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. 2021 Oct 19;12(1):8006–8019. doi: 10.1080/21655979.2021.1981795

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© 2021 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — CASC9 promoted cell proliferation, migration and invasion in OVCAR-3 and OVCAR-3/DDP cells. A, Cell inhibitory rate in OVCAR-3 and OVCAR-3/DDP cells was evaluated by CCK-8 assay. *P < 0.05. B, CASC9 mRNA level was detected after transfection with shRNA-CASC9 or OverExp-CASC9. ***P < 0.001 versus control; ^###P < 0.001 versus vector. C, Cell proliferation were identified by CCK-8 assay after transfection with shRNA-CASC9 or OverExp-CASC9. D, Cell inhibitory rate was identified by CCK-8 assay after transfection with shRNA-CASC9 or OverExp-CASC9. E, Cell migration was evaluated by wound healing assay after transfection with shRNA-CASC9 or OverExp-CASC9. F, Cell invasion was examined by transwell assay after transfection with shRNA-CASC9 or OverExp-CASC9. Data are expressed as mean ± SD. *P < 0.05, ***P < 0.001 versus OVCAR-3 cells; ^#P < 0.05, ^##P < 0.01, ^###P < 0.001 versus OVCAR-3/DDP+vector