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Journal of Experimental & Clinical Cancer Research : CR logoLink to Journal of Experimental & Clinical Cancer Research : CR
. 2022 Jan 21;41:30. doi: 10.1186/s13046-021-02238-5

Correction to: ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 transcription in nasopharyngeal carcinoma

Hao Peng 1,#, Jian Zhang 2,#, Pan-Pan Zhang 2,#, Lei Chen 1, Ling-Long Tang 1, Xiao-Jing Yang 2, Qing-Mei He 2, Xin Wen 2, Ying Sun 1, Na Liu 2, Ying-Qin Li 2,✉, Jun Ma 1,✉
PMCID: PMC8780747  PMID: 35062998

Correction to: J Exp Clin Cancer Res 38, 11 (2019)

https://doi.org/10.1186/s13046-018-0997-7

Following publication of the original article [1], the authors identified some minor errors in Supplemental Figs. 1 and 4, specifically:

  • Fig. S1b: incorrect sample size listed; correct sample size is 25

  • Fig. S4b: incorrect image used for SUNE1-ARNTL (24 h); correct image is now used

The authors provided the journal with their original data. The corrected figures are given here. The corrections do not have any effect on the final conclusions of the paper. The original article has been corrected.

Supplementary Information

13046_2021_2238_MOESM2_ESM.tif (2.2MB, tif)

Additional file 2: Fig. S1. ARNTL methylation levels in the GSE52068 and GSE62366 nasopharyngeal carcinoma datasets.

13046_2021_2238_MOESM5_ESM.tif (10.6MB, tif)

Additional file 5: Fig. S4. Overexpression of ARNTL had no impact on nasopharyngeal carcinoma cells invasion and migration. (A) Images of Transwell invasion (left) and migration (right) assay with ARNTL-overexpression or Vector-overexpression SUNE1 and HONE1 cells. (B) Images of wound healing assay with ARNTL-overexpression or Vector-overexpression SUNE1 and HONE1 cells.

Footnotes

Hao Peng, Jian Zhang and Pan-Pan Zhang contributed equally to this work.

Contributor Information

Ying-Qin Li, Email: liyingq@sysucc.org.cn.

Jun Ma, Email: majun2@mail.sysu.edu.cn.

Reference

  • 1.Peng H, Zhang J, Zhang PP, et al. ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 transcription in nasopharyngeal carcinoma. J Exp Clin Cancer Res. 2019;38:11. doi: 10.1186/s13046-018-0997-7. [DOI] [PMC free article] [PubMed] [Google Scholar]

Associated Data

This section collects any data citations, data availability statements, or supplementary materials included in this article.

Supplementary Materials

13046_2021_2238_MOESM2_ESM.tif (2.2MB, tif)

Additional file 2: Fig. S1. ARNTL methylation levels in the GSE52068 and GSE62366 nasopharyngeal carcinoma datasets.

13046_2021_2238_MOESM5_ESM.tif (10.6MB, tif)

Additional file 5: Fig. S4. Overexpression of ARNTL had no impact on nasopharyngeal carcinoma cells invasion and migration. (A) Images of Transwell invasion (left) and migration (right) assay with ARNTL-overexpression or Vector-overexpression SUNE1 and HONE1 cells. (B) Images of wound healing assay with ARNTL-overexpression or Vector-overexpression SUNE1 and HONE1 cells.

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