Yating He, Xiaofeng Ma, Daojing Li and Junwei Hao. Thiamet G mediates neuroprotection in experimental stroke by modulating Microglia/macrophage polarization and inhibiting NF-κB p65 signaling. J Cereb Blood Flow Metab 2017: 37(8); 2938–2951. DOI: 10.1177/0271678X16679671.
After the publication of this article, the authors noticed that the images in Figure 4(a), (c) and Figure 6(b) were inadvertently mislaid. The correct images are supplied below.
Figure 4.
Co-expression of Iba-1 and M1/M2 phenotype markers. Brain slices were prepared at 72 h after MCAO. Immunofluorescent images were captured microscopically in the peri-infarct area of cortex. (a) Cortex co-stained for Iba-1 (microglia marker) (red) and CD16/32(M1 marker) (green) in the peri-infarct area. (c) Cortex co-stained for Iba-1 (microglia marker) (red) and CD206(M2 marker) (green) in the peri-infarct area.
Figure 6.
Effect of TMG on NF-kB transcriptional activity in MCAO mice. Brain sections were prepared at 72 h after stroke. Immunofluorescent images were captured microscopically. (b) Cortex splices co-stained with iNOS (red) and DAPI (blue) in the peri-infarct area.