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. 2023 Mar 10;80(4):85. doi: 10.1007/s00018-023-04725-8

Correction: An oncogene addiction phosphorylation signature and its derived scores inform tumor responsiveness to targeted therapies

Eleonora Orlando 1,2,#, Matúš Medo 1,2,#, Ariel Bensimon 3, Aurélie Quintin 1,2, Rahel Riedo 1,2, Selina M Roth 1,2, Carsten Riether 4,5, Thomas M Marti 6,7, Daniel M Aebersold 1,2, Michaela Medová 1,2,#, Ruedi Aebersold 3,8,✉,#, Yitzhak Zimmer 1,2,✉,#
PMCID: PMC9998563  PMID: 36894640

Correction: Cellular and Molecular Life Sciences (2022) 80:6 10.1007/s00018-022-04634-2

In the published article reference 41 was missed in the third paragraph, under discussion section in the page 15, and the below mentioned reference has been incorrectly processed, and the error in the below references has been now updated.

Previously, we have reported that aberrant activation of the MET receptor modulates the cellular response to IR by rewiring key DNA damage response (DDR)-related phosphorylations in some tumor cell lines featuring MET activation [41]. Assuming that a MET–DDR interface underlies MET dependency, here we monitored 116 DDR- and RTK signalling-associated phosphosites in a panel of MET-positive, MET-responsive as well as non-responsive tumor models following targeted MET inhibition. This analysis revealed 14 METi-modulated phosphorylation events that were present solely in MET-addicted models, thus representing ‘MET-asa-driver’ footprints.

The original article has been updated.

Footnotes

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Contributor Information

Ruedi Aebersold, Email: aebersold@imsb.biol.ethz.ch.

Yitzhak Zimmer, Email: yitzhak.zimmer@insel.ch.

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