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. 2022 Jan 26;45(2):812–823. doi: 10.1007/s10753-021-01586-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — RSV alleviates the CCl₄-induced rat model of hepatic fibrosis. Rat HF models were established with or without RSV treatment. Liver tissues were collected after 3 months. A Livers were sectioned for H&E, Masson’s trichrome, and Sirius red staining in the treatment groups. Representative images were shown on the left (scale bars, 40 μm) and summarized results are on the right. B mRNA levels of COL1A1 and PPAR-γ in livers were measured by qPCR. Data are normalized to expression levels of control. C Protein levels of COL1A1 and PPAR-γ in livers were measured by western blot. Representative images were shown on the left and summarized results are on the right. A–C Data are mean ± SD from three independent experiments (n = 6 rats per group). More than 12 rats were used for each experiment to make sure that at least 3 rats were survived in the CCl₄ group 3 months after HF development. *p < 0 .05, **p < 0.01, and ***p < 0.001.