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Journal of Experimental & Clinical Cancer Research : CR logoLink to Journal of Experimental & Clinical Cancer Research : CR
. 2022 Aug 3;41:236. doi: 10.1186/s13046-022-02454-7

Correction: 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase

Wan Li 1,2,#, Jinyi Liu 2,3,#, Weiqi Fu 1,2, Xiangjin Zheng 1,2, Liwen Ren 1,2, Shiwei Liu 4, Jinhua Wang 1,2,, Tengfei Ji 1,, Guanhua Du 1,2,
PMCID: PMC9347118  PMID: 35918768

Correction: J Exp Clin Cancer Res 37, 132 (2018)

https://doi.org/10.1186/s13046-018-0805-4

Following publication of the original article [1], an error was identified in Fig. 3; specifically:

  • Figure 3e and 3f, panels “U251-MG 48h Control” and “U87-MG 48h Control” were inadvertently duplicated

Fig. 3.

Fig. 3

AKBA induces mitochondria-dependent apoptosis in U251 and U87-MG cells in a dose-dependent manner. Flow cytometry using Annexin V-FITC staining shows that AKBA increases apoptosis of U251 (a) and U87-MG (b) cells. Flow cytometry using JC-1 staining shows that AKBA reduces mitochondrial membrane potential in U251(c) and U87-MG (d) cells. Flow cytometry using caspase 3/7 live-cell staining shows that AKBA increases the activity of caspase 3/7 in U251 (e) and U87-MG (f) cells. Western blotting results show that AKBA induces expression of cleaved-caspase 3 and cleaved-PARP in U251 (g) and U87-MG (h) cells. The experiments were performed in triplicate

The correction does not have any effect on the results or conclusions of the paper.

Footnotes

Wan Li and Jinyi Liu contributed equally to this work.

Contributor Information

Jinhua Wang, Email: wjh@imm.ac.cn.

Tengfei Ji, Email: jitf@imm.ac.cn.

Guanhua Du, Email: dugh@imm.ac.cn.

Reference

  • 1.Li W, Liu J, Fu W, et al. 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase. J Exp Clin Cancer Res. 2018;37:132. doi: 10.1186/s13046-018-0805-4. [DOI] [PMC free article] [PubMed] [Google Scholar]

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