y5=G11+G9+G8 of the matrix (17) are negative. Further more, for the remaining eigenvalues we can utilize the following equation

f(λ)=λ4+B3λ3+B2λ2+B1λ+B0, (18)

where

B3=G8Y1Y3Y4+Y7,B2=Y1Y3+Y1Y4+Y3Y4Y1Y7Y3Y7Y4Y7G8Y1G8Y3G8Y4G4G3Y8G2Y2Y8,B1=G8Y1Y3+G8Y1Y4+G8Y3Y4Y1Y3Y4+Y1Y3Y7+Y1Y4Y7+Y3Y4Y7G4G8G3Y8G2G8Y2Y8,+G4G3Y3Y8+G2Y2Y4Y8B0=G8G4G3Y3Y8G8Y1Y3Y4Y1Y3Y4Y7+G2G8Y2Y4Y8. (19)

The coefficient B3 can easily be shown to be positive and B2,B1,B0 are also positive as shown below:

B2=Y1Y3R2+Y1Y4R1/R0+Y3Y4Y1Y7Y3Y7Y4Y7G8Y1G8Y3G8Y4>0,B1=G8Y1Y3R2/R0+G8Y1Y4R1/R0+G8Y3Y42Y1Y3Y4+Y1Y3Y7+Y1Y4Y7+Y3Y4Y7>0,B0=G8G4G3Y3Y8G8Y1Y3Y4Y1Y3Y4Y7+G2G8Y2Y4Y8=Y1Y3Y4Y7>0. (20)

Since it is not hard to show that B0B32B12B1B2B3<0, the Routh–Hurwitz stability conditions for Eq. (18) are satisfied. Thus all the eigenvalues of the Eq. (18) have a negative real part. Accordingly, the EEP N is LAS for R0>1.

Case study

The parameters used in the system (1) are estimated depend on the total number of conformed incidents, and deaths data in Khyber Pukhtunkhwa Pakistan. The ordinary Least Square Solution (OLS) is utilized to reduce the error terms for the daily reports, and the related relative error is used in the goodness of fit.

mini=1nIiIˆl2ι=1nIι2 (21)

where Ii is the reported total number of infected, and Iˆi is the simulated total number of infected. The simulated cumulative number of infected are calculated by summing the individuals transit from the infected compartment to the recovered compartment for each day. The Fig. 1 shows the fit of model to the data. Estimated values of parameters are shown in Table 3.

Fig. 1.

Fig. 1

The graphical results show the reported data for the novel corona virus disease in the district Swat Khyber Pukhtunkhwa Pakistan from 10th January 2020 to 1th March 2021 versus model fitting.

Table 3.

Explanation of the parameters given in model (1).

Parameter Value Source
G 120.0166 Fitted
G1 7.7110 Estimated
G2 6.6110 Fitted
G3 0.1573 Fitted
G8 4.37 Fitted
G4 0.212 Fitted
G5 0.0081 Fitted
G11 0.4166 Fitted
G10 2.0166 Fitted
G9 0.66 Fitted
G7 0.0166 Estimated
G6 1.2081 Estimated

Qualitative analysis of the COVID-19 model

In the present section, we are going prove the uniqueness, existence, Ulam–Hyers stability of the solution for the proposed model with help of fixed point approaches. Before that, we rewrite the model (3) as

FFMD0Ψ,ΞS(t)=F1(t,S,E,I,A,H,R),FFMD0Ψ,ΞE(t)=F2(t,S,E,I,A,H,R),FFMD0Ψ,ΞI(t)=F3(t,S,E,I,A,H,R),FFMD0Ψ,ΞA(t)=F4(t,S,E,I,A,H,R),FFMD0Ψ,ΞH(t)=F5(t,S,E,I,A,H,R),FFMD0Ψ,ΞR(t)=F5(t,S,E,I,A,H,R). (22)

Where

F1(t,S,E,I,A,H,R)=GG1G2I+ASNG8S,F2(t,S,E,I,A,H,R)=G1G2I+ASNG4+G8E,F3(t,S,E,I,A,H,R)=1G3G4EG5+G6+G8I,F4(t,S,E,I,A,H,R)=G3G4EG10+G7+G8A,F5(t,S,E,I,A,H,R)=G5I+G10AG11+G9+G8H,F6(t,S,E,I,A,H,R)=G6I+G7A+G11+G9HG8R. (23)

We will communicate (23) with the below identical system

FFMD0Ψ,ΞW(t)=ϖ(t,W(t)),W(0)=Ψ00 (24)

where

W(t)=S(t)E(t)I(t)A(t)H(t)R(t),W0(t)=S0(t)E0(t)I0(t)A0(t)H0(t)R0(t),ϖ(t,W(t))=F1(t,S,E,I,A,H,R)F2(t,S,E,I,A,H,R)F3(t,S,E,I,A,H,R)F4(t,S,E,I,A,H,R)F5(t,S,E,I,A,H,R)F6(t,S,E,I,A,H,R). (25)

The system (22) can be turned to the following formula,

W(t)=W0+Ξ(1Ψ)tΞ1ϖ(t,W(t))M(Ψ)+ΞΨM(Ψ)0tϑΨ1(tϑ)Ψ1ϖ(ϑ,W(ϑ))dϑ. (26)

Next, for the analysis, the below assumptions H1 and H2 should be fulfilled:

  • H1:ϖ:J×FR is continuous and there exists two constants τϖ,ηϖ>0 such that |ϖ(t,W(t))|τϖ+|W(t)|ηϖ for tJ and WF.

  • H2 : there should be exists constant Lϖ>0 such that |ϖ(t,W1(t))ϖ(t,W2(t))|Lϖ|W1(t)W2(t)|, for tJ and WF.

Theorem 3

Assume thatH1andH2holds. Then Eq. (24) identical to the system (22) has a solution, provided that

Ξ(1Ψ)TΞ1M(Ψ)Lϖ<1 (27)

and

Δ1=|W1|+Ξ(1Ψ)TΞ1M(Ψ)+ΞΨB(Ψ,Ξ)TΞ+Ψ1M(Ψ) (28)

Proof

We turn the given system (22) into a fixed point problems, i.e

W=ΦW,WF Where the operator Φ:FF defined by

(ΦW)(t)=W0+Ξ(1Ψ)tΞ1ϖ(t,W(t))M(Ψ)+ΞΨM(Ψ)0tϑΞ1(tϑ)Ψ1ϖ(ϑ,W(ϑ))dϑ. (29)

Let

Πζ=WF:Wζ (30)

is close, convex, bounded subset with

ζΔ21Δ2 (31)

where

Δ2=Ξ(1Ψ)TΨ1M(Ψ)+ΞΨB(Ψ,Ξ)Tp+q1M(Ψ)ηϖ (32)

Define the operator Φ1, Φ2 such that Φ=Φ1+Φ2

Φ1W(t)=W0+Ξ(1Ψ)tΞ1ϖ(t,W(t))M(Ψ),Φ2W(t)=ΞΨM(Ψ)0tϑΞ1(tϑ)Ψ1ϖ(ϑ,W(ϑ))dϑ. (33)

Now we split the proof in the following steps as:

Step (1): Φ1W(t)+Φ2W(t)Πζ for all W, WΠζ. Indeed, we have

Φ1W+Φ2W=maxtJ|W0|+Ξ(1Ψ)tΞ1M(Ψ)|ϖ(t,W(t))|+ΞΨM(Ψ)0tϑΞ1(ϑt)Ψ1|ϖ(ϑ,W(t))|dϑ}|W0|+Ξ(Ψ1)tΞ1M(Ψ)[τϖ+Wηϖ]+ΞΨM(Ψ)0tϑΞ1(ϑt)Ψ1[τϖ+Wηϖ]dϑ}=|W0|+Ξ(Ψ1)tΞ1M(Ψ)+ΞΨB(Ψ,Ξ)tp+q1M(Ψ)τϖ+Ξ(1Ψ)tΞ1M(Ψ)+ΞΨB(Ψ,Ξ)tp+q1M(Ψ)ηϖζΔ1+Δ2ζζ. (34)

This proves that

Φ1W(t)+Φ2W(t)Πζ. (35)

Step (3) : Φ1 is contraction.

Let W1,W2Φζ. Then via (H2), we get

|Φ1W1Φ1W2|=maxtJΞ(1Ψ)tΞ1M(Ψ)|ϖ(t,W1(t))ϖ(t,W2(t))|maxtJΞ(1Ψ)tΞ1M(Ψ)Lϖ|W1(t)W2(t)|Ξ(1Ψ)TΞ1M(Ψ)LϖW1W2. (36)

Step (3) : Φ2 is relatively compact.

case 1 : Φ2 is continuous. Due to W(t) is continuous, then Φ2W(t) is continuous too.

case 2 : Φ2 is uniformly bounded on Φζ. Let W(t)Πζ. Then, we have

Φ1W=maxtJΞΨM(Ψ)0tϑΞ1(tϑ)|ϖ(ϑ,W(ϑ))|dϑΞΨB(Ψ,Ξ)TΞ+Ψ1M(Ψ)[τϖ+ζηϖ]. (37)

Hence Φ2 is uniformly bounded on Πζ.

case 3 : Φ2 is equicontinuous.

Let WΠζ and 0<t1<t2<T. Then

Φ2W(t2)Φ2W(t1)=maxtJ|ΞΨM(Ψ)0tϑΞ1(t2ϑ)Ψ1ϖ(ϑ,W(ϑ))dϑΞΨM(Ψ)0t1ϑΞ1(t1ϑ)Ψ1ϖ(ϑ,W(ϑ))dϑ|ΞΨM(Ψ)t1tt2ϑΞ1(t2ϑ)Ψ1|ϖ(ϑ,W(ϑ))|dϑ+ΞΨM(Ψ)0t1ϑΞ1(t2ϑ)Ψ1(t1ϑ)Ψ1×|ϖ(ϑ,W(ϑ))|dϑΞΨB(Ψ,Ξ)τϖ+ζηϖM(Ψ)(t2t1)p+q1. (38)

It follows that

Φ2W(t2)Φ2W(t1)0, as t1t2.

Thus, by Arzela–Ascoli theorem, we deduce that Φ2 is completely continuous. The Eq. (26) has at least one solution, so the proposed model has unique solution. □

Theorem 4

Assume that(H2)holds. if

Λ1=Ξ(1Ψ)TΞ1M(Ψ)+ΞΨB(Ψ,Ξ)TΞ+Ψ1M(Ψ)Lϖ<1, (39)

then the integral Eq. (24) has a unique solution which implies that the model (3) has a unique solution.

Proof

Taking the operator Φ:FF defined by (26). Let W1,W2F and tJ. Then

|ΦW1ΦW2|maxtJΞ(1Ψ)tΞ1M(Ψ)|ϖ(t,W1(t))ϖ(t,W2(t))|+maxtJΞΨM(Ψ)0tϑΞ1(tϑ)Ψ1|ϖ(t,W1(t))ϖ(t,W2(t))|dϑΛ|W1W2|. (40)

due to, Φ is contraction. Thus (26) has a unique solution, which yield that the model (3) has a unique solution. □

Hyers–Ulam Stability

Definition 1 [32]

The fractal fractional integral system given by Eqs. (5) is said to be Hyers–Ulam stable if exist constants Δi>0,iN6 satisfying: For every γi>0,iN8, for

|S(t)1ΨB(Ψ)L1(Ψ,t,S(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L1(Ψ,δ,S(δ))dδ|γ1,|E(t)1ΨB(Ψ)L2(Ψ,t,E(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L2(Ψ,δ,E(δ))dδ|γ2,|I(t)1ΨB(Ψ)L3(Ψ,t,I(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L3(Ψ,δ,I(δ))dδ|γ3,|A(t)1ΨB(Ψ)L4(Ψ,t,A(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L4(Ψ,δ,A(δ))dδ|γ4,|H(t)1ΨB(Ψ)L5(Ψ,t,H(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L5(Ψ,δ,H(δ))dδ|γ5,|R(t)1ΨB(Ψ)L6(Ψ,t,R(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L6(Ψ,δ,R(t)(δ))dδ|γ6. (41)

there exist S˙,E˙,I˙,A˙,H˙,R˙ which are satisfying

S˙(t)=1ΨB(Ψ)L1(Ψ,t,S(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L1(Ψ,δ,S˙(δ))dδ,E˙(t)=1ΨB(Ψ)L2(Ψ,t,Es(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L2(Ψ,δ,E˙(δ))dδ,I˙(t)=1ΨB(Ψ)L3(Ψ,t,I(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L3(Ψ,δ,I˙(δ))dδ,A˙(t)=1ΨB(Ψ)L4(Ψ,t,A(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L4(Ψ,δ,A˙(δ))dδ,H˙(t)=1ΨB(Ψ)L5(Ψ,t,H(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L5(Ψ,δ,H˙(δ))dδ,R˙(t)=1ΨB(Ψ)L6(Ψ,t,R(t))+ΨB(Ψ)Γ(Ψ)×0t(tδ)Ψ1L6(Ψ,δ,R˙(δ))dδ. (42)

Such that

|SS˙|ζ1γ1,|EE˙|ζ2γ2,|II˙|ζ3γ3,|AA˙|ζ4γ4,|HH˙|ζ5γ5,|RR˙|ζ6γ6.

Theorem 5

Presume that the assumption of Theorem 4 are satisfied. Then the model (3) will be UH stable.

Proof

The fractal fractional model (5) has at least one solution (S,E,I,A,H,R) satisfying equations of system (5). Then, we have

SS˙1ΨB(Ψ)L1(Ψ,t,S)L1(Ψ,t,S˙)+ΨB(Ψ)Γ(Ψ)0t(tδ)Ψ1L1(Ψ,t,S)L1(Ψ,t,S˙)dδ1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ)Ψ1SS˙ (43)
EE˙1ΨB(Ψ)L2(Ψ,t,E)L2(Ψ,t,E˙)+ΨB(Ψ)Γ(Ψ)0t(tδ)Ψ1L2(Ψ,t,E)L2(Ψ,t,E˙)dδ1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ)Ψ2EE˙ (44)
II˙1ΨB(Ψ)L3(Ψ,t,I)L3(Ψ,t,I˙)+ΨB(Ψ)Γ(Ψ)0t(tδ)Ψ1L3(Ψ,t,I)L3(Ψ,t,I˙)dδ1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ)Ψ3II˙ (45)
AA˙1ΨB(Ψ)L4(Ψ,t,A)L4(Ψ,t,A˙)+ΨB(Ψ)Γ(Ψ)0t(tδ)Ψ1L4(Ψ,t,A)L4(Ψ,t,A˙)dδ1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ)Ψ4AA˙ (46)
HH˙1ΨB(Ψ)L5(Ψ,t,H)L5(Ψ,t,H˙)+ΨB(Ψ)Γ(Ψ)0t(tδ)Ψ1L5(Ψ,t,H)L5(Ψ,t,H˙)dδ1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ)Ψ5HH˙ (47)
RR˙1ΨB(Ψ)L6(Ψ,t,R)L6(Ψ,t,R˙)+ΨB(Ψ)Γ(Ψ)0t(tδ)Ψ1L6(Ψ,t,R)L6(Ψ,t,R˙)dδ1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ)Ψ6RR˙ (48)

Taking, γi=Ψi,Δi=1ΨB(Ψ)+ΨB(Ψ)Γ(Ψ), this implies

SS˙γ1Δ1 (49)

Similarly, we have the followings

EE˙γ2Δ2II˙γ3Δ3AA˙γ4Δ4HH˙γ5Δ5RR˙γ6Δ6. (50)

Hence the proof is accomplished. □

Simulation results & numerical schemes

With the help of the numerical scheme as presented in the above sections, the models are simulated under various fractional orders for model (5). This is very important to show the feasibility of the reported work and investigate the validity of the analytical work using large-scale numerical simulation. It is important to point out that, unlike traditional numerical analysis, there are not as many options to choose schemes for the numerical analysis of the fractional order epidemiological models simulations. For the numerical solution of the fractal fractional model (5), we utilized the initial conditions, i.e, S(0)=150,E(0)=100,I(0)=50,A(0)=80,H(0)=80,R(0)=10, and the parameters value taken from Table 2. We can clear see from Fig. 2, Fig. 3, Fig. 4, the Adams–Bashforth method is faster to capture the solution of the nonlinear fractal fractional model as compared to the Newton polynomial method.

Fig. 2.

Fig. 2

Simulation results for the proposed model (5) via Newton polynomial for the different values fractal dimension Ξ and fractional order Ψ.

Fig. 3.

Fig. 3

Simulation results for the proposed model (5) via Adams–Bashforth method for the different values fractal dimension Ξ and fractional order Ψ.

Fig. 4.

Fig. 4

Simulation results for the proposed model (5) via Adams–Bashforth method for another set of initial condition at different values fractal dimension Ξ and fractional order Ψ.

Solution by Newton polynomial

The numerical scheme for Newton polynomial we can see in Fig. 2 the initial days the susceptible class is decreasing at different fractional order. Consequently, the exposed class first increases which also increase the infected class for initial few days. In same line the recovered class is raising which indicate that the infection is considerably reducing or they are going to die due to infection. Also, the virus class first increase and after the due to reduction in infection this class is also went on reducing. First, we can express model (1) as follows:

FFMD0,tΨ,Ξ[S]=GG1G2I+ASNG8S,FFMD0,tΨ,Ξ[E]=G1G2I+ASNG4+G8E,FFMD0,tΨ,Ξ[I]=1G3G4EG5+G6+G8I,FFMD0,tΨ,Ξ[A]=G3G4EG10+G7+G8A,FFMD0,tΨ,Ξ[H]=G5I+G10AG11+G9+G8H,FFMD0,tΨ,Ξ[R]=G6I+G7A+G11+G9HG8R. (51)

Now, we can rewrite the above system as:

FFMD0,tΨ,Ξ[S(t)]=S(t,S,E,I,A,H,R),FFMD0,tΨ,Ξ[E(t)]=E(t,S,E,I,A,H,R),FFMD0,tΨ,Ξ[I(t)]=I(t,S,E,I,A,H,R),FFMD0,tΨ,Ξ[A(t)]=A(t,S,E,I,A,H,R),FFMD0,tΨ,Ξ[H(t)]=H(t,S,E,I,A,H,R),FFMD0,tΨ,Ξ[R(t)]=R(t,S,E,I,A,H,R). (52)

Applying the fractal fractional integral and plugging Newton polynomial into these equations, we can get;

Sa+1=1ΨAB(Ψ)+S(ta,Sa,Ea,Ia,Aa,Ha,Ra)+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+1)μ=2aS(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Π+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+2)×μ=2aS(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)S(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Σ+Ψ(Δt)Ψ2AB(Ψ)Γ(Ψ+3)×μ=2aS(tμ,Sμ,Eμ,Iμ,Aμ,Hμ,Rμ)2S(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)+S(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Δ
Ea+1=1ΨAB(Ψ)+E(ta,Sa,Ea,Ia,Aa,Ha,Ra)+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+1)μ=2aE(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Π+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+2)×μ=2aE(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)E(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Σ+Ψ(Δt)Ψ2AB(Ψ)Γ(Ψ+3)×μ=2aE(tμ,Sμ,Eμ,Iμ,Aμ,Hμ,Rμ)2E(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)+E(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Δ
Ia+1=1ΨAB(Ψ)+I(ta,Sa,Ea,Ia,Aa,Ha,Ra)+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+1)μ=2aI(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Π+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+2)×μ=2aI(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)I(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Σ+Ψ(Δt)Ψ2AB(Ψ)Γ(Ψ+3)×μ=2aI(tμ,Sμ,Eμ,Iμ,Aμ,Hμ,Rμ)2I(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)+I(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Δ
Aa+1=1ΨAB(Ψ)+A(ta,Sa,Ea,Ia,Aa,Ha,Ra)+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+1)μ=2aA(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Π+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+2)×μ=2aA(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)A(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Σ+Ψ(Δt)Ψ2AB(Ψ)Γ(Ψ+3)×μ=2aA(tμ,Sμ,Eμ,Iμ,Aμ,Hμ,Rμ)2A(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)+A(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Δ
Ha+1=1ΨAB(Ψ)+H(ta,Sa,Ea,Ia,Aa,Ha,Ra)+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+1)μ=2aH(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Π+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+2)×μ=2aH(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)H(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Σ+Ψ(Δt)Ψ2AB(Ψ)Γ(Ψ+3)×μ=2aH(tμ,Sμ,Eμ,Iμ,Aμ,Hμ,Rμ)2H(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)+H(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Δ
Ra+1=1ΨAB(Ψ)+R(ta,Sa,Ea,Ia,Aa,Ha,Ra)+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+1)μ=2aR(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Π+Ψ(Δt)ΨAB(Ψ)Γ(Ψ+2)×μ=2aR(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)R(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Σ+Ψ(Δt)Ψ2AB(Ψ)Γ(Ψ+3)×μ=2aR(tμ,Sμ,Eμ,Iμ,Aμ,Hμ,Rμ)2R(tμ1,Sμ1,Eμ1,Iμ1,Aμ1,Hμ1,Rμ1)+R(tμ2,Sμ2,Eμ2,Iμ2,Aμ2,Hμ2,Rμ2)Δ

where

Δ=(aμ+1)Ψ2(aμ)2+(3Ψ+10)(aμ)+2ΦI2+9Ψ+12(aμ)Ψ2(aμ)2+(5Ψ+10)(aμ)+6ΨI2+18Ψ+12,
Σ=(aμ+1)Ψ(aμ+3+2Ψ)(aμ)Ψ(aμ+3+3Ψ),
Π=[(aμ+1)Ψ(aμ)Ψ].

Numerical scheme by Adams Bashforth method

The numerical scheme for Adams Bashforth method we can see in Fig. 3, Fig. 4 the initial days the susceptible class is decreasing at different fractional order. Consequently, the exposed class first increases which also increase the infected class for initial few days. In same line the recovered class is raising which indicate that the infection is considerably reducing or they are going to die due to infection. Also, the virus class first increase and after the due to reduction in infection this class is also went on reducing. First, we can express model (1) as follows:

FFMD0ΨS=ΞtΞ1F1(t,S,E,I,A,H,R,)FFMD0ΨE=ΞtΞ1F2(t,S,E,I,A,H,R,)FFMD0ΨI=ΞtΞ1F3(t,S,E,I,A,H,R,)FFMD0ΨA=ΞtΞ1F4(t,S,E,I,A,H,R,)FFMD0ΨH=ΞtΞ1F5(t,S,E,I,A,H,R,)FFMD0ΨR=ΞtΞ1F6(t,S,E,I,A,H,R,). (53)

Now, applying the fractal fractional integral to both sides of (53), we obtained the following system

S(t)S(0)=Ξ(1Ψ)tΞ1M(Ψ)F1(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F1(s,S,E,I,A,H,R)dsE(t)E(0)=Ξ(1Ψ)tΞ1M(Ψ)F2(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F2(s,S,E,I,A,H,R)dsI(t)I(0)=Ξ(1Ψ)tΞ1M(Ψ)F3(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F3(s,S,E,I,A,H,R)dsA(t)A(0)=Ξ(1Ψ)tΞ1M(Ψ)F3(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F3(s,S,E,I,A,H,R)dsH(t)H(0)=Ξ(1Ψ)tΞ1M(Ψ)F4(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F4(s,S,E,I,A,H,R)dsR(t)R(0)=Ξ(1Ψ)tΞ1M(Ψ)F5(t,S,E,I,A,H,R)+ΞΨM(Ψ)0tsΞ1(ts)Ψ1F5(s,S,E,I,A,H,R)ds (54)

Set t=tm+1 for m=0,1,2,, it follows that

Stm+1S(0)=Ξ(1Ψ)tmΞ1M(Ψ)F1tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1sΞ1tm+1sΨ1F1(s,S,E,I,A,H,R)dsEtm+1E(0)=Ξ(1Ψ)tmΞ1M(Ψ)F2tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1sΞ1tm+1sΨ1F2(s,S,E,I,A,H,R)dsAtm+1A(0)=Ξ(1Ψ)tmΞ1M(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1sΞ1tm+1sΨ1F3(s,S,E,I,A,H,R)dsHtm+1H(0)=Ξ(1Ψ)tmΞ1M(Ψ)F4tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1sΞ1tm+1sΨ1F4(s,S,E,I,A,H,R)dsRtm+1R(0)=Ξ(1Ψ)tmΞ1M(Ψ)F5tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1sΞ1tm+1sΨ1F5(s,S,E,I,A,H,R)ds (55)

Here, we approximate the functions sΞ1F(tm,S,E,A,B,R) through the interpolation polynomial with h=tn+1tn as follows

xi(s):=sΞ1Fi(s,S,E,I,A,H,R)(ttn1)htnΞ1Fitn,S(tn),E(tn),A(tn),H(tn),R(tn)(ttn)htn1Ξ1Fitn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1),

Plugging the last equation into (55), we may write (55) as

S(tm+1)S(0)=Ξ(1Ψ)tmΞ1M(Ψ)F1tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1tm+1sΨ1x1(s)ds,E(tm+1)E(0)=Ξ(1Ψ)tmΞ1M(Ψ)F2tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1tm+1sΨ1x2(s)ds,I(tm+1)I(0)=Ξ(1Ψ)tmΞ1M(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1tm+1sΨ1x3(s)ds,A(tm+1)A(0)=Ξ(1Ψ)tmΞ1M(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1tm+1sΨ1x3(s)ds,H(tm+1)H(0)=Ξ(1Ψ)tmΞ1M(Ψ)F5tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1tm+1sΨ1x4(s)ds,R(tm+1)R(0)=Ξ(1Ψ)tmΞ1M(Ψ)F6tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtntn+1tm+1sΨ1x5(s)ds. (56)

By the approximate the functions xi(s), (56) becomes

Stm+1=S(0)+Ξ(1Ψ)tmΞ1M(Ψ)F1tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F1tn,S(tn),E(tn),A(tn),H(tn),R(tn)hN1tn1Ξ1F1tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hN2 (57)
Etm+1=E(0)+Ξ(1Ψ)tmΞ1M(Ψ)F2tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F2tn,S(tn),E(tn),A(tn),H(tn),R(tn)hN1tn1Ξ1F2tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hN2 (58)
Itm+1=I(0)+Ξ(1Ψ)tmΞ1M(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F3tn,S(tn),E(tn),A(tn),H(tn),R(tn)hN1tn1Ξ1F3tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hN2 (59)
Atm+1=A(0)+Ξ(1Ψ)tmΞ1M(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F4tn,S(tn),E(tn),A(tn),H(tn),R(tn)hN1tn1Ξ1F3tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hN2 (60)
Htm+1=H(0)+Ξ(1Ψ)tmΞ1M(Ψ)F4tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F5tn,S(tn),E(tn),A(tn),H(tn),R(tn)hN1tn1Ξ1F4tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hN2 (61)
Rtm+1=R(0)+Ξ(1Ψ)tmΞ1M(Ψ)F6tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F5tn,S(tn),E(tn),A(tn),H(tn),R(tn)hN1tn1Ξ1F5tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hN2 (62)

where

N1=tntn+1(tm+1s)Ψ1(ttn1)ds

and

N2=tntn+1(tm+1s)Ψ1(ttn)ds.

By simple calculations, we get

N1=1p(tn+1tn1)(tm+1tn+1)Ψ(tntn1)(tm+1tn)Ψ1Ψ(Ψ+1)(tm+1tn+1)Ψ+1(tm+1tn)Ψ+1,

and

N2=1p(tn+1tn)(tm+1tn+1)Ψ1Ψ(Ψ+1)(tm+1tn+1)Ψ+1(tm+1tn)Ψ+1.

put tn=nh, we get

N1=hΨ+1Ψ(Ψ+1)(m+1n)(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ), (63)

and

N2=hΨ+1Ψ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ). (64)

Substituting (63), (64) into Eqs. (57)(62), we get  

S(tm+1)=S(0)+Ξ(1Ψ)tmM(Ψ)F1tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F1tn,S(tn),E(tn),A(tn),H(tn),M(tn)hΨΨ(Ψ+1)(m+1n)Ψ(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ)tn1Ξ1F1tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hΨΨ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ)
E(tm+1)=E(0)+Ξ(1Ψ)tmM(Ψ)F2tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F2tn,S(tn),E(tn),A(tn),H(tn),M(tn)hΨΨ(Ψ+1)(m+1n)Ψ(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ)tn1Ξ1F2tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hΨΨ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ)
I(tm+1)=I(0)+Ξ(1Ψ)tmM(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F3tn,S(tn),E(tn),A(tn),H(tn),M(tn)hΨΨ(Ψ+1)(m+1n)Ψ(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ)tn1Ξ1F3tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hΨΨ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ)
A(tm+1)=A(0)+Ξ(1Ψ)tmM(Ψ)F3tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F3tn,S(tn),E(tn),A(tn),H(tn),M(tn)hΨΨ(Ψ+1)(m+1n)Ψ(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ)tn1Ξ1F3tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hΨΨ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ)
H(tm+1)=H(0)+Ξ(1Ψ)tmM(Ψ)F4tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F4tn,S(tn),E(tn),A(tn),H(tn),M(tn)hΨΨ(Ψ+1)(m+1n)Ψ(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ)tn1Ξ1F4tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hΨΨ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ)
R(tm+1)=R(0)+Ξ(1Ψ)tmM(Ψ)F5tm,S,E,I,A,H,R+ΞΨM(Ψ)n=1mtnΞ1F5tn,S(tn),E(tn),A(tn),H(tn),M(tn)hΨΨ(Ψ+1)(m+1n)Ψ(mn+2+Ψ)(mn)Ψ(mn+2+2Ψ)tn1Ξ1F5tn1,S(tn1),E(tn1),A(tn1),H(tn1),R(tn1)hΨΨ(Ψ+1)(m+1n)Ψ+1(mn)Ψ(mn+1+Ψ)

Conclusion

This study presents a novel approach for understanding the dynamics of the mathematical modeling approach that provides strong conclusions on the transmission mechanism of the newly but deeply investigated COVID-19 pandemic driven infections. To define the proposed model the COVID-19, the infected people are divided into two classes, namely, detected and undetected classes. The Fractal fractional order derivative with fractal dimension Ξ and fractional order Ψ in ABC sense is used to more readily investigate the infection dynamics. After the model definition, at first, we introduced the fundamental and essential numerical provisions of the fractal fractional COVID-19 pandemic model. We make use of the fractional order stability approach for the local stability of both endemic as well as the disease-free equilibrium points. The fractal and fractional order mathematical model in the ABC sense are solved numerically via Newton polynomial and Adams–Bashforth techniques. We believe that the attempt made in this work will provide fruitful insights for adopting strategies in reducing the continuous COVID-19 pandemic.

CRediT authorship contribution statement

Jian-Cun Zhou: Conceptualization, Data curation, Methodology, Writing - original draft. Soheil Salahshour: Software, Validation, Formal analysis, Review editing. Ali Ahmadian: Supervision, Project administration, Funding acquisition. Norazak Senu: Visualization, Software, Funding acquisition.

Declaration of Competing Interest

The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Acknowledgment

This work was supported by the Key projects of Hunan Provincial Department of Education, China (Grant No.: 20A88).

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