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. 2022 Jan 4;13(1):e03144-21. doi: 10.1128/mBio.03144-21

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Copyright © 2022 Vadovics et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license.

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FIG 1 — Effects of HI Candida and zymosan on HO-1-N-1 and HSC-2 oral squamous cell carcinoma cells in vitro. (A) Normalized migration activity of OSCC cells in the presence of HI C. albicans, HI C parapsilosis, and zymosan measured by a wound healing assay (n = 3). (B) Normalized total secreted matrix metalloproteinase (MMP) activity of OSCC cells in the presence of HI C. albicans, HI C. parapsilosis, and zymosan measured by a total MMP activity kit (n = 4). (C) Normalized amounts of metabolites of OSCC cells in the presence of HI C. albicans, HI C. parapsilosis, and zymosan as measured by HPLC-HRMS (n = 4). FUM, fumaric acid; GA-3P, glyceraldehyde-3P; ASP, aspartic acid; ERY, erythrose-4P; OXA, oxaloacetic acid; SUC, succinic acid; G/F-6P, glucose/fructose-6p; MET, methionine; control, tumor cells without any treatment. Unpaired t test; *, P ≤ 0.05; **, P ≤ 0.01; ****, P ≤ 0.0001. ns, nonsignificant.