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. 2023 Jul 28;11(8):656. doi: 10.3390/toxics11080656

Correction: Woodlief et al. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics 2021, 9, 100

Tracey Woodlief 1,*, Samuel Vance 1, Qing Hu 1, Jamie DeWitt 1
PMCID: PMC10459841  PMID: 37624225

Error in Figure/Table

Error in figure x-axis (Figure 1A: Hepatic peroxisome proliferation). In the original publication [1] there was a mistake in Figure 1A as published. The x-axis was supposed to be labeled with the following doses: 0, 0.00025, 0.025, or 2.5 PFOA. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Figure 1.

Figure 1

Hepatic peroxisome proliferation (percent change from 0 mg/kg control) of male and female C57BL/6 mice orally exposed to (A): PFMOAA, (B): PFMOPrA, or (C): PFMOBA for 30 days. Acyl-CoA oxidase activity was measured in livers that had been collected from animals one day after exposure ended. n = 4–6/dose for PFMOAA, PFMOPrA, PFMOBA, and PFOA-positive control (note that the PFOA-positive control was included from animals evaluated in a separate PFAS study). No error bars are present due to how the data were calculated. Abbreviations: perfluoro-2-methoxyacetic acid (PFMOAA), perfluoro-2-methoxypropanoic acid (PFMOPrA), perfluoro-4-methoxybutanioc acid (PFMOBA), and perfluorooctanoic acid (PFOA). * p < 0.05 from same-sex control group.

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Reference

  • 1.Woodlief T., Vance S., Hu Q., DeWitt J. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics. 2021;9:100. doi: 10.3390/toxics9050100. [DOI] [PMC free article] [PubMed] [Google Scholar]

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