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. 2022 Mar 22;12(4):484. doi: 10.3390/biom12040484

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Signaling pathways arising following non-voltage Ca²⁺ activation in PAECs. Calmodulin (caM); endoplasmic reticulum (ER); endothelial cells (ECs); endothelial nitric oxide synthase (eNOS); inositol triphosphate receptor (IP3R); myosin light-chain kinase (MLCK); myeloid cell leukemia (MCL); nitric oxide (NO); nuclear factor of activated T cells (NFAT); store-inhibited channels (SICs); storage-operated Ca²⁺ entry (SOCE); store-operated channels (SOCs); receptor-operated channels (ROC); ryanodin receptor (RyR); transient receptor potential canonical channels (TRPC).