Epithelial STAT6 O-GlcNAcylation drives a concerted anti-helminth alarmin response dependent on tuft cell hyperplasia and Gasdermin C

In our recent paper entitled “Epithelial STAT6 O-GlcNAcylation drives anti-helminth immunity via a concerted anti-helminth alarmin response dependent on tuft cell hyperplasia and Gasdermin C” (Immunity 55:623–638 2022), we reported the role of GSDMC in serving as a conduit for the release of interleukin-33 from intestinal epithelial cells. We relied on a mouse strain lacking the 4 Gsdmc genes to support our conclusion. Regrettably, we referred incorrectly to a different strain in the STAR Methods and did not include the genetic characterization of the Gsdmc1,2,3,4-flox mouse strain in our article. This information is now accessible as a supplemental file (Methods S1). The authors apologize for any confusion the absence of this information may have created.