Abstract
In both animals and humans, Klüver-Bucy syndrome is produced by bilateral temporal lobectomy. It is characterised by hypersexuality, visual agnosia, strong oral tendencies, dietary changes, and hypermetamorphosis. Recurrent, postictal Klüver-Bucy syndrome occurred transiently after seizures in a female who had undergone unilateral temporal lobectomy. The pathophysiological mechanism may have been postictal dysfunction of the remaining temporal lobe, producing a transient functional bilateral temporal lobectomy.
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