In the article as published originally, the incorrect nomenclature was used when referring to several members of the plppr family in the second paragraph of the Introduction.
The sentence “Plppr1 drives cell autonomous signaling pathways to participate in the regulation of spinal density and subsequent memory formation (11, 12)” should read “Plppr4 drives cell autonomous signaling pathways to participate in the regulation of spinal density and subsequent memory formation (11, 12)”.
The sentence “Plppr2 induces collateral branch growth in axons (14)” should read “Plppr3 induces collateral branch growth in axons (14)”.
The sentence “Plppr3 induces neurites that are resistant to growth inhibitors associated with brain injury and can help restore function after spinal cord injury (15)” should read “Plppr1 induces neurites that are resistant to growth inhibitors associated with brain injury and can help restore function after spinal cord injury (15)”.
The sentence “The Plppr4 protein is highly expressed during the development and regeneration of synapses, can regulate synaptic lysophosphatidic acid (LPA) levels and is associated with epilepsy and brain damage.” should read “The Plppr2 protein is highly expressed during the development and regeneration of synapses, can regulate synaptic lysophosphatidic acid (LPA) levels and is associated with epilepsy and brain damage.”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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