Author Correction: Neuroprotection by acetyl-11-keto-β-boswellic acid, in ischemic brain injury involves the Nrf2/HO-1 defense pathway

Correction to: Scientific Reports 10.1038/srep07002, published online 11 November 2014

This Article contains errors.

In Figure 5A, the image for His H3 was inadvertently duplicated from Figure 5A. A corrected version of Figure 5 and its accompanying legend are included below.

Figure 5.

AKBA induces expression of Nrf2 and Nrf2-binding activity in primary cultured neurons. All data represent the mean ± SD of triplicate independent experiments. (A) AKBA induced Nrf2 expression in a concentration-dependent manner. *P < 0.05 vs control (B) After 2 hour treatment with vehicle or 50 μM AKBA, nuclear extracts were prepared and were used to analyze Nrf2 bingding activity by EMSA. (C) Cells were transiently transfected with control or Nrf2 siRNA for 48 h (transfection efficiency was checked by Western analysis), followed by treatment with 50 μM of AKBA for an additional 8 h. Nuclear extracts were analyzed for Nrf2 levels. (D) Representative immunoblots for HO-1 following 50 μM of AKBA treatment for 24 h in control and Nrf2 siRNA-treated cells. *P < 0.05 vs si-control group without AKBA and ^#P < 0.05 vs si-control group with AKBA.